RANKL inhibition through osteoprotegerin blocks bone loss in experimental periodontitis. - Wikidata - wikimedia - TriplyDB

RANKL inhibition through osteoprotegerin blocks bone loss in experimental periodontitis.

RANKL inhibition through osteoprotegerin blocks bone loss in experimental periodontitis.

http://www.wikidata.org/entity/Q36973041

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Periodontitis and bone metabolism The role of osteoimmunology in periodontal disease Host modulation therapy: An indispensable part of perioceutics β-Glucans (Saccharomyces cereviseae) Reduce Glucose Levels and Attenuate Alveolar Bone Loss in Diabetic Rats with Periodontal Disease The enduring importance of animal models in understanding periodontal disease FAM5C contributes to aggressive periodontitis.Treatment of experimental periodontal disease with antimicrobial photodynamic therapy in nicotine-modified rats.Infection of RANKL-primed RAW-D macrophages with Porphyromonas gingivalis promotes osteoclastogenesis in a TNF-α-independent manner.Irradiation by light-emitting diode light as an adjunct to facilitate healing of experimental periodontitis in vivo.Review of osteoimmunology and the host response in endodontic and periodontal lesions.Inflammation and uncoupling as mechanisms of periodontal bone loss Mechanisms of Bone Resorption in Periodontitis.RANKL inhibitors induce osteonecrosis of the jaw in mice with periapical disease.Pre-clinical models for oral and periodontal reconstructive therapies.Langerhans cells down-regulate inflammation-driven alveolar bone loss.In vitro evaluation of osteoprotegerin in chitosan for potential bone defect applications.Biochemical markers as predictors of bone remodelling in dental disorders: a narrative description of literature Periostin is down-regulated during periodontal inflammation.OPG-Fc but Not Zoledronic Acid Discontinuation Reverses Osteonecrosis of the Jaws (ONJ) in Mice.Differential patterns of receptor activator of nuclear factor kappa B ligand/osteoprotegerin expression in human periapical granulomas: possible association with progressive or stable nature of the lesions.Association of periodontitis with rheumatoid arthritis and atherosclerosis: Novel paradigms in etiopathogeneses and management?Toll gates to periodontal host modulation and vaccine therapy High- and low-dose OPG-Fc cause osteopetrosis-like changes in infant mice.RANKL inhibition for the management of patients with benign metabolic bone disorders.Comparable outcomes in fracture reduction and bone properties with RANKL inhibition and alendronate treatment in a mouse model of osteogenesis imperfecta.What can the periodontal community learn from the pathophysiology of rheumatoid arthritis?The RANKL-OPG system in clinical periodontology.Advanced reconstructive technologies for periodontal tissue repair.Periodontal disease: a genetic perspective.Rheumatoid arthritis in the elderly and its relationship with periodontitis: a review.Local delivery of recombinant osteoprotegerin enhances postorthodontic tooth stability.Personalized medicine: an update of salivary biomarkers for periodontal diseases.Efficacy of stem cells on periodontal regeneration: Systematic review of pre-clinical studies.The osteoimmunology of alveolar bone loss.The emerging roles of B cells as partners and targets in periodontitis.Transglutaminase 2 up-regulation is associated with RANKL/OPG pathway in cultured HPDL cells and THP-1-differentiated macrophages.In vivo osteoprotegerin gene therapy preventing bone loss induced by periodontitis.Activation of RANKL-induced osteoclasts and memory T lymphocytes by Porphyromonas gingivalis is serotype dependant.Inflammatory osteolysis: a conspiracy against bone.Murine Experimental Root Canal Infection: Cytokine Expression in Response to F. nucleatum and E. faecalis.

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P2860

RANKL inhibition through osteoprotegerin blocks bone loss in experimental periodontitis.

http://www.wikidata.org/entity/Q36973041

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2007 nî lūn-bûn

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RANKL inhibition through osteoprotegerin blocks bone loss in experimental periodontitis.

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RANKL inhibition through osteoprotegerin blocks bone loss in experimental periodontitis.

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RANKL inhibition through osteoprotegerin blocks bone loss in experimental periodontitis.

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JOP.2007.070073

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Journal of Periodontology

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RANKL inhibition through osteoprotegerin blocks bone loss in experimental periodontitis

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Chan Ho Park

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James V Sugai

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Paul J Kostenuik

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Qiming Jin

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P2860

Osteoprotegerin: a novel secreted protein involved in the regulation of bone density

osteoprotegerin-deficient mice develop early onset osteoporosis and arterial calcification

Bisphosphonate-induced exposed bone (osteonecrosis/osteopetrosis) of the jaws: risk factors, recognition, prevention, and treatment

OPGL is a key regulator of osteoclastogenesis, lymphocyte development and lymph-node organogenesis

The roles of osteoprotegerin and osteoprotegerin ligand in the paracrine regulation of bone resorption.

Functional human T-cell immunity and osteoprotegerin ligand control alveolar bone destruction in periodontal infection.

Osteoprotegerin reduces osteoclast numbers and prevents bone erosion in collagen-induced arthritis.

Osteoprotegerin and RANKL regulate bone resorption, density, geometry and strength.

Novel host response therapeutic approaches to treat periodontal diseases.

Three-dimensional micro-computed tomographic imaging of alveolar bone in experimental bone loss or repair.

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