Mechanical and energetic properties of papillary muscle from ACTC E99K transgenic mouse models of hypertrophic cardiomyopathy. - Wikidata - wikimedia - TriplyDB

Mechanical and energetic properties of papillary muscle from ACTC E99K transgenic mouse models of hypertrophic cardiomyopathy.

Mechanical and energetic properties of papillary muscle from ACTC E99K transgenic mouse models of hypertrophic cardiomyopathy.

http://www.wikidata.org/entity/Q36923785

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Phosphorylation of the regulatory light chain of myosin in striated muscle: methodological perspectives Why Is there a Limit to the Changes in Myofilament Ca2+-Sensitivity Associated with Myopathy Causing Mutations?Investigations into the Sarcomeric Protein and Ca2+-Regulation Abnormalities Underlying Hypertrophic Cardiomyopathy in Cats (Felix catus).The dilated cardiomyopathy-causing mutation ACTC E361G in cardiac muscle myofibrils specifically abolishes modulation of Ca(2+) regulation by phosphorylation of troponin I A dilated cardiomyopathy mutation blunts adrenergic response and induces contractile dysfunction under chronic angiotensin II stress Using baculovirus/insect cell expressed recombinant actin to study the molecular pathogenesis of HCM caused by actin mutation A331P Age- and strain-related aberrant Ca2+ release is associated with sudden cardiac death in the ACTC E99K mouse model of hypertrophic cardiomyopathy.Molecular Defects in Cardiac Myofilament Ca2+-Regulation Due to Cardiomyopathy-Linked Mutations Can Be Reversed by Small Molecules Binding to Troponin.Classifying Cardiac Actin Mutations Associated With Hypertrophic Cardiomyopathy.Isogenic Pairs of hiPSC-CMs with Hypertrophic Cardiomyopathy/LVNC-Associated ACTC1 E99K Mutation Unveil Differential Functional Deficits

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Mechanical and energetic properties of papillary muscle from ACTC E99K transgenic mouse models of hypertrophic cardiomyopathy.

http://www.wikidata.org/entity/Q36923785

description

2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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2013年论文

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2013年论文

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name

Mechanical and energetic prope ...... f hypertrophic cardiomyopathy.

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Mechanical and energetic prope ...... f hypertrophic cardiomyopathy.

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Mechanical and energetic prope ...... f hypertrophic cardiomyopathy.

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AJPHEART.00951.2012

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American Journal of Physiology - Heart and Circulatory Physiology

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Mechanical and energetic prope ...... f hypertrophic cardiomyopathy.

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Christina Rowlands

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Kenneth MacLeod

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Mavin N Kashyap

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Michael A Ferenczi

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Nancy A Curtin

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Petr G Vikhorev

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Roger C Woledge

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Steven Marston

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Weihua Song

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P2860

Functional consequences of the mutations in human cardiac troponin I gene found in familial hypertrophic cardiomyopathy

Hypertrophic cardiomyopathy: distribution of disease genes, spectrum of mutations, and implications for a molecular diagnosis strategy

The genetic basis for cardiomyopathy: from mutation identification to mechanistic paradigms

Increased myofilament Ca2+ sensitivity and diastolic dysfunction as early consequences of Mybpc3 mutation in heterozygous knock-in mice

Contemporary definitions and classification of the cardiomyopathies: an American Heart Association Scientific Statement from the Council on Clinical Cardiology, Heart Failure and Transplantation Committee; Quality of Care and Outcomes Research and F

Thin filament mutations: developing an integrative approach to a complex disorder

Altered cardiac excitation-contraction coupling in mutant mice with familial hypertrophic cardiomyopathy

The Genetic Basis for Cardiomyopathy

Decreased energetics in murine hearts bearing the R92Q mutation in cardiac troponin T

Overexpression of the Na(+)/Ca(2+) exchanger and inhibition of the sarcoplasmic reticulum Ca(2+)-ATPase in ventricular myocytes from transgenic mice.

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