The transforming mutation E17K/AKT1 is not a major event in B-cell-derived lymphoid leukaemias.
about
Cancer associated E17K mutation causes rapid conformational drift in AKT1 pleckstrin homology (PH) domainThe PI3K-Akt-mTOR pathway in initiation and progression of thyroid tumors.The oncogenic mutation in the pleckstrin homology domain of AKT1 in endometrial carcinomas.Differential effects of AKT1(p.E17K) expression on human mammary luminal epithelial and myoepithelial cells.Reduced expression of the tumor suppressor PHLPP1 enhances the antiapoptotic B-cell receptor signal in chronic lymphocytic leukemia B-cells.Differential effects of selective inhibitors targeting the PI3K/AKT/mTOR pathway in acute lymphoblastic leukemia.Oncogenic E17K mutation in the pleckstrin homology domain of AKT1 promotes v-Abl-mediated pre-B-cell transformation and survival of Pim-deficient cells.Infrequent occurrence of PIK3CA mutations in chronic lymphocytic leukemia.
P2860
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P2860
The transforming mutation E17K/AKT1 is not a major event in B-cell-derived lymphoid leukaemias.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
The transforming mutation E17K ...... l-derived lymphoid leukaemias.
@en
type
label
The transforming mutation E17K ...... l-derived lymphoid leukaemias.
@en
prefLabel
The transforming mutation E17K ...... l-derived lymphoid leukaemias.
@en
P2093
P2860
P356
P1476
The transforming mutation E17K ...... ll-derived lymphoid leukaemias
@en
P2093
H A Mohammad
M S EL-Khateeb
S I Ismail
P2860
P2888
P304
P356
10.1038/SJ.BJC.6604512
P407
P577
2008-08-01T00:00:00Z