G-CSF activation of AKT is not sufficient to prolong neutrophil survival
about
Kinase AKT controls innate immune cell development and functionPathogenesis of ELANE-mutant severe neutropenia revealed by induced pluripotent stem cellsCellular stress pathways in pediatric bone marrow failure syndromes: many roads lead to neutropenia.Molecular control of PtdIns(3,4,5)P3 signaling in neutrophils.Deficiency of PI3-Kinase catalytic isoforms p110γ and p110δ in mice enhances the IL-17/G-CSF axis and induces neutrophilia.
P2860
G-CSF activation of AKT is not sufficient to prolong neutrophil survival
description
2013 nî lūn-bûn
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2013年の論文
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2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
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2013年论文
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name
G-CSF activation of AKT is not sufficient to prolong neutrophil survival
@en
type
label
G-CSF activation of AKT is not sufficient to prolong neutrophil survival
@en
prefLabel
G-CSF activation of AKT is not sufficient to prolong neutrophil survival
@en
P2093
P2860
P356
P1476
G-CSF activation of AKT is not sufficient to prolong neutrophil survival
@en
P2093
Carlos Cabrera
Elissa Calloway
Erica Silva
Liliana R Souza
Morgan L McLemore
P2860
P304
P356
10.1189/JLB.1211591
P577
2013-04-04T00:00:00Z