Innate immune adaptor MyD88 mediates neutrophil recruitment and myocardial injury after ischemia-reperfusion in mice.
about
Heart-resident CCR2+ macrophages promote neutrophil extravasation through TLR9/MyD88/CXCL5 signaling.Epithelial cell-specific MyD88 signaling mediates ischemia/reperfusion-induced intestinal injury independent of microbial status.Innate immunity mediates myocardial preconditioning through Toll-like receptor 2 and TIRAP-dependent signaling pathwaysHuman myocardium releases heat shock protein 27 (HSP27) after global ischemia: the proinflammatory effect of extracellular HSP27 through toll-like receptor (TLR)-2 and TLR4Bone marrow MyD88 signaling modulates neutrophil function and ischemic myocardial injuryHelicobacter infection alters MyD88 and Trif signalling in response to intestinal ischaemia-reperfusion.Role of cardiac- and myeloid-MyD88 signaling in endotoxin shock: a study with tissue-specific deletion models.Toll-like receptor 4 signaling confers cardiac protection against ischemic injury via inducible nitric oxide synthase- and soluble guanylate cyclase-dependent mechanisms.MyD88 is a critical regulator of hematopoietic cell-mediated neuroprotection seen after stroke.Cutting edge: IL-1α is a crucial danger signal triggering acute myocardial inflammation during myocardial infarction.The combination of red palm oil and rooibos show anti-inflammatory effects in rats.Rho-kinase activation in leukocytes plays a pivotal role in myocardial ischemia/reperfusion injury.MyD88 and Trif signaling play distinct roles in cardiac dysfunction and mortality during endotoxin shock and polymicrobial sepsis.Myocardial ischemia activates an injurious innate immune signaling via cardiac heat shock protein 60 and Toll-like receptor 4.Nonhematopoietic toll-like receptor 2 contributes to neutrophil and cardiac function impairment during polymicrobial sepsisToll-like receptors and myocardial inflammation.Cardioprotective Effect of Licochalcone D against Myocardial Ischemia/Reperfusion Injury in Langendorff-Perfused Rat Hearts.Inhalation of carbon monoxide reduces skeletal muscle injury after hind limb ischemia-reperfusion injury in mice.Activation of toll-like receptor 4 is necessary for trauma hemorrhagic shock-induced gut injury and polymorphonuclear neutrophil primingCardiac RNA induces inflammatory responses in cardiomyocytes and immune cells via Toll-like receptor 7 signalingThe role of Interleukin Receptor Associated Kinase (IRAK)-M in regulation of myofibroblast phenotype in vitro, and in an experimental model of non-reperfused myocardial infarction.TLR2 was overexpressed independent of IL-6 in patients with valvular atrial fibrillation.Toll-like receptor signaling: a critical modulator of cell survival and ischemic injury in the heartMyocardial TLR4 is a determinant of neutrophil infiltration after global myocardial ischemia: mediating KC and MCP-1 expression induced by extracellular HSC70.Role of extracellular RNA and TLR3-Trif signaling in myocardial ischemia-reperfusion injury.Transcriptomic effects of adenosine 2A receptor deletion in healthy and endotoxemic murine myocardium.Toll-like receptor 2 plays a critical role in cardiac dysfunction during polymicrobial sepsis.Toll-like receptors: a novel target for therapeutic intervention in intestinal and hepatic ischemia-reperfusion injury?Innate immunity in the adult mammalian heart: for whom the cell tolls.Therapeutic targeting of innate immunity in the failing heart.Toll-like receptors: new players in myocardial ischemia/reperfusion injury.The emerging role of innate immunity in the heart and vascular system: for whom the cell tolls.The innate immune response in reperfused myocardium.Toll-like receptor signaling pathways in cardiovascular diseases: challenges and opportunities.Extracellular MicroRNAs Induce Potent Innate Immune Responses via TLR7/MyD88-Dependent Mechanisms.Inflammation - Cause or Consequence of Heart Failure or Both?The Biological Basis for Cardiac Repair After Myocardial Infarction: From Inflammation to Fibrosis.Exosomes as critical agents of cardiac regeneration triggered by cell therapy.Effect of dexmedetomidine on lung ischemia‑reperfusion injury.Genomics in cardiovascular diseases: analysis of the importance of the toll-like receptor signaling pathway
P2860
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P2860
Innate immune adaptor MyD88 mediates neutrophil recruitment and myocardial injury after ischemia-reperfusion in mice.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Innate immune adaptor MyD88 me ...... ischemia-reperfusion in mice.
@en
type
label
Innate immune adaptor MyD88 me ...... ischemia-reperfusion in mice.
@en
prefLabel
Innate immune adaptor MyD88 me ...... ischemia-reperfusion in mice.
@en
P2093
P2860
P1476
Innate immune adaptor MyD88 me ...... ischemia-reperfusion in mice.
@en
P2093
Emmanuel S Buys
Helene Thibault
Huailong Zhao
Jean C Bopassa
Marielle Scherrer-Crosbie
Michael J Raher
Ulrich Schmidt
P2860
P304
H1311-H1318
P356
10.1152/AJPHEART.00119.2008
P577
2008-07-25T00:00:00Z