Controlling mutation: intervening in evolution as a therapeutic strategy.
about
Separation of recombination and SOS response in Escherichia coli RecA suggests LexA interaction sitesAtypical Role for PhoU in Mutagenic Break Repair under Stress in Escherichia coliBiology of Acinetobacter baumannii: Pathogenesis, Antibiotic Resistance Mechanisms, and Prospective Treatment OptionsInhibitors of RecA activity discovered by high-throughput screening: cell-permeable small molecules attenuate the SOS response in Escherichia coli.Mutability and importance of a hypermutable cell subpopulation that produces stress-induced mutants in Escherichia coli.Mycobacterium tuberculosis interactome analysis unravels potential pathways to drug resistance.The sigma(E) stress response is required for stress-induced mutation and amplification in Escherichia coli.Separate DNA Pol II- and Pol IV-dependent pathways of stress-induced mutation during double-strand-break repair in Escherichia coli are controlled by RpoS.The evolution of stress-induced hypermutation in asexual populations.MsDpo4-a DinB Homolog from Mycobacterium smegmatis-Is an Error-Prone DNA Polymerase That Can Promote G:T and T:G Mismatches.Mutation as a stress response and the regulation of evolvabilityAntibiotic resistance acquired through a DNA damage-inducible response in Acinetobacter baumannii.Programmed genetic instability: a tumor-permissive mechanism for maintaining the evolvability of higher species through methylation-dependent mutation of DNA repair genes in the male germ line.DinB upregulation is the sole role of the SOS response in stress-induced mutagenesis in Escherichia coli.Multiple strategies for translesion synthesis in bacteriaRole of reactive oxygen species in antibiotic action and resistance.Ecology and evolution as targets: the need for novel eco-evo drugs and strategies to fight antibiotic resistance.What limits the efficiency of double-strand break-dependent stress-induced mutation in Escherichia coli?Bacterial stress responses as determinants of antimicrobial resistance.Microbial persistence and the road to drug resistance.The Small RNA GcvB Promotes Mutagenic Break Repair by Opposing the Membrane Stress Response.An experimental evaluation of drug-induced mutational meltdown as an antiviral treatment strategy.Genome-wide stochastic adaptive DNA amplification at direct and inverted DNA repeats in the parasite LeishmaniaSpecificity determinants for autoproteolysis of LexA, a key regulator of bacterial SOS mutagenesis.Stress-Induced Mutagenesis.The Transient Multidrug Resistance Phenotype of Salmonella enterica Swarming Cells Is Abolished by Sub-inhibitory Concentrations of Antimicrobial Compounds.RNA-Mediated cis Regulation in Acinetobacter baumannii Modulates Stress-Induced Phenotypic Variation.Suramin is a potent and selective inhibitor of Mycobacterium tuberculosis RecA protein and the SOS response: RecA as a potential target for antibacterial drug discovery.DNA polymerase IV primarily operates outside of DNA replication forks in Escherichia coli.Stress-induced mutagenesis and complex adaptation.Effect of recA inactivation on mutagenesis of Escherichia coli exposed to sublethal concentrations of antimicrobials.
P2860
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P2860
Controlling mutation: intervening in evolution as a therapeutic strategy.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Controlling mutation: intervening in evolution as a therapeutic strategy.
@en
type
label
Controlling mutation: intervening in evolution as a therapeutic strategy.
@en
prefLabel
Controlling mutation: intervening in evolution as a therapeutic strategy.
@en
P2860
P1476
Controlling mutation: intervening in evolution as a therapeutic strategy.
@en
P2093
Floyd E Romesberg
Ryan T Cirz
P2860
P304
P356
10.1080/10409230701597741
P577
2007-09-01T00:00:00Z