Myeloid-Derived Suppressor Cells Endow Stem-like Qualities to Breast Cancer Cells through IL6/STAT3 and NO/NOTCH Cross-talk Signaling.
about
Inflammatory cytokine IL6 cooperates with CUDR to aggravate hepatocyte-like stem cells malignant transformation through NF-κB signalingMechanism of immune evasion in breast cancer.Myeloid-derived suppressor cells: Important contributors to tumor progression and metastasis.Tumor-associated myeloid cells as guiding forces of cancer cell stemness.Chemokines in the cancer microenvironment and their relevance in cancer immunotherapy.Targeting interlukin-6 to relieve immunosuppression in tumor microenvironment.Tumor-related interleukins: old validated targets for new anti-cancer drug development.A niche that triggers aggressiveness within BRCA1-IRIS overexpressing triple negative tumors is supported by reciprocal interactions with the microenvironment.Modeling of Interactions between Cancer Stem Cells and their Microenvironment: Predicting Clinical Response.γ-Secretase inhibitor reduces immunosuppressive cells and enhances tumour immunity in head and neck squamous cell carcinoma.Myeloid-Derived Suppressor Cells: Immune-Suppressive Cells That Impair Antitumor Immunity and Are Sculpted by Their Environment.IL6 blockade potentiates the anti-tumor effects of γ-secretase inhibitors in Notch3-expressing breast cancer.Inhibition of SRC family kinases reduces myeloid-derived suppressor cells in head and neck cancer.CXCR2+ MDSCs promote breast cancer progression by inducing EMT and activated T cell exhaustion.Notch Signaling in Myeloid Cells as a Regulator of Tumor Immune Responses.Cancer-associated fibroblasts promote stem cell-like properties of hepatocellular carcinoma cells through IL-6/STAT3/Notch signaling.Interplay between inflammatory tumor microenvironment and cancer stem cells.The impact of PI3K inhibitors on breast cancer cell and its tumor microenvironment.Cancer Cells Exploit Notch Signaling to Redefine a Supportive Cytokine Milieu
P2860
Q37408343-9B779D44-95C6-4700-8336-357C8D8376A6Q37712385-E7CF9ECF-0B8F-479D-9D6B-A9DF474431E8Q38702891-97557790-62FF-4C79-899F-575EF2F66B22Q39236676-825621C1-8ADE-48A5-A568-7D90BF96FCD9Q39337489-A61757C3-9BB4-4446-9702-17BFF04E19FFQ39390630-96C8BDD3-142C-46D9-9F79-2BA25ECA887AQ41716360-5FE4B0CD-ED42-47C0-9B99-5FB4036B5CE1Q47275348-4C0DEF6A-693C-4856-A091-09D57265A0D7Q47561430-AFB6F6DC-AFA9-4186-AAE8-9C45CCC37C91Q48164135-FE9CA843-A161-4ED8-A661-6916C980A19CQ48293607-82D53D96-8B99-4D73-9384-AC816145ECAFQ48632592-4EBB976E-5605-42CA-AE50-FA6B71479277Q48949605-2C557BA4-79C5-4ED8-A0CF-AD2B3FF61376Q49569708-933736EC-EB09-485C-98AB-4AF0C5260CC4Q55311061-13B129B3-EF05-49B0-8AF2-36763AF67E91Q55333053-BD6CF919-F8E7-4197-94D4-BC3D98E7BDB7Q55375210-1C6131B8-49F3-48E6-8D9E-F8EE1C2628B8Q55435506-8989C57B-3DD8-4191-9570-473AD759D0A8Q58781616-02E153A9-AD65-4C96-A877-52A9A8502EF5
P2860
Myeloid-Derived Suppressor Cells Endow Stem-like Qualities to Breast Cancer Cells through IL6/STAT3 and NO/NOTCH Cross-talk Signaling.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Myeloid-Derived Suppressor Cel ...... NO/NOTCH Cross-talk Signaling.
@en
type
label
Myeloid-Derived Suppressor Cel ...... NO/NOTCH Cross-talk Signaling.
@en
prefLabel
Myeloid-Derived Suppressor Cel ...... NO/NOTCH Cross-talk Signaling.
@en
P2093
P2860
P50
P1433
P1476
Myeloid-Derived Suppressor Cel ...... NO/NOTCH Cross-talk Signaling
@en
P2093
Alfred E Chang
Andrzej Kurylcio
Andrzej Stanisławek
Celina Kleer
Dongjun Peng
Elzbieta Staroslawska
Franciszek Szubstarski
Ilona Kryczek
Linda Vatan
P2860
P304
P356
10.1158/0008-5472.CAN-15-2528
P407
P577
2016-04-06T00:00:00Z