Epstein-Barr virus with heterogeneous DNA disrupts latency
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Genome rearrangements activate the Epstein-Barr virus gene whose product disrupts latency.Identification of a novel element involved in regulation of the lytic switch BZLF1 gene promoter of Epstein-Barr virusRestricted expression of EBV latent genes and T-lymphocyte-detected membrane antigen in Burkitt's lymphoma cells.Both Epstein-Barr virus (EBV)-encoded trans-acting factors, EB1 and EB2, are required to activate transcription from an EBV early promoter.Control of Epstein-Barr virus reactivation by activated CD40 and viral latent membrane protein 1.Interferon regulatory factor 7 is negatively regulated by the Epstein-Barr virus immediate-early gene, BZLF-1Immunofluorescence microscopy and flow cytometry characterization of chemical induction of latent Epstein-Barr virus.Protein kinase C-independent activation of the Epstein-Barr virus lytic cycle.Transfection of a rearranged viral DNA fragment, WZhet, stably converts latent Epstein-Barr viral infection to productive infection in lymphoid cells.Direct BRLF1 binding is required for cooperative BZLF1/BRLF1 activation of the Epstein-Barr virus early promoter, BMRF1.Epithelial cell retention of transcriptionally active, P3HR-1-derived heterogeneous Epstein-Barr virus DNA with concurrent loss of parental virus.YY1 binds to and regulates cis-acting negative elements in the Epstein-Barr virus BZLF1 promoter.Characterization of the ZI domains in the Epstein-Barr virus BZLF1 gene promoter: role in phorbol ester induction.Epstein-Barr virus intrastrain recombination in oral hairy leukoplakia.DNA-binding-defective mutants of the Epstein-Barr virus lytic switch activator Zta transactivate with altered specificitiesDeletion of the UL4 gene sequence of equine herpesvirus 1 precludes the generation of defective interfering particlesSecond-site homologous recombination in Epstein-Barr virus: insertion of type 1 EBNA 3 genes in place of type 2 has no effect on in vitro infection.Characterization of the Epstein-Barr virus BZLF1 protein transactivation domain.The Epstein-Barr virus (EBV) DR enhancer contains two functionally different domains: domain A is constitutive and cell specific, domain B is transactivated by the EBV early protein R.Expression of the BZLF1 latency-disrupting gene differs in standard and defective Epstein-Barr viruses.Defective viral DNA in Epstein-Barr virus-associated oral hairy leukoplakia.The zta transactivator involved in induction of lytic cycle gene expression in Epstein-Barr virus-infected lymphocytes binds to both AP-1 and ZRE sites in target promoter and enhancer regions.Identification of phorbol ester response elements in the promoter of Epstein-Barr virus putative lytic switch gene BZLF1In vitro transcriptional activation, dimerization, and DNA-binding specificity of the Epstein-Barr virus Zta protein.The Epstein-Barr virus (EBV) ORI1yt enhancer is not B-cell specific and does not respond synergistically to the EBV transcription factors R and Z.The Epstein-Barr virus Zta transactivator: a member of the bZIP family with unique DNA-binding specificity and a dimerization domain that lacks the characteristic heptad leucine zipper motif.Efficient transcription of the Epstein-Barr virus immediate-early BZLF1 and BRLF1 genes requires protein synthesis.Responsiveness of the Epstein-Barr virus NotI repeat promoter to the Z transactivator is mediated in a cell-type-specific manner by two independent signal regions.Errant processing and structural alterations of genomes present in a varicella-zoster virus vaccine.Mapping of genes in BamHI fragment M of Epstein-Barr virus DNA that may determine the fate of viral infection.Palindromic structure and polypeptide expression of 36 kilobase pairs of heterogeneous Epstein-Barr virus (P3HR-1) DNA.Amplification of Epstein-Barr virus (EBV) DNA by superinfection with a strain of EBV derived from nasopharyngeal carcinoma.Promiscuous trans activation of gene expression by an Epstein-Barr virus-encoded early nuclear proteinSequences of the Epstein-Barr Virus (EBV) large internal repeat form the center of a 16-kilobase-pair palindrome of EBV (P3HR-1) heterogeneous DNA.Two strains of Epstein-Barr virus (B95-8 and a P3HR-1 subclone) that lack defective genomes induce early antigen and cause abortive infection of Raji cells.P3HR-1 Epstein-Barr virus with heterogeneous DNA is an independent replicon maintained by cell-to-cell spread.Latent and viral replicative transcription in vivo from the BamHI K fragment of Epstein-Barr virus DNA.Construction and use of cDNA clones for the mapping and identification of Epstein-Barr virus early P3HR-1 mRNAs.Constitutive expression of Epstein-Barr virus-encoded RNAs and nuclear antigen during latency and after induction of Epstein-Barr virus replication.Epstein-Barr virus gene expression in P3HR1-superinfected Raji cells.
P2860
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P2860
Epstein-Barr virus with heterogeneous DNA disrupts latency
description
1984 nî lūn-bûn
@nan
1984年の論文
@ja
1984年論文
@yue
1984年論文
@zh-hant
1984年論文
@zh-hk
1984年論文
@zh-mo
1984年論文
@zh-tw
1984年论文
@wuu
1984年论文
@zh
1984年论文
@zh-cn
name
Epstein-Barr virus with heterogeneous DNA disrupts latency
@en
type
label
Epstein-Barr virus with heterogeneous DNA disrupts latency
@en
prefLabel
Epstein-Barr virus with heterogeneous DNA disrupts latency
@en
P2093
P2860
P1433
P1476
Epstein-Barr virus with heterogeneous DNA disrupts latency
@en
P2093
P2860
P304
P407
P577
1984-04-01T00:00:00Z