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Yersinia controls type III effector delivery into host cells by modulating Rho activityRandom mutagenesis identifies a C-terminal region of YopD important for Yersinia type III secretion functionCD8(+) T cells restrict Yersinia pseudotuberculosis infection: bypass of anti-phagocytosis by targeting antigen-presenting cellsAdherence to human cells of a cryptic Haemophilus genospecies responsible for genital and neonatal infections.YopJ-promoted cytotoxicity and systemic colonization are associated with high levels of murine interleukin-18, gamma interferon, and neutrophils in a live vaccine model of Yersinia pseudotuberculosis infectionEffector CD8+ T cells are generated in response to an immunodominant epitope in type III effector YopE during primary Yersinia pseudotuberculosis infectionInfluence of PhoP and intra-species variations on virulence of Yersinia pseudotuberculosis during the natural oral infection route.Cytotoxic T-cell-mediated response against Yersinia pseudotuberculosis in HLA-B27 transgenic rat.The presence of professional phagocytes dictates the number of host cells targeted for Yop translocation during infectionDelineation of regions of the Yersinia YopM protein required for interaction with the RSK1 and PRK2 host kinases and their requirement for interleukin-10 production and virulenceA transposon site hybridization screen identifies galU and wecBC as important for survival of Yersinia pestis in murine macrophagesThe GAP activity of type III effector YopE triggers killing of Yersinia in macrophagesYersinia pseudotuberculosis produces a cytotoxic necrotizing factorIQGAP1 is important for activation of caspase-1 in macrophages and is targeted by Yersinia pestis type III effector YopM.Mathematical relationship between cytokine concentrations and pathogen levels during infection.Inhibition of the Fc receptor-mediated oxidative burst in macrophages by the Yersinia pseudotuberculosis tyrosine phosphatase.Invasin production by Yersinia pestis is abolished by insertion of an IS200-like element within the inv gene.Urease is not involved in the virulence of Yersinia pseudotuberculosis in miceType III secretion system-dependent translocation of ectopically expressed Yop effectors into macrophages by intracellular Yersinia pseudotuberculosis.Immunization with live aroA recombinant Salmonella typhimurium producing invasin inhibits intestinal translocation of Yersinia pseudotuberculosisMicrobiota-Dependent Sequelae of Acute Infection Compromise Tissue-Specific ImmunityThe ability to replicate in macrophages is conserved between Yersinia pestis and Yersinia pseudotuberculosis.Interleukin-10 induction is an important virulence function of the Yersinia pseudotuberculosis type III effector YopM.CCR2+ Inflammatory Dendritic Cells and Translocation of Antigen by Type III Secretion Are Required for the Exceptionally Large CD8+ T Cell Response to the Protective YopE69-77 Epitope during Yersinia Infection.Molecular typing of Yersinia pseudotuberculosis by using an IS200-like elementUncovering an Important Role for YopJ in the Inhibition of Caspase-1 in Activated Macrophages and Promoting Yersinia pseudotuberculosis Virulence.Caspase-1 activation in macrophages infected with Yersinia pestis KIM requires the type III secretion system effector YopJ.Type III secretion decreases bacterial and host survival following phagocytosis of Yersinia pseudotuberculosis by macrophages.Lack of antibody response to invasin in humans with yersiniosisAdhesins and host serum factors drive Yop translocation by yersinia into professional phagocytes during animal infection.Oral vaccination with LcrV from Yersinia pestis KIM delivered by live attenuated Salmonella enterica serovar Typhimurium elicits a protective immune response against challenge with Yersinia pseudotuberculosis and Yersinia enterocolitica.Cells infected with Yersinia present an epitope to class I MHC-restricted CTL.The response regulator PhoP of Yersinia pseudotuberculosis is important for replication in macrophages and for virulenceFlagellin-Mediated Protection against Intestinal Yersinia pseudotuberculosis Infection Does Not Require Interleukin-22Yersinia pseudotuberculosis uses Ail and YadA to circumvent neutrophils by directing Yop translocation during lung infection.Yops of Yersinia spp. pathogenic for humans.Yersinia pestis can bypass protective antibodies to LcrV and activation with gamma interferon to survive and induce apoptosis in murine macrophagesYersinia pestis can reside in autophagosomes and avoid xenophagy in murine macrophages by preventing vacuole acidification.The response regulator PhoP negatively regulates Yersinia pseudotuberculosis and Yersinia pestis biofilms.CCR2+ inflammatory monocytes are recruited to Yersinia pyogranulomas and dictate adaptive responses at the expense of innate immunity during oral infection.
P2860
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P2860
description
1992 nî lūn-bûn
@nan
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
1992年论文
@zh
1992年论文
@zh-cn
name
Invasin expression in Yersinia pseudotuberculosis
@en
type
label
Invasin expression in Yersinia pseudotuberculosis
@en
prefLabel
Invasin expression in Yersinia pseudotuberculosis
@en
P2860
P1476
Invasin expression in Yersinia pseudotuberculosis
@en
P2093
P2860
P304
P407
P577
1992-10-01T00:00:00Z