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A novel pathway of chronic allograft rejection mediated by NK cells and alloantibody

A novel pathway of chronic allograft rejection mediated by NK cells and alloantibody

http://www.wikidata.org/entity/Q36887068

about

Transplantation tolerance and its outcome during infections and inflammation The perfect storm: HLA antibodies, complement, FcγRs, and endothelium in transplant rejection Role of complement and NK cells in antibody mediated rejection MHC class I signaling: new functional perspectives for an old molecule Effector mechanisms of rejection Monocyte recruitment by HLA IgG-activated endothelium: the relationship between IgG subclass and FcγRIIa polymorphisms.Immunosenescence in renal transplantation: a changing balance of innate and adaptive immunity Immune-mediated vascular injury and dysfunction in transplant arteriosclerosis The Complement System and Antibody-Mediated Transplant Rejection The Humoral Theory of Transplantation: Epitope Analysis and the Pathogenicity of HLA Antibodies.Pros and cons for C4d as a biomarker.Defunctioning polymorphism in the immunoglobulin G inhibitory receptor (FcγRIIB-T/T232) does not impact on kidney transplant or recipient survival.Detection of C3d-binding donor-specific anti-HLA antibodies at diagnosis of humoral rejection predicts renal graft loss.3-dimensional digital reconstruction of the murine coronary system for the evaluation of chronic allograft vasculopathy Class II alloantibody and mortality in simultaneous liver-kidney transplantation.Recent advances in renal transplantation: antibody-mediated rejection takes center stage Capillary C4d and Kidney Allograft Outcome in Relation to Morphologic Lesions Suggestive of Antibody-Mediated Rejection Antibody-Dependent NK Cell Activation Is Associated with Late Kidney Allograft Dysfunction and the Complement-Independent Alloreactive Potential of Donor-Specific Antibodies Antibody-mediated rejection of single class I MHC-disparate cardiac allografts.HLA class I: an unexpected role in integrin β4 signaling in endothelial cells Non-Complement-Binding De Novo Donor-Specific Anti-HLA Antibodies and Kidney Allograft Survival.Ganciclovir prophylaxis improves late murine cytomegalovirus-induced renal allograft damage.Ischemia-reperfusion injury accelerates human antibody-mediated transplant vasculopathy Chronic alloantibody mediated rejection.HLA class I antibody-mediated endothelial and smooth muscle cell activation.Alloantibody and complement promote T cell-mediated cardiac allograft vasculopathy through noncanonical nuclear factor-κB signaling in endothelial cells.Minimizing the risk of allo-sensitization to optimize the benefit of allogeneic cardiac-derived stem/progenitor cells.Impact of donor-specific antibodies on the outcomes of kidney graft: Pathophysiology, clinical, therapy.Antibodies to HLA Molecules Mimic Agonistic Stimulation to Trigger Vascular Cell Changes and Induce Allograft Injury Clinical role of the renal transplant biopsy.The impact of donor-specific anti-HLA antibodies on late kidney allograft failure.Improving survival during heart transplantation: diagnosis of antibody-mediated rejection and techniques for the prevention of graft injury.Transplant tolerance: new insights and strategies for long-term allograft acceptance.Lymphodepletional strategies in transplantation.Innate immune cells in transplantation.Antibody-mediated graft injury: complement-dependent and complement-independent mechanisms.NK cells after transplantation: friend or foe.Interacting mechanisms in the pathogenesis of cardiac allograft vasculopathy.Evolving perspectives of mTOR complexes in immunity and transplantation.Achieving incompatible transplantation through desensitization: current perspectives and future directions.

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A novel pathway of chronic allograft rejection mediated by NK cells and alloantibody

http://www.wikidata.org/entity/Q36887068

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

@zh-cn

name

A novel pathway of chronic allograft rejection mediated by NK cells and alloantibody

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type

label

A novel pathway of chronic allograft rejection mediated by NK cells and alloantibody

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prefLabel

A novel pathway of chronic allograft rejection mediated by NK cells and alloantibody

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J.1600-6143.2011.03836.X

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American Journal of Transplantation

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A novel pathway of chronic allograft rejection mediated by NK cells and alloantibody

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P2093

A Alessandrini

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C M Chase

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D Sebastian

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J C Madsen

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P Della Pelle

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P S Russell

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R B Colvin

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T Hirohashi

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P2860

MLK3 is required for mitogen activation of B-Raf, ERK and cell proliferation

Contemporary concepts in prevention and treatment of cardiac allograft vasculopathy.

TGF-beta utilizes SMAD3 to inhibit CD16-mediated IFN-gamma production and antibody-dependent cellular cytotoxicity in human NK cells

Effect of antibodies on endothelium.

Complement independent antibody-mediated endarteritis and transplant arteriopathy in mice.

T-cell depletion eliminates the development of cardiac allograft vasculopathy in mice rendered tolerant by the induction of mixed chimerism.

Anti-HLA class I antibodies activate endothelial cells and promote chronic rejection.

Antibody-mediated renal allograft rejection: diagnosis and pathogenesis.

Frontiers in nephrology: the varied faces of natural killer cells in transplantation--contributions to both allograft immunity and tolerance.

Viral infection induces de novo lesions of coronary allograft vasculopathy through a natural killer cell-dependent pathway.

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