Nonviable mutants of simian virus 40 with deletions near the 3' end of gene A define a function for large T antigen required after onset of viral DNA replication.
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Association of insulin receptor substrate 1 with simian virus 40 large T antigenp53 targets simian virus 40 large T antigen for acetylation by CBPTransactivation of a ribosomal gene by simian virus 40 large-T antigen requires at least three activities of the protein.Suppression of the translation defect phenotype specific for a virus-associated RNA-deficient adenovirus mutant in monkey cells by simian virus 40.Effect of the molecular nature of mutation on the efficiency of intrachromosomal gene conversion in mouse cellsRemoval of a small C-terminal region of JCV and SV40 large T antigens has differential effects on transformationEmergent human pathogen simian virus 40 and its role in cancer.Mapping the transcriptional transactivation function of simian virus 40 large T antigenMechanisms of interference with simian virus 40 (SV40) DNA replication by trans-dominant mutants of SV40 large T antigen.A DNA replication-positive mutant of simian virus 40 that is defective for transformation and the production of infectious virions.The growth of simian virus 40 (SV40) host range/adenovirus helper function mutants in an African green monkey cell line that constitutively expresses the SV40 agnoprotein.Large T-antigen mutants define multiple steps in the initiation of simian virus 40 DNA replicationLinker insertion mutants of simian virus 40 large T antigen that show trans-dominant interference with wild-type large T antigen map to multiple sites within the T-antigen gene.Simian virus 40 host range/helper function mutations cause multiple defects in viral late gene expression.Properties of the simian virus 40 (SV40) large T antigens encoded by SV40 mutants with deletions in gene A.Critical spatial requirement within the origin of simian virus 40 DNA replication.Two separable functional domains of simian virus 40 large T antigen: carboxyl-terminal region of simian virus 40 large T antigen is required for efficient capsid protein synthesis.Mutations near the carboxyl terminus of the simian virus 40 large tumor antigen alter viral host rangeAbsence of a structural basis for intracellular recognition and differential localization of nuclear and plasma membrane-associated forms of simian virus 40 large tumor antigen.Complementation for replication by unrelated animal viruses containing DNA genomesCarboxyl-terminal mutants of the large tumor antigen of simian virus 40: a role for the early protein late in the lytic cycle.Intracistronic complementation in the simian virus 40 A gene.Deletion of 43 amino acids in the NH2-terminal half of the large tumor antigen of simian virus 40 results in a non-karyophilic protein capable of transforming established cells.
P2860
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P2860
Nonviable mutants of simian virus 40 with deletions near the 3' end of gene A define a function for large T antigen required after onset of viral DNA replication.
description
1983 nî lūn-bûn
@nan
1983年の論文
@ja
1983年論文
@yue
1983年論文
@zh-hant
1983年論文
@zh-hk
1983年論文
@zh-mo
1983年論文
@zh-tw
1983年论文
@wuu
1983年论文
@zh
1983年论文
@zh-cn
name
Nonviable mutants of simian vi ...... nset of viral DNA replication.
@en
type
label
Nonviable mutants of simian vi ...... nset of viral DNA replication.
@en
prefLabel
Nonviable mutants of simian vi ...... nset of viral DNA replication.
@en
P2860
P1433
P1476
Nonviable mutants of simian vi ...... nset of viral DNA replication.
@en
P2093
P2860
P304
P407
P577
1983-09-01T00:00:00Z