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Neurotoxic Antibodies against the Prion Protein Do Not Trigger Prion Replication.Cystatin F is a biomarker of prion pathogenesis in mice.An In Vivo (11)C-(R)-PK11195 PET and In Vitro Pathology Study of Microglia Activation in Creutzfeldt-Jakob Disease.Prion pathogenesis is unaltered in the absence of SIRPα-mediated "don't-eat-me" signaling.Neil3 induced neurogenesis protects against prion disease during the clinical phase.Prion disease: experimental models and reality.Prion disease pathogenesis in the absence of the commensal microbiota.Oral Prion Disease Pathogenesis Is Impeded in the Specific Absence of CXCR5-Expressing Dendritic Cells.Role of the JAK/STAT signaling pathway in regulation of innate immunity in neuroinflammatory diseases.The Role of Microglia in Prion Diseases: A Paradigm of Functional Diversity.The spread of prion-like proteins by lysosomes and tunneling nanotubes: Implications for neurodegenerative diseases.Microglia in prion diseases.Activation of microglia by retroviral infection correlates with transient clearance of prions from the brain but does not change incubation time.Lessons Learned about Neurodegeneration from Microglia and Monocyte Depletion Studies.Alterations in the Interplay between Neurons, Astrocytes and Microglia in the Rat Dentate Gyrus in Experimental Models of Neurodegeneration.Plasma cholesterol level determines in vivo prion propagationLet's make microglia great again in neurodegenerative disorders.What Is Our Current Understanding of PrPSc-Associated Neurotoxicity and Its Molecular Underpinnings?Electron microscopic and confocal laser microscopy analysis of amyloid plaques in chronic wasting disease transmitted to transgenic mice.Microglia are required for protection against lethal coronavirus encephalitis in mice.Flow Cytometric Detection of PrPSc in Neurons and Glial Cells from Prion-Infected Mouse Brains.Disease-associated protein seeding suggests a dissociation between misfolded protein accumulation and neurodegeneration in prion disease.Lipopolysaccharide-Induced Microglia Activation Promotes the Survival of Midbrain Dopaminergic Neurons In Vitro.Microglia Are Critical in Host Defense Against Prion Disease.
P2860
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P2860
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
A neuroprotective role for microglia in prion diseases.
@en
type
label
A neuroprotective role for microglia in prion diseases.
@en
prefLabel
A neuroprotective role for microglia in prion diseases.
@en
P2093
P2860
P50
P356
P1476
A neuroprotective role for microglia in prion diseases.
@en
P2093
Caihong Zhu
Elisabeth J Rushing
Irina Abakumova
Jeppe Falsig
Petra Schwarz
Uli S Herrmann
P2860
P304
P356
10.1084/JEM.20151000
P407
P577
2016-05-16T00:00:00Z