ras-transformed cells: altered levels of phosphatidylinositol-4,5-bisphosphate and catabolites.
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Ras oncogenes: split personalitiesPost-translational processing of p21ras is two-step and involves carboxyl-methylation and carboxy-terminal proteolysis.Malignant transformation by ras and other oncogenes produces common alterations in inositol phospholipid signaling pathwaysNIH-3T3 cells transformed by the EJ-ras oncogene exhibit reduced platelet-derived growth factor-mediated Ca2+ mobilization.p21ras-induced responsiveness of phosphatidylinositol turnover to bradykinin is a receptor number effect.Effects of ras-encoded proteins and platelet-derived growth factor on inositol phospholipid turnover in NRK cellsAntibody to phosphatidylinositol 4,5-bisphosphate inhibits oncogene-induced mitogenesis.Platelet-derived growth factor stimulates formation of active p21ras.GTP complex in Swiss mouse 3T3 cells.Growing and developing Dictyostelium cells express different ras genes.Deregulated expression of human c-jun transforms primary rat embryo cells in cooperation with an activated c-Ha-ras gene and transforms rat-1a cells as a single gene.Identification of a protein associated with p21ras by chemical crosslinking.Increased conversion of phosphatidylinositol to phosphatidylinositol phosphate in Dictyostelium cells expressing a mutated ras gene.Dynamic fatty acylation of p21N-ras.Ras-mediated cell cycle arrest is altered by nuclear oncogenes to induce Schwann cell transformation.Transformation stimulates glucose transporter gene expression in the absence of protein kinase CIntroduction of v-Ha-ras oncogene induces differentiation of cultured human medullary thyroid carcinoma cellsDefective HLA class II expression in a regulatory mutant is partially complemented by activated ras oncogenes.Polylysine-containing peptides, including the carboxyl-terminal segment of the human c-Ki-ras 2 protein, affect the activity of some key membrane enzymes.Loss of platelet-derived growth factor-stimulated phospholipase activity in NIH-3T3 cells expressing the EJ-ras oncogene.Opposing effects of a ras oncogene on growth factor-stimulated phosphoinositide hydrolysis: desensitization to platelet-derived growth factor and enhanced sensitivity to bradykinin.Transmembrane signals and protooncogene induction evoked by carcinogenic metals and prevented by zincAlterations in epidermal biochemistry as a consequence of stage-specific genetic changes in skin carcinogenesis.Phosphatidylinositol metabolism and polyoma-mediated transformation.Mutated c-Ha-ras oncogene alters cytokeratin expression in the human breast epithelial cell line MCF-10A.Deregulation of hamster fibroblast proliferation by mutated ras oncogenes is not mediated by constitutive activation of phosphoinositide-specific phospholipase C.Dexamethasone-dependent inhibition of differentiation of C2 myoblasts bearing steroid-inducible N-ras oncogenes.Growth factors, signaling pathways, and the regulation of proliferation and differentiation in BC3H1 muscle cells. II. Two signaling pathways distinguished by pertussis toxin and a potential role for the ras oncogene.Posttranslational membrane attachment and dynamic fatty acylation of a neuronal growth cone protein, GAP-43.Membrane-bound phosphatases in Escherichia coli: sequence of the pgpB gene and dual subcellular localization of the pgpB productT-type calcium channels in adrenal glomerulosa cells: GTP-dependent modulation by angiotensin II.Inhibition of myogenesis by the H-ras oncogene: implication of a role for protein kinase C.Mutated alpha subunit of the Gq protein induces malignant transformation in NIH 3T3 cellsRegulation of protein kinase C activity in neuronal differentiation induced by the N-ras oncogene in PC-12 cellsAlterations of G-protein coupling function in phosphoinositide signaling pathways of cells transformed by ras and other membrane-associated and cytoplasmic oncogenes.Induction of differentiation in v-Ha-ras-transformed MDCK cells by prostaglandin E2 and 8-bromo-cyclic AMP is associated with a decrease in steady-state level of inositol 1,4,5-trisphosphateModulation of maturation and ribosomal protein S6 phosphorylation in Xenopus oocytes by microinjection of oncogenic ras protein and protein kinase C.Role of phosphatidylinositide metabolism in ras-induced Xenopus oocyte maturation.Effect of a dominant inhibitory Ha-ras mutation on mitogenic signal transduction in NIH 3T3 cells.Effect of a dominant inhibitory Ha-ras mutation on neuronal differentiation of PC12 cells.Elevated phosphocholine concentration in ras-transformed NIH 3T3 cells arises from increased choline kinase activity, not from phosphatidylcholine breakdown
P2860
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P2860
ras-transformed cells: altered levels of phosphatidylinositol-4,5-bisphosphate and catabolites.
description
1986 nî lūn-bûn
@nan
1986年の論文
@ja
1986年論文
@yue
1986年論文
@zh-hant
1986年論文
@zh-hk
1986年論文
@zh-mo
1986年論文
@zh-tw
1986年论文
@wuu
1986年论文
@zh
1986年论文
@zh-cn
name
ras-transformed cells: altered ...... -bisphosphate and catabolites.
@en
ras-transformed cells: altered ...... -bisphosphate and catabolites.
@nl
type
label
ras-transformed cells: altered ...... -bisphosphate and catabolites.
@en
ras-transformed cells: altered ...... -bisphosphate and catabolites.
@nl
prefLabel
ras-transformed cells: altered ...... -bisphosphate and catabolites.
@en
ras-transformed cells: altered ...... -bisphosphate and catabolites.
@nl
P2093
P356
P1433
P1476
ras-transformed cells: altered ...... -bisphosphate and catabolites.
@en
P2093
Chahwala SB
Fleischman LF
P304
P356
10.1126/SCIENCE.3001936
P407
P577
1986-01-01T00:00:00Z