The relationship between the severity of hemolysis, clinical manifestations and risk of death in 415 patients with sickle cell anemia in the US and Europe - Wikidata - wikimedia - TriplyDB

The relationship between the severity of hemolysis, clinical manifestations and risk of death in 415 patients with sickle cell anemia in the US and Europe

The relationship between the severity of hemolysis, clinical manifestations and risk of death in 415 patients with sickle cell anemia in the US and Europe

http://www.wikidata.org/entity/Q36862364

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Secondhand Smoke Is an Important Modifiable Risk Factor in Sickle Cell Disease: A Review of the Current Literature and Areas for Future Research Decreased hematocrit-to-viscosity ratio and increased lactate dehydrogenase level in patients with sickle cell anemia and recurrent leg ulcers Men with Sickle Cell Anemia and Priapism Exhibit Increased Hemolytic Rate, Decreased Red Blood Cell Deformability and Increased Red Blood Cell Aggregate Strength Inflammation in Sickle Cell Disease: Differential and Down-Expressed Plasma Levels of Annexin A1 Protein Genetic determinants of haemolysis in sickle cell anaemia.Hematopoietic stem cell transplantation in thalassemia major and sickle cell disease: indications and management recommendations from an international expert panel.Risk factors for mortality in adult patients with sickle cell disease: a meta-analysis of studies in North America and Europe.Cellular, pharmacological, and biophysical evaluation of explanted lungs from a patient with sickle cell disease and severe pulmonary arterial hypertension The co-inheritance of alpha-thalassemia and sickle cell anemia is associated with better hematological indices and lower consultations rate in Cameroonian patients and could improve their survival.Risk factors for death in 632 patients with sickle cell disease in the United States and United Kingdom Reduction of the six-minute walk distance in children with sickle cell disease is correlated with silent infarct: results from a cross-sectional evaluation in a single center in Belgium Elevated pulse pressure is associated with hemolysis, proteinuria and chronic kidney disease in sickle cell disease.Peroxisome proliferator-activated receptor-α-mediated transcription of miR-199a2 attenuates endothelin-1 expression via hypoxia-inducible factor-1α Male gender, increased blood viscosity, body mass index and triglyceride levels are independently associated with systemic relative hypertension in sickle cell anemia.Vasculopathy and pulmonary hypertension in sickle cell disease.Predictors of abnormal bone mass density in adult patients with homozygous sickle-cell disease.Circulating cell membrane microparticles transfer heme to endothelial cells and trigger vasoocclusions in sickle cell disease.Low forced expiratory volume is associated with earlier death in sickle cell anemia Impairment of neutrophil oxidative burst in children with sickle cell disease is associated with heme oxygenase-1.Association between oxidative stress and vascular reactivity in children with sickle cell anaemia and sickle haemoglobin C disease.Pathophysiology and treatment of pulmonary hypertension in sickle cell disease Red Blood Cells Store and Release Interleukin-33 Nonhematopoietic Nrf2 dominantly impedes adult progression of sickle cell anemia in mice.Haptoglobin Preserves Vascular Nitric Oxide Signaling during Hemolysis Compartmentalization Is Key in Limiting Nitric Oxide Scavenging by Cell-Free Hemoglobin Hematologic and hemorheological determinants of resting and exercise-induced hemoglobin oxygen desaturation in children with sickle cell disease Association between plasma free haem and incidence of vaso-occlusive episodes and acute chest syndrome in children with sickle cell disease Effect of Lead Exposure on the Status of Reticulocyte Count Indices among Workers from Lead Battery Manufacturing Plant.Plasma concentration of platelet-derived microparticles is related to painful vaso-occlusive phenotype severity in sickle cell anemia.Does increased red blood cell deformability raise the risk for osteonecrosis in sickle cell anemia?Mouse models of NPM1-mutated acute myeloid leukemia: biological and clinical implications.Peptide inhibitor of complement c1, a novel suppressor of classical pathway activation: mechanistic studies and clinical potential.Fetal Hemoglobin is Associated with Peripheral Oxygen Saturation in Sickle Cell Disease in Tanzania.Prothrombotic aspects of sickle cell disease.Minireview: Genetic basis of heterogeneity and severity in sickle cell disease.Emerging point-of-care technologies for sickle cell disease screening and monitoring.Microparticles in sickle cell anaemia: promise and pitfalls.B-type natriuretic peptide and plasma hemoglobin levels following transfusion of shorter-storage versus longer-storage red blood cells: Results from the TOTAL randomized trial.Association between sickle cell anemia and alpha thalassemia reveals a high prevalence of the α3.7 triplication in congolese patients than in worldwide series.Intravascular hemolysis and the pathophysiology of sickle cell disease.

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P2860

A central role for free heme in the pathogenesis of severe sepsis

The critical roles of platelet activation and reduced NO bioavailability in fatal pulmonary arterial hypertension in a murine hemolysis model

A guide to modern statistical analysis of immunological data.

Chronic hyper-hemolysis in sickle cell anemia: association of vascular complications and mortality with less frequent vasoocclusive pain

Reconstructing sickle cell disease: a data-based analysis of the "hyperhemolysis paradigm" for pulmonary hypertension from the perspective of evidence-based medicine.

Nitric oxide scavenging by red blood cell microparticles and cell-free hemoglobin as a mechanism for the red cell storage lesion.

Pulmonary hypertension and nitric oxide depletion in sickle cell disease.

Cell-free hemoglobin limits nitric oxide bioavailability in sickle-cell disease.

Dysregulated arginine metabolism, hemolysis-associated pulmonary hypertension, and mortality in sickle cell disease

Elevated tricuspid regurgitation velocity and decline in exercise capacity over 22 months of follow up in children and adolescents with sickle cell anemia.

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