Involvement of functional protein kinase C in the mitogenic response to the H-ras oncogene product.
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Activation of extracellular signal-regulated kinase, ERK2, by p21ras oncoproteinIncreased expression of the Ras suppressor Rsu-1 enhances Erk-2 activation and inhibits Jun kinase activationRas oncogenes: split personalitiesDeregulated expression of human c-jun transforms primary rat embryo cells in cooperation with an activated c-Ha-ras gene and transforms rat-1a cells as a single gene.Ras-mediated cell cycle arrest is altered by nuclear oncogenes to induce Schwann cell transformation.Transformation stimulates glucose transporter gene expression in the absence of protein kinase CIdentification of an intracellular protein that specifically interacts with photoaffinity-labeled oncogenic p21 protein.Alterations in epidermal biochemistry as a consequence of stage-specific genetic changes in skin carcinogenesis.Molecular signal integration. Interplay between serine, threonine, and tyrosine phosphorylationUpstream CREs participate in the basal activity of minute virus of mice promoter P4 and in its stimulation in ras-transformed cells.Regulation of protein kinase C activity in neuronal differentiation induced by the N-ras oncogene in PC-12 cellsTransfected human beta-polymerase promoter contains a ras-responsive elementModulation of maturation and ribosomal protein S6 phosphorylation in Xenopus oocytes by microinjection of oncogenic ras protein and protein kinase C.Effect of a dominant inhibitory Ha-ras mutation on mitogenic signal transduction in NIH 3T3 cells.Effect of a dominant inhibitory Ha-ras mutation on neuronal differentiation of PC12 cells.Antisense-fos RNA causes partial reversion of the transformed phenotypes induced by the c-Ha-ras oncogene.Oncogene N-ras mediates selective inhibition of c-fos induction by nerve growth factor and basic fibroblast growth factor in a PC12 cell lineCells that overproduce protein kinase C are more susceptible to transformation by an activated H-ras oncogene.Induction of a mitogen-responsive gene after expression of the Ha-ras oncogene in NIH 3T3 fibroblastsNovel revertants of H-ras oncogene-transformed R6-PKC3 cellsEvidence that the ras oncogene-encoded p21 protein induces oocyte maturation via activation of protein kinase C.GTP-binding proteins and potassium channels involved in synaptic plasticity and learning.The nuclear protooncogenes c-jun and c-fos as regulators of DNA replication.Metabolic effects of signal transduction inhibition in cancer assessed by magnetic resonance spectroscopy.ras-induced c-fos expression and proliferation in living rat fibroblasts involves C-kinase activation and the serum response element pathway.Ras genes and acute myeloid leukaemia.S-Phase-specific activation of PKC alpha induces senescence in non-small cell lung cancer cells.Oncogenicity of AKR mink cell focus-inducing murine leukemia virus correlates with induction of chronic phosphatidylinositol signal transduction.p21H-ras-induced morphological transformation and increases in c-myc expression are independent of functional protein kinase C.Transcriptional activation of c-fos by oncogenic Ha-Ras in mouse mammary epithelial cells requires the combined activities of PKC-lambda, epsilon and zeta.p21ras and protein kinase C function in distinct and interdependent signaling pathways in C3H 10T1/2 fibroblasts.Multiple sources of sn-1,2-diacylglycerol in platelet-derived-growth-factor-stimulated Swiss 3T3 fibroblasts. Evidence for activation of phosphoinositidase C and phosphatidylcholine-specific phospholipase D.Phorbol ester and bryostatin differentially regulate the hydrolysis of phosphatidylethanolamine in Ha-ras- and raf-oncogene-transformed NIH 3T3 cells.The radiosensitivity of human keratinocytes: influence of activated c-H-ras oncogene expression and tumorigenicity.Expression of ras oncogene leads to down-regulation of protein kinase C.Desensitization of prostaglandin F2 alpha-stimulated inositol phosphate generation in NIH-3T3 fibroblasts transformed by overexpression of normal c-Ha-ras-1, c-Ki-ras-2 and c-N-ras genes.Potentiation of oncogenic N-ras-induced neurite outgrowth and ornithine decarboxylase activity by phorbol dibutyrate and protein kinase inhibitor H-8.Activation of phospholipase D by growth factors and oncogenes in murine fibroblasts follow alternative but cross-talking pathways.Differential effects of phorbol ester on the in vitro invasiveness of malignant and non-malignant human fibroblast cells.Lack of translocation of protein kinase C from the cytosol to the membranes in vasopressin-stimulated hepatocytes.
P2860
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P2860
Involvement of functional protein kinase C in the mitogenic response to the H-ras oncogene product.
description
1987 nî lūn-bûn
@nan
1987年の論文
@ja
1987年論文
@yue
1987年論文
@zh-hant
1987年論文
@zh-hk
1987年論文
@zh-mo
1987年論文
@zh-tw
1987年论文
@wuu
1987年论文
@zh
1987年论文
@zh-cn
name
Involvement of functional prot ...... to the H-ras oncogene product.
@en
type
label
Involvement of functional prot ...... to the H-ras oncogene product.
@en
prefLabel
Involvement of functional prot ...... to the H-ras oncogene product.
@en
P2093
P2860
P356
P1476
Involvement of functional prot ...... to the H-ras oncogene product.
@en
P2093
Aaronson SA
Blumberg PM
Fleming TP
P2860
P304
P356
10.1128/MCB.7.11.4146
P407
P577
1987-11-01T00:00:00Z