Caveolin-1-dependent infectious entry of human papillomavirus type 31 in human keratinocytes proceeds to the endosomal pathway for pH-dependent uncoating.
about
Two highly conserved cysteine residues in HPV16 L2 form an intramolecular disulfide bond and are critical for infectivity in human keratinocytesHuman papillomavirus infection is inhibited by host autophagy in primary human keratinocytesThe cytoskeleton in papillomavirus infectionConcepts of papillomavirus entry into host cellsEntry of human papillomavirus type 16 by actin-dependent, clathrin- and lipid raft-independent endocytosisTarget cell cyclophilins facilitate human papillomavirus type 16 infectionDirect binding of retromer to human papillomavirus type 16 minor capsid protein L2 mediates endosome exit during viral infectionExosomes surf on filopodia to enter cells at endocytic hot spots, traffic within endosomes, and are targeted to the ERTranslocation of the papillomavirus L2/vDNA complex across the limiting membrane requires the onset of mitosisA clathrin independent macropinocytosis-like entry mechanism used by bluetongue virus-1 during infection of BHK cells.Cellular receptor binding and entry of human papillomavirus.Intracellular fate of strains of Escherichia coli isolated from dairy cows with acute or chronic mastitis.Essential roles for soluble virion-associated heparan sulfonated proteoglycans and growth factors in human papillomavirus infections.Papillomavirus infection requires gamma secretase.Vesicular trafficking of incoming human papillomavirus 16 to the Golgi apparatus and endoplasmic reticulum requires γ-secretase activity.Targeting human dendritic cells via DEC-205 using PLGA nanoparticles leads to enhanced cross-presentation of a melanoma-associated antigenThe HPV16 and MusPV1 papillomaviruses initially interact with distinct host components on the basement membraneThe high risk HPV16 L2 minor capsid protein has multiple transport signals that mediate its nucleocytoplasmic traffic.Human papillomavirus L2 facilitates viral escape from late endosomes via sorting nexin 17.Kallikrein-8 Proteolytically Processes Human Papillomaviruses in the Extracellular Space To Facilitate Entry into Host CellsOpposing effects of bacitracin on human papillomavirus type 16 infection: enhancement of binding and entry and inhibition of endosomal penetrationA Dual Role for the Nonreceptor Tyrosine Kinase Pyk2 during the Intracellular Trafficking of Human Papillomavirus 16.Cyclophilins facilitate dissociation of the human papillomavirus type 16 capsid protein L1 from the L2/DNA complex following virus entry.Cleavage of the HPV16 Minor Capsid Protein L2 during Virion Morphogenesis Ablates the Requirement for Cellular Furin during De Novo Infection.Recent developments in the interactions between caveolin and pathogens.Identification of a role for the trans-Golgi network in human papillomavirus 16 pseudovirus infection.Genome-wide siRNA screen identifies the retromer as a cellular entry factor for human papillomavirus.Human papillomavirus types 16, 18, and 31 share similar endocytic requirements for entry.Early events during BK virus entry and disassembly.Multiple heparan sulfate binding site engagements are required for the infectious entry of human papillomavirus type 16.Human papillomavirus molecular biology and disease association.Identification of TRAPPC8 as a host factor required for human papillomavirus cell entryViral entry mechanisms: human papillomavirus and a long journey from extracellular matrix to the nucleus.The tetraspanin CD151 in papillomavirus infection.Inhibition by cellular vacuolar ATPase impairs human papillomavirus uncoating and infectionCrosslinked multilamellar liposomes for controlled delivery of anticancer drugsHost-cell factors involved in papillomavirus entry.Principles of polyoma- and papillomavirus uncoating.Metabolism and function of hepatitis B virus cccDNA: Implications for the development of cccDNA-targeting antiviral therapeutics.Cruising the cellular highways: How human papillomavirus travels from the surface to the nucleus.
P2860
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P2860
Caveolin-1-dependent infectious entry of human papillomavirus type 31 in human keratinocytes proceeds to the endosomal pathway for pH-dependent uncoating.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Caveolin-1-dependent infectiou ...... ay for pH-dependent uncoating.
@en
type
label
Caveolin-1-dependent infectiou ...... ay for pH-dependent uncoating.
@en
prefLabel
Caveolin-1-dependent infectiou ...... ay for pH-dependent uncoating.
@en
P2860
P356
P1433
P1476
Caveolin-1-dependent infectiou ...... ay for pH-dependent uncoating.
@en
P2093
Angela Wandinger-Ness
Jessica L Smith
P2860
P304
P356
10.1128/JVI.01014-08
P407
P577
2008-07-30T00:00:00Z