Inflammation and functional iron deficiency regulate fibroblast growth factor 23 production. - Wikidata - wikimedia - TriplyDB

Inflammation and functional iron deficiency regulate fibroblast growth factor 23 production.

Inflammation and functional iron deficiency regulate fibroblast growth factor 23 production.

http://www.wikidata.org/entity/Q36863458

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Fibroblast Growth Factor-23-A Potential Uremic Toxin The systemic nature of CKD.Advanced glycation end products stimulate gene expression of fibroblast growth factor 23.High fibroblast growth factor 23 levels are associated with decreased ferritin levels and increased intravenous iron doses in hemodialysis patients.FGF23 is synthesised locally by renal tubules and activates injury-primed fibroblasts.The production of fibroblast growth factor 23 is controlled by TGF-β2.Effects of intravenous iron on fibroblast growth factor 23 (FGF23) in haemodialysis patients: a randomized controlled trial.Choice of High-Dose Intravenous Iron Preparation Determines Hypophosphatemia Risk Fibroblast growth factor 23 levels are elevated and associated with severe acute kidney injury and death following cardiac surgery Inflammation regulates fibroblast growth factor 23 production.Proteomic Biomarkers Panel: New Insights in Chronic Kidney Disease.Effect of Mineral and Bone Metabolism on Restless Legs Syndrome in Hemodialysis Patients.Is Fibroblast growth factor 23 the leading cause of increased mortality among chronic kidney disease patients? A narrative review The metabolic bone disease associated with the Hyp mutation is independent of osteoblastic HIF1α expression.Fibroblast Growth Factor 23 and Anemia in the Chronic Renal Insufficiency Cohort Study.Fibroblast Growth Factor 23: Mineral Metabolism and Beyond.Ironing out the cross talk between FGF23 and inflammation.Fibroblast growth factor 23 actions in inflammation: a key factor in CKD outcomes.Cardiac actions of fibroblast growth factor 23.Novel Faces of Fibroblast Growth Factor 23 (FGF23): Iron Deficiency, Inflammation, Insulin Resistance, Left Ventricular Hypertrophy, Proteinuria and Acute Kidney Injury.Overview of iron metabolism in health and disease.Treatment of Pediatric Chronic Kidney Disease-Mineral and Bone Disorder.C-Terminal Fibroblast Growth Factor 23, Iron Deficiency, and Mortality in Renal Transplant Recipients.Dietary factors and fibroblast growth factor-23 levels in young adults with African ancestry.Spleen contributes significantly to increased circulating levels of fibroblast growth factor 23 in response to lipopolysaccharide-induced inflammation.Genetic Ablation of Fgf23 or Klotho Does not Modulate Experimental Heart Hypertrophy Induced by Pressure Overload.Fibroblast growth factor 23 directly targets hepatocytes to promote inflammation in chronic kidney disease.Fibroblast growth factor 23: fueling the fire.Effects of dietary iron intake and chronic kidney disease on fibroblast growth factor 23 metabolism in wild-type and hepcidin knockout mice.Longitudinal FGF23 Trajectories and Mortality in Patients with CKD.Klotho Inhibits Interleukin-8 Secretion from Cystic Fibrosis Airway Epithelia.FGF23 expression in rodents is directly induced via erythropoietin after inhibition of hypoxia inducible factor proline hydroxylase.Oral iron supplementation with sodium ferrous citrate reduces the serum intact and c-terminal fibroblast growth factor 23 levels of maintenance haemodialysis patients.Genetic background influences cardiac phenotype in murine chronic kidney disease.Inflammation and elevated levels of fibroblast growth factor 23 are independent risk factors for death in chronic kidney disease.Increased Plasmodium chabaudi malaria mortality in mice with nutritional iron deficiency can be reduced by short-term adjunctive iron supplementation.Evaluation of a New Fully Automated Assay for Plasma Intact FGF23.Iron Chelation Resulting in Renal Phosphate Wasting.Continued Search for Therapies to Favorably Modify Phosphate and FGF23 Levels in CKD.Acute blood loss stimulates fibroblast growth factor 23 production.

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Inflammation and functional iron deficiency regulate fibroblast growth factor 23 production.

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Inflammation and functional ir ...... t growth factor 23 production.

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Aline Martin

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Chia Chi Sun

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Christina Spaulding

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Polypeptide GalNAc-transferase T3 and familial tumoral calcinosis. Secretion of fibroblast growth factor 23 requires O-glycosylation

Degradation of MEPE, DMP1, and release of SIBLING ASARM-peptides (minhibins): ASARM-peptide(s) are directly responsible for defective mineralization in HYP

Molecular insights into the klotho-dependent, endocrine mode of action of fibroblast growth factor 19 subfamily members

Regulation and function of the FGF23/klotho endocrine pathways

Mechanisms of anemia in CKD.

Effect of ascorbate on the activity of hypoxia-inducible factor in cancer cells

Mutations in GALNT3, encoding a protein involved in O-linked glycosylation, cause familial tumoral calcinosis

A mouse model of anemia of inflammation: complex pathogenesis with partial dependence on hepcidin

Elevated fibroblast growth factor 23 is a risk factor for kidney transplant loss and mortality.

Interleukin 1 induces hypoxia-inducible factor 1 in human gingival and synovial fibroblasts

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