Beta-cell replication is the primary mechanism subserving the postnatal expansion of beta-cell mass in humans. - Wikidata - wikimedia - TriplyDB

Beta-cell replication is the primary mechanism subserving the postnatal expansion of beta-cell mass in humans.

Beta-cell replication is the primary mechanism subserving the postnatal expansion of beta-cell mass in humans.

http://www.wikidata.org/entity/Q36971500

about

Induction of human beta-cell proliferation and engraftment using a single G1/S regulatory molecule, cdk6 RETRACTED: Betatrophin: a hormone that controls pancreatic β cell proliferation Beneficial endocrine but adverse exocrine effects of sitagliptin in the human islet amyloid polypeptide transgenic rat model of type 2 diabetes: interactions with metformin β-cell replacement sources for type 1 diabetes: a focus on pancreatic ductal cells Dynamic pathology of islet endocrine cells in type 2 diabetes: β-Cell growth, death, regeneration and their clinical implications Report from IPITA-TTS Opinion Leaders Meeting on the Future of β-Cell Replacement Maternal microchimerism: friend or foe in type 1 diabetes?Role of microRNAs in islet beta-cell compensation and failure during diabetes Concise review: pancreas regeneration: recent advances and perspectives Maintenance of β-cell maturity and plasticity in the adult pancreas: developmental biology concepts in adult physiology IL-2 immunotherapy reveals potential for innate beta cell regeneration in the non-obese diabetic mouse model of autoimmune diabetes β-cell dysfunction: Its critical role in prevention and management of type 2 diabetes.The Rise and the Fall of Betatrophin/ANGPTL8 as an Inducer of β-Cell Proliferation Natural history of β-cell adaptation and failure in type 2 diabetes Insulin-producing cells generated from dedifferentiated human pancreatic beta cells expanded in vitro Epoxypukalide induces proliferation and protects against cytokine-mediated apoptosis in primary cultures of pancreatic β-cells Differential and complementary effects of glucose and prolactin on islet DNA synthesis and gene expression In vitro proliferation of adult human beta-cells Pancreatic islet plasticity: interspecies comparison of islet architecture and composition Metabolism Disrupting Chemicals and Metabolic Disorders Islet biology, the CDKN2A/B locus and type 2 diabetes risk.Adult tissue sources for new β cells.Dynamics of beta-cell turnover: evidence for beta-cell turnover and regeneration from sources of beta-cells other than beta-cell replication in the HIP rat Adaptive changes in pancreatic beta cell fractional area and beta cell turnover in human pregnancy.Cytoplasmic-nuclear trafficking of G1/S cell cycle molecules and adult human β-cell replication: a revised model of human β-cell G1/S control Is dynamic autocrine insulin signaling possible? A mathematical model predicts picomolar concentrations of extracellular monomeric insulin within human pancreatic islets.ALDH expression characterizes G1-phase proliferating beta cells during pregnancy.β-cell failure in type 2 diabetes: postulated mechanisms and prospects for prevention and treatment.Pancreatic β-cell proliferation in obesity.Recurrence of autoimmunity following pancreas transplantation.Age-associated changes of islet endocrine cells and the effects of body mass index in Japanese.Slow and steady is the key to beta-cell replication.EGF regulates survivin stability through the Raf-1/ERK pathway in insulin-secreting pancreatic β-cells.Development of factors to convert frequency to rate for beta-cell replication and apoptosis quantified by time-lapse video microscopy and immunohistochemistry.The negative cell cycle regulators, p27(Kip1), p18(Ink4c), and GSK-3, play critical role in maintaining quiescence of adult human pancreatic β-cells and restrict their ability to proliferate.Current status of islet cell replacement and regeneration therapy.Cyclin D2 is essential for the compensatory beta-cell hyperplastic response to insulin resistance in rodents Insulin secretion and insulin-producing tumors Neuropeptide Y expression marks partially differentiated β cells in mice and humans Beta-cell replication is increased in donor organs from young patients after prolonged life support.

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P2860

Beta-cell replication is the primary mechanism subserving the postnatal expansion of beta-cell mass in humans.

http://www.wikidata.org/entity/Q36971500

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Alexandra E Butler

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Anil Bhushan

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Juris J Meier

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Peter C Butler

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Robert A Rizza

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Ryan Galasso

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Travis Monchamp

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Yoshifumi Saisho

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P2860

Islet transplantation as a treatment for diabetes - a work in progress

Replication of genome-wide association signals in UK samples reveals risk loci for type 2 diabetes

Beta-cell deficit and increased beta-cell apoptosis in humans with type 2 diabetes

Five-year follow-up after clinical islet transplantation.

Pancreas and islet transplantation for patients with diabetes.

In vivo derivation of glucose-competent pancreatic endocrine cells from bone marrow without evidence of cell fusion.

Pancreatic beta cells in insulin-dependent diabetes.

The potential for stem cell therapy in diabetes.

Pancreas volumes in humans from birth to age one hundred taking into account sex, obesity, and presence of type-2 diabetes

Islet pathology and the pathogenesis of type 1 and type 2 diabetes mellitus revisited.

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10.2337/DB07-1369

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57

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