Chemotherapy-induced apoptosis in a transgenic model of neuroblastoma proceeds through p53 induction. - Wikidata - wikimedia - TriplyDB

Chemotherapy-induced apoptosis in a transgenic model of neuroblastoma proceeds through p53 induction.

Chemotherapy-induced apoptosis in a transgenic model of neuroblastoma proceeds through p53 induction.

http://www.wikidata.org/entity/Q36944205

about

Dinosaurs and ancient civilizations: reflections on the treatment of cancer Neoplasia: the second decade.Biocompatibility and reduced drug absorption of sol-gel-treated poly(dimethyl siloxane) for microfluidic cell culture applications.Preclinical models for neuroblastoma: establishing a baseline for treatment Myc proteins as therapeutic targets.miRNA expression profiling of the murine TH-MYCN neuroblastoma model reveals similarities with human tumors and identifies novel candidate miRNAs Galectin-3 impairment of MYCN-dependent apoptosis-sensitive phenotype is antagonized by nutlin-3 in neuroblastoma cells miR-380-5p represses p53 to control cellular survival and is associated with poor outcome in MYCN-amplified neuroblastoma.Tumor development, growth characteristics and spectrum of genetic aberrations in the TH-MYCN mouse model of neuroblastoma.Glial progenitors as targets for transformation in glioma.Evaluation of clinically translatable MR imaging biomarkers of therapeutic response in the TH-MYCN transgenic mouse model of neuroblastoma Exosomal miR-10a derived from amniotic fluid stem cells preserves ovarian follicles after chemotherapy.Targeting gastrin-releasing peptide as a new approach to treat aggressive refractory neuroblastomas.Targeting ALK in neuroblastoma--preclinical and clinical advancements.Mcl1 downregulation sensitizes neuroblastoma to cytotoxic chemotherapy and small molecule Bcl2-family antagonists.Neuroblastoma and MYCN Mdm2 deficiency suppresses MYCN-Driven neuroblastoma tumorigenesis in vivo The War on Cancer rages on.Apoptotic cell death in neuroblastoma.p53 family: Therapeutic targets in neuroblastoma.Percentage tumor necrosis following chemotherapy in neuroblastoma correlates with MYCN status but not survival.Genetically engineered murine models--contribution to our understanding of the genetics, molecular pathology and therapeutic targeting of neuroblastoma.Neuroblastoma models for insights into tumorigenesis and new therapies.Targeting p53-null neuroblastomas through RLIP76.p53 is a direct transcriptional target of MYCN in neuroblastoma Oncolytic adenovirus armed with shRNA targeting MYCN gene inhibits neuroblastoma cell proliferation and in vivo xenograft tumor growth.RLIP76 Inhibition: A Promising Developmental Therapy for Neuroblastoma.Direct effects of Bmi1 on p53 protein stability inactivates oncoprotein stress responses in embryonal cancer precursor cells at tumor initiation.Overexpression of centrosomal protein Nlp confers breast carcinoma resistance to paclitaxel.Influence of p53 expression on sensitivity of cancer cells to bleomycin.Downregulation of MYCN through PI3K Inhibition in Mouse Models of Pediatric Neural Cancer.p53, SKP2, and DKK3 as MYCN Target Genes and Their Potential Therapeutic Significance.Alterations of ER, PR, HER-2/neu, and P53 protein expression in ductal breast carcinomas and clinical implications.miR-10a rejuvenates aged human mesenchymal stem cells and improves heart function after myocardial infarction through KLF4.

P2860

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P2860

Chemotherapy-induced apoptosis in a transgenic model of neuroblastoma proceeds through p53 induction.

http://www.wikidata.org/entity/Q36944205

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

@zh-tw

2008年论文

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2008年论文

@zh

2008年论文

@zh-cn

name

Chemotherapy-induced apoptosis ...... roceeds through p53 induction.

@en

type

label

Chemotherapy-induced apoptosis ...... roceeds through p53 induction.

@en

prefLabel

Chemotherapy-induced apoptosis ...... roceeds through p53 induction.

@en

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Chemotherapy-induced apoptosis ...... roceeds through p53 induction.

@en

P2093

David D Goldenberg

http://www.w3.org/2001/XMLSchema#string

Grace E Kim

http://www.w3.org/2001/XMLSchema#string

Katherine K Matthay

http://www.w3.org/2001/XMLSchema#string

Kim Nguyen

http://www.w3.org/2001/XMLSchema#string

Louis Chesler

http://www.w3.org/2001/XMLSchema#string

Matt Grimmer

http://www.w3.org/2001/XMLSchema#string

Rodney Collins

http://www.w3.org/2001/XMLSchema#string

Slava Yakovenko

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Tarik Tihan

http://www.w3.org/2001/XMLSchema#string

William A Weiss

http://www.w3.org/2001/XMLSchema#string

P2860

PUMA couples the nuclear and cytoplasmic proapoptotic function of p53

Suppression of tumorigenesis by the p53 target PUMA

Puma is an essential mediator of p53-dependent and -independent apoptotic pathways

Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumours

Molecular biology of neuroblastoma.

Treatment of high-risk neuroblastoma with intensive chemotherapy, radiotherapy, autologous bone marrow transplantation, and 13-cis-retinoic acid. Children's Cancer Group.

Targeted expression of MYCN causes neuroblastoma in transgenic mice.

Aggressive childhood neuroblastomas do not express caspase-8: an important component of programmed cell death.

Differential expression of bcl2 protooncogene in neuroblastoma and other human tumor cell lines of neural origin.

p53 determines multidrug sensitivity of childhood neuroblastoma.

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1268-1274

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10.1593/NEO.08778

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11

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10

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2008-11-01T00:00:00Z

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23416675

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18953436

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2570603

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