Influenza A virus inhibits alveolar fluid clearance in BALB/c mice
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Influenza lung injury: mechanisms and therapeutic opportunitiesTranscriptomic analysis reveals novel mechanistic insight into murine biological responses to multi-walled carbon nanotubes in lungs and cultured lung epithelial cellsInfluenza A induced cellular signal transduction pathways.Influenza A H1N1 induces declines in alveolar gas exchange in mice consistent with rapid post-infection progression from acute lung injury to ARDS.Heterozygosity for the F508del mutation in the cystic fibrosis transmembrane conductance regulator anion channel attenuates influenza severityInflammatory Responses Regulating Alveolar Ion Transport during Pulmonary Infections.P2Y6 receptors are involved in mediating the effect of inactivated avian influenza virus H5N1 on IL-6 & CXCL8 mRNA expression in respiratory epithelium.Activation of A1-adenosine receptors promotes leukocyte recruitment to the lung and attenuates acute lung injury in mice infected with influenza A/WSN/33 (H1N1) virusInfluenza causes prolonged disruption of the alveolar-capillary barrier in mice unresponsive to mesenchymal stem cell therapy.Alcohol worsens acute lung injury by inhibiting alveolar sodium transport through the adenosine A1 receptor.Postinfection A77-1726 treatment improves cardiopulmonary function in H1N1 influenza-infected miceAnimal Models for Influenza Virus Pathogenesis and Transmission.Infection of mice with influenza A/WSN/33 (H1N1) virus alters alveolar type II cell phenotype.Selective targeting of alveolar type II respiratory epithelial cells by anti-surfactant protein-C antibody-conjugated lipoplexesTGF-β-induced IL-6 prevents development of acute lung injury in influenza A virus-infected F508del CFTR-heterozygous mice.Influenza A infection enhances antigen-induced airway inflammation and hyperresponsiveness in young but not aged mice.Ecto-5'-nucleotidase CD73 modulates the innate immune response to influenza infection but is not required for development of influenza-induced acute lung injury.Role of epithelial sodium channels in the regulation of lung fluid homeostasisMacrophage-epithelial paracrine crosstalk inhibits lung edema clearance during influenza infection.Influenza matrix protein 2 alters CFTR expression and function through its ion channel activity.Chloride secretion across adult alveolar epithelial cells contributes to cardiogenic edema.Effect of ventilation pressure on alveolar fluid clearance and beta-agonist responses in mice.Influenza virus M2 protein inhibits epithelial sodium channels by increasing reactive oxygen species.Estradiol activates epithelial sodium channels in rat alveolar cells through the G protein-coupled estrogen receptor.Contribution of the Purinergic Receptor P2X7 to Development of Lung Immunopathology during Influenza Virus Infection.Acute lung injury: how macrophages orchestrate resolution of inflammation and tissue repairEndothelial activation and dysfunction in the pathogenesis of influenza A virus infectionMouse Models of Acute Respiratory Distress Syndrome: A Review of Analytical Approaches, Pathologic Features, and Common Measurements.Purinergic signalling links mechanical breath profile and alveolar mechanics with the pro-inflammatory innate immune response causing ventilation-induced lung injury.Inhibition of lectin-mediated innate host defences in vivo modulates disease severity during influenza virus infection.Cation currents in human airway epithelial cells induced by infection with influenza A virus.Local and Systemic Immune Responses to Influenza A Virus Infection in Pneumonia and Encephalitis Mouse Models.Influenza virus infection alters ion channel function of Airway and alveolar cells: Mechanisms and physiological sequelae.Lethal H1N1 influenza A virus infection alters the murine alveolar type II cell surfactant lipidome.ATP catabolism by tissue non-specific alkaline phosphatase contributes to development of ARDS in influenza-infected mice.Double-stranded RNA induces similar pulmonary dysfunction to respiratory syncytial virus in BALB/c mice.Ion channels of the lung and their role in disease pathogenesis.Listeriolysin O Causes ENaC Dysfunction in Human Airway Epithelial Cells.Stimulation of alpha2-adrenergic receptors impairs influenza virus infection.
P2860
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P2860
Influenza A virus inhibits alveolar fluid clearance in BALB/c mice
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Influenza A virus inhibits alveolar fluid clearance in BALB/c mice
@en
type
label
Influenza A virus inhibits alveolar fluid clearance in BALB/c mice
@en
prefLabel
Influenza A virus inhibits alveolar fluid clearance in BALB/c mice
@en
P2093
P2860
P1476
Influenza A virus inhibits alveolar fluid clearance in BALB/c mice
@en
P2093
Eduardo R Lazarowski
Erin N Z Yu
Ian C Davis
Joan E Durbin
Kendra E Wolk
Nancy A Jewell
Russell K Durbin
Zachary P Traylor
P2860
P304
P356
10.1164/RCCM.200803-455OC
P407
P577
2008-08-08T00:00:00Z