Formation of Streptococcus pneumoniae non-phase-variable colony variants is due to increased mutation frequency present under biofilm growth conditions
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Experimental evolution in biofilm populations.Phenotypic and Genotypic Characteristics of Small Colony Variants and Their Role in Chronic InfectionSubinhibitory arsenite concentrations lead to population dispersal in Thiomonas spBdlA, DipA and induced dispersion contribute to acute virulence and chronic persistence of Pseudomonas aeruginosaThe MerR-like regulator BrlR impairs Pseudomonas aeruginosa biofilm tolerance to colistin by repressing PhoPQThe MerR-like transcriptional regulator BrlR contributes to Pseudomonas aeruginosa biofilm toleranceRole of mutation in Pseudomonas aeruginosa biofilm development.Elevated levels of the second messenger c-di-GMP contribute to antimicrobial resistance of Pseudomonas aeruginosa.Early biofilm formation on microtiter plates is not correlated with the invasive disease potential of Streptococcus pneumoniae.Proteomic comparisons of opaque and transparent variants of Streptococcus pneumoniae by two dimensional-differential gel electrophoresis.Biofilm is a Major Virulence Determinant in Bacterial Colonization of Chronic Skin Ulcers Independently from the Multidrug Resistant Phenotype.Coinfection with Haemophilus influenzae promotes pneumococcal biofilm formation during experimental otitis media and impedes the progression of pneumococcal diseaseStreptococcus pneumoniae in biofilms are unable to cause invasive disease due to altered virulence determinant production.Planktonic aggregates of Staphylococcus aureus protect against common antibioticsInstability of ackA (acetate kinase) mutations and their effects on acetyl phosphate and ATP amounts in Streptococcus pneumoniae D39.Increased mutability of Staphylococci in biofilms as a consequence of oxidative stressA point mutation in cpsE renders Streptococcus pneumoniae nonencapsulated and enhances its growth, adherence and competence.Population diversification in Staphylococcus aureus biofilms may promote dissemination and persistence.The novel species Streptococcus tigurinus and its association with oral infection.SagS contributes to the motile-sessile switch and acts in concert with BfiSR to enable Pseudomonas aeruginosa biofilm formationLuxS mediates iron-dependent biofilm formation, competence, and fratricide in Streptococcus pneumoniae.The Small Molecule DAM Inhibitor, Pyrimidinedione, Disrupts Streptococcus pneumoniae Biofilm Growth In Vitro.Immunization with Pneumococcal Surface Protein K of Nonencapsulated Streptococcus pneumoniae Provides Protection in a Mouse Model of Colonization.Parallel Evolution in Streptococcus pneumoniae Biofilms.The MerR-like regulator BrlR confers biofilm tolerance by activating multidrug efflux pumps in Pseudomonas aeruginosa biofilms.Anatomical site-specific contributions of pneumococcal virulence determinants.The putative enoyl-coenzyme A hydratase DspI is required for production of the Pseudomonas aeruginosa biofilm dispersion autoinducer cis-2-decenoic acid.Antimicrobial tolerance of Pseudomonas aeruginosa biofilms is activated during an early developmental stage and requires the two-component hybrid SagS.Characterization of Streptococcus tigurinus small-colony variants causing prosthetic joint infection by comparative whole-genome analyses.Biofilm formation in Streptococcus pneumoniae.Hydrogen peroxide-dependent DNA release and transfer of antibiotic resistance genes in Streptococcus gordonii.Analysis of multidrug resistant group B streptococci with reduced penicillin susceptibility forming small, less hemolytic colonies.The novel two-component regulatory system BfiSR regulates biofilm development by controlling the small RNA rsmZ through CafAPyruvate oxidase influences the sugar utilization pattern and capsule production in Streptococcus pneumoniae.Streptococcus pneumoniae TIGR4 Phase-Locked Opacity Variants Differ in Virulence Phenotypes.Mutability in Pseudomonas viridiflava as a programmed balance between antibiotic resistance and pathogenicity.Divide and conquer: the Pseudomonas aeruginosa two-component hybrid SagS enables biofilm formation and recalcitrance of biofilm cells to antimicrobial agents via distinct regulatory circuits.Susceptibility of Pseudomonas aeruginosa Dispersed Cells to Antimicrobial Agents Is Dependent on the Dispersion Cue and Class of the Antimicrobial Agent Used.
P2860
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P2860
Formation of Streptococcus pneumoniae non-phase-variable colony variants is due to increased mutation frequency present under biofilm growth conditions
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Formation of Streptococcus pne ...... nder biofilm growth conditions
@en
type
label
Formation of Streptococcus pne ...... nder biofilm growth conditions
@en
prefLabel
Formation of Streptococcus pne ...... nder biofilm growth conditions
@en
P2860
P356
P1476
Formation of Streptococcus pne ...... nder biofilm growth conditions
@en
P2093
Karin Sauer
Magee Allegrucci
P2860
P304
P356
10.1128/JB.00707-08
P407
P577
2008-07-25T00:00:00Z