MicroRNA-21 promotes fibrosis of the kidney by silencing metabolic pathways - Wikidata - wikimedia - TriplyDB

MicroRNA-21 promotes fibrosis of the kidney by silencing metabolic pathways

MicroRNA-21 promotes fibrosis of the kidney by silencing metabolic pathways

http://www.wikidata.org/entity/Q36901558

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Key fibrogenic mediators: old players. Renin–angiotensin system Alteration of Fatty Acid Oxidation in Tubular Epithelial Cells: From Acute Kidney Injury to Renal Fibrogenesis Recent advances in understanding of chronic kidney disease TGF-β/Smad signaling in renal fibrosis Emerging role of microRNAs in lipid metabolism MicroRNAs in the pathogenesis of cystic kidney disease MicroRNAs as potential therapeutic targets in kidney disease Using microRNA as an alternative treatment for hyperlipidemia and cardiovascular disease: cardio-miRs in the pipeline MicroRNAs in fibrosis: opportunities and challenges TGF-β1/Smads and miR-21 in Renal Fibrosis and Inflammation MicroRNAs in acute kidney injury Stop chronic kidney disease progression: Time is approaching Turning 21: Induction of miR-21 as a Key Switch in the Inflammatory Response Mini-review: emerging roles of microRNAs in the pathophysiology of renal diseases Anti-miRs competitively inhibit microRNAs in Argonaute complexes Peroxisomes and Kidney Injury Circulating microRNAs are not eliminated by hemodialysis TGFβ-stimulated microRNA-21 utilizes PTEN to orchestrate AKT/mTORC1 signaling for mesangial cell hypertrophy and matrix expansion Systems biology combining human- and animal-data miRNA and mRNA data identifies new targets in ureteropelvic junction obstruction The proximal tubule is the primary target of injury and progression of kidney disease: role of the glomerulotubular junction.Anti-microRNA-21 oligonucleotides prevent Alport nephropathy progression by stimulating metabolic pathways MiR-21/Smad 7 signaling determines TGF-β1-induced CAF formation MiRNAs in kidney transplantation: potential role as new biomarkers.MicroRNA-21 in pancreatic ductal adenocarcinoma tumor-associated fibroblasts promotes metastasis Silencing SMOC2 ameliorates kidney fibrosis by inhibiting fibroblast to myofibroblast transformation European contribution to the study of ROS: A summary of the findings and prospects for the future from the COST action BM1203 (EU-ROS).MicroRNAs are potential therapeutic targets in fibrosing kidney disease: lessons from animal models.Evaluation of microRNAs miR-196a, miR-30a-5P, and miR-490 as biomarkers of disease activity among patients with FSGS.Defining the acute kidney injury and repair transcriptome The role of microRNAs in diabetic nephropathy.The myriad essential roles of microRNAs in cardiovascular homeostasis and disease.Parallel analysis of mRNA and microRNA microarray profiles to explore functional regulatory patterns in polycystic kidney disease: using PKD/Mhm rat model.Increased circulating miR-21 levels are associated with kidney fibrosis.MicroRNAs and their applications in kidney diseases miR-21 in ischemia/reperfusion injury: a double-edged sword?Identification of the endosomal sorting complex required for transport-I (ESCRT-I) as an important modulator of anti-miR uptake by cancer cells.MicroRNAs in renal fibrosis.Transition of mesothelial cell to fibroblast in peritoneal dialysis: EMT, stem cell or bystander?MicroRNA profiling of pericardial fluid samples from patients with heart failure MicroRNA-21 in glomerular injury.

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MicroRNA-21 promotes fibrosis of the kidney by silencing metabolic pathways

http://www.wikidata.org/entity/Q36901558

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2012年论文

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name

MicroRNA-21 promotes fibrosis of the kidney by silencing metabolic pathways

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type

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MicroRNA-21 promotes fibrosis of the kidney by silencing metabolic pathways

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MicroRNA-21 promotes fibrosis of the kidney by silencing metabolic pathways

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SCITRANSLMED.3003205

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Science Translational Medicine

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MicroRNA-21 promotes fibrosis of the kidney by silencing metabolic pathways

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Aaron N Chang

http://www.w3.org/2001/XMLSchema#string

Aarti Kalra

http://www.w3.org/2001/XMLSchema#string

Ana P Castano

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B Nelson Chau

http://www.w3.org/2001/XMLSchema#string

Cuiyan Xin

http://www.w3.org/2001/XMLSchema#string

Deidre A MacKenna

http://www.w3.org/2001/XMLSchema#string

Didier Portilla

http://www.w3.org/2001/XMLSchema#string

Geoffrey Linn

http://www.w3.org/2001/XMLSchema#string

Jeremy S Duffield

http://www.w3.org/2001/XMLSchema#string

Jian Li

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P2860

MicroRNA-21 targets peroxisome proliferators-activated receptor-alpha in an autoregulatory loop to modulate flow-induced endothelial inflammation

MPV17 encodes an inner mitochondrial membrane protein and is mutated in infantile hepatic mitochondrial DNA depletion

Human Mpv17-like protein is localized in peroxisomes and regulates expression of antioxidant enzymes

MicroRNA 21 promotes glioma invasion by targeting matrix metalloproteinase regulators

Reduced expression of vacuole membrane protein 1 affects the invasion capacity of tumor cells

Sorting out the roles of PPAR alpha in energy metabolism and vascular homeostasis

Stress-dependent cardiac remodeling occurs in the absence of microRNA-21 in mice

Targeted disruption of the alpha isoform of the peroxisome proliferator-activated receptor gene in mice results in abolishment of the pleiotropic effects of peroxisome proliferators

Detecting microRNA binding and siRNA off-target effects from expression data

Stage-specific action of matrix metalloproteinases influences progressive hereditary kidney disease.

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121

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4

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