Hox-C9 activates the intrinsic pathway of apoptosis and is associated with spontaneous regression in neuroblastoma
about
Therapeutic targeting of the MYC signal by inhibition of histone chaperone FACT in neuroblastoma.The pre-rRNA processing factor DEF is rate limiting for the pathogenesis of MYCN-driven neuroblastoma.Histone deacetylase 2 and N-Myc reduce p53 protein phosphorylation at serine 46 by repressing gene transcription of tumor protein 53-induced nuclear protein 1FOXP1 inhibits cell growth and attenuates tumorigenicity of neuroblastoma.HOXC9 directly regulates distinct sets of genes to coordinate diverse cellular processes during neuronal differentiationNucleolar protein PES1 is a marker of neuroblastoma outcome and is associated with neuroblastoma differentiation.MYCN amplification confers enhanced folate dependence and methotrexate sensitivity in neuroblastoma.Exosome-mediated transfer of microRNAs within the tumor microenvironment and neuroblastoma resistance to chemotherapy.Augmentor α and β (FAM150) are ligands of the receptor tyrosine kinases ALK and LTK: Hierarchy and specificity of ligand-receptor interactionsMYC Disrupts the Circadian Clock and Metabolism in Cancer Cells.Genome-wide methylation profiling identifies novel methylated genes in neuroblastoma tumorsHomeobox C9 suppresses Beclin1-mediated autophagy in glioblastoma by directly inhibiting the transcription of death-associated protein kinase 1.Transmembrane adaptor protein PAG1 is a novel tumor suppressor in neuroblastoma.Multiple components of the spliceosome regulate Mcl1 activity in neuroblastoma.MYCN promotes neuroblastoma malignancy by establishing a regulatory circuit with transcription factor AP4.microRNA-449a functions as a tumor suppressor in neuroblastoma through inducing cell differentiation and cell cycle arrest.Effects of a novel long noncoding RNA, lncUSMycN, on N-Myc expression and neuroblastoma progression.Upregulation of LYAR induces neuroblastoma cell proliferation and survival.Retinoic acid and TGF-β signalling cooperate to overcome MYCN-induced retinoid resistance.Transcription factor activating protein 2 beta (TFAP2B) mediates noradrenergic neuronal differentiation in neuroblastomaTranslational compensation of genomic instability in neuroblastomaMutational dynamics between primary and relapse neuroblastomas.Thymosin-β4 is a determinant of drug sensitivity for Fenretinide and Vorinostat combination therapy in neuroblastoma.Impact of Disseminated Neuroblastoma Cells on the Identification of the Relapse-Seeding CloneN-Myc-induced up-regulation of TRPM6/TRPM7 channels promotes neuroblastoma cell proliferation.Regulation of myeloid cells by activated T cells determines the efficacy of PD-1 blockade.Achaete-Scute Homolog 1 Expression Controls Cellular Differentiation of Neuroblastoma.Transcriptional Profiling Reveals a Common Metabolic Program in High-Risk Human Neuroblastoma and Mouse Neuroblastoma Sphere-Forming Cells.EYA1 expression and subcellular localization in neuroblastoma and its association with prognostic markersProtein arginine methyltransferase 1 is a novel regulator of MYCN in neuroblastoma.Wnt signalling is a bi-directional vulnerability of cancer cells.Integrative Analysis of Transcription Factor Combinatorial Interactions Using a Bayesian Tensor Factorization Approach.Neuroblastoma cells undergo transcriptomic alterations upon dissemination into the bone marrow and subsequent tumor progression.Elevated TIMP-1 expression is associated with a prometastatic phenotype, disease relapse, and poor survival in neuroblastoma.Combinatorial action of Grainyhead, Extradenticle and Notch in regulating Hox mediated apoptosis in Drosophila larval CNS.Neural crest derived progenitor cells contribute to tumor stroma and aggressiveness in stage 4/M neuroblastoma.FTY-720 induces apoptosis in neuroblastoma via multiple signaling pathways.Identification of GPC2 as an Oncoprotein and Candidate Immunotherapeutic Target in High-Risk Neuroblastoma.Cell death-based treatment of neuroblastoma.Combined epigenetic and differentiation-based treatment inhibits neuroblastoma tumor growth and links HIF2α to tumor suppression.
P2860
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P2860
Hox-C9 activates the intrinsic pathway of apoptosis and is associated with spontaneous regression in neuroblastoma
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Hox-C9 activates the intrinsic ...... us regression in neuroblastoma
@en
type
label
Hox-C9 activates the intrinsic ...... us regression in neuroblastoma
@en
prefLabel
Hox-C9 activates the intrinsic ...... us regression in neuroblastoma
@en
P2093
P2860
P356
P1476
Hox-C9 activates the intrinsic ...... us regression in neuroblastoma
@en
P2093
A Oberthuer
F Berthold
F Westermann
J Theissen
P2860
P356
10.1038/CDDIS.2013.84
P577
2013-04-11T00:00:00Z