High- and low-dose OPG-Fc cause osteopetrosis-like changes in infant mice. - Wikidata - wikimedia - TriplyDB

High- and low-dose OPG-Fc cause osteopetrosis-like changes in infant mice.

High- and low-dose OPG-Fc cause osteopetrosis-like changes in infant mice.

http://www.wikidata.org/entity/Q37463716

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RANKL inhibitors induce osteonecrosis of the jaw in mice with periapical disease.Are Changes in Composition in Response to Treatment of a Mouse Model of Osteogenesis Imperfecta Sex-dependent?Bioprinting and Organ-on-Chip Applications Towards Personalized Medicine for Bone Diseases.Physiological role of receptor activator nuclear factor-kB (RANK) in denervation-induced muscle atrophy and dysfunction.Bone quality changes associated with aging and disease: a review.Impaired bone remodeling in children with osteogenesis imperfecta treated and untreated with bisphosphonates: the role of DKK1, RANKL, and TNF-α.Recombinant osteoprotegerin effects during orthodontic movement in a rat model.The Effect of Stontium Ranelate on Fracture Reduction in Osteogenesis Imperfecta is Comparable to Recent Bisphosphonate Data.

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High- and low-dose OPG-Fc cause osteopetrosis-like changes in infant mice.

http://www.wikidata.org/entity/Q37463716

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 27 August 2012

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

@cs

name

High- and low-dose OPG-Fc cause osteopetrosis-like changes in infant mice.

@en

High- and low-dose OPG-Fc cause osteopetrosis-like changes in infant mice.

@nl

type

label

High- and low-dose OPG-Fc cause osteopetrosis-like changes in infant mice.

@en

High- and low-dose OPG-Fc cause osteopetrosis-like changes in infant mice.

@nl

prefLabel

High- and low-dose OPG-Fc cause osteopetrosis-like changes in infant mice.

@en

High- and low-dose OPG-Fc cause osteopetrosis-like changes in infant mice.

@nl

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Pediatric Research

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High- and low-dose OPG-Fc cause osteopetrosis-like changes in infant mice

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Cathleen Raggio

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Edward DiCarlo

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Erin Carter

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Kostas Verdelis

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Nancy Pleshko

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Ram Posham

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Renee Bargman

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P2860

RANK is essential for osteoclast and lymph node development

Tumor necrosis factor receptor family member RANK mediates osteoclast differentiation and activation induced by osteoprotegerin ligand

The receptor activator of nuclear factor-kappaB ligand inhibitor osteoprotegerin is a bone-protective agent in a rat model of chronic renal insufficiency and hyperparathyroidism.

The RANKL inhibitor OPG-Fc increases cortical and trabecular bone mass in young gonad-intact cynomolgus monkeys.

Randomized trial of denosumab in patients receiving adjuvant aromatase inhibitors for nonmetastatic breast cancer.

Defective pro alpha 2(I) collagen synthesis in a recessive mutation in mice: a model of human osteogenesis imperfecta.

Osteoprotegerin reverses osteoporosis by inhibiting endosteal osteoclasts and prevents vascular calcification by blocking a process resembling osteoclastogenesis.

Receptor activator of nuclear factor kappaB ligand and osteoprotegerin regulation of bone remodeling in health and disease.

RANKL inhibition through osteoprotegerin blocks bone loss in experimental periodontitis.

Towards a better understanding and new therapeutics of osteopetrosis.

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495-501

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scholarly article

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10.1038/PR.2012.118

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5

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Adele L. Boskey

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2012-08-27T00:00:00Z

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230748336

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3888234

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