Anaerobiosis, type 1 fimbriae, and growth phase are factors that affect invasion of HEp-2 cells by Salmonella typhimurium
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Salmonella typhimurium encodes an SdiA homolog, a putative quorum sensor of the LuxR family, that regulates genes on the virulence plasmid.Type III protein secretion systems in bacterial pathogens of animals and plantsThe cost of virulence: retarded growth of Salmonella Typhimurium cells expressing type III secretion system 1DNA-binding activities of the HilC and HilD virulence regulatory proteins of Salmonella enterica serovar TyphimuriumOxygen restriction increases the infective potential of Listeria monocytogenes in vitro in Caco-2 cells and in vivo in guinea pigsGenome-wide screen of Salmonella genes expressed during infection in pigs, using in vivo expression technologyPerturbation of the small intestine microbial ecology by streptomycin alters pathology in a Salmonella enterica serovar typhimurium murine model of infectionMolecular basis of the interaction of Salmonella with the intestinal mucosa.Salmonella pathogenicity island 1-independent induction of apoptosis in infected macrophages by Salmonella enterica serotype typhimurium.Osmolarity and growth phase overlap in regulation of Salmonella typhi adherence to and invasion of human intestinal cellsIdentification of a novel Salmonella invasion locus homologous to Shigella ipgDEEnvironmental regulation of Salmonella typhi invasion-defective mutantsInoculum composition and Salmonella pathogenicity island 1 regulate M-cell invasion and epithelial destruction by Salmonella typhimurium.Multiple fimbrial adhesins are required for full virulence of Salmonella typhimurium in miceEvolution of host adaptation in Salmonella enterica.The Salmonella enterica serotype Typhimurium lpf, bcf, stb, stc, std, and sth fimbrial operons are required for intestinal persistence in miceUse of a dynamic in vitro attachment and invasion system (DIVAS) to determine influence of growth rate on invasion of respiratory epithelial cells by group B StreptococcusA HilA-independent pathway to Salmonella typhimurium invasion gene transcription.InvF is required for expression of genes encoding proteins secreted by the SPI1 type III secretion apparatus in Salmonella typhimurium.FimW is a negative regulator affecting type 1 fimbrial expression in Salmonella enterica serovar typhimurium.Hha is a negative modulator of transcription of hilA, the Salmonella enterica serovar Typhimurium invasion gene transcriptional activatorDifferential regulation of Salmonella typhimurium type III secreted proteins by pathogenicity island 1 (SPI-1)-encoded transcriptional activators InvF and hilA.Analysis of virulence of clinical isolates of Salmonella enteritidis in vivo and in vitro.Microgravity as a novel environmental signal affecting Salmonella enterica serovar Typhimurium virulenceCharacterization of FimY as a coactivator of type 1 fimbrial expression in Salmonella enterica serovar Typhimurium.Role of cross talk in regulating the dynamic expression of the flagellar Salmonella pathogenicity island 1 and type 1 fimbrial genesThe flagellar regulator fliT represses Salmonella pathogenicity island 1 through flhDC and fliZ.Extracellular glutathione decreases the ability of Burkholderia cenocepacia to penetrate into epithelial cells and to induce an inflammatory responseIdentification and characterization of a Salmonella typhimurium oxygen-regulated gene required for bacterial internalization.Mechanism of hilA repression by 1,2-propanediol consists of two distinct pathways, one dependent on and the other independent of catabolic production of propionate, in Salmonella enterica serovar Typhimurium.HilE interacts with HilD and negatively regulates hilA transcription and expression of the Salmonella enterica serovar Typhimurium invasive phenotype.Chloramphenicol and tetracycline decrease motility and increase invasion and attachment gene expression in specific isolates of multidrug-resistant Salmonella enterica serovar Typhimurium.Contribution of fimbrial operons to attachment to and invasion of epithelial cell lines by Salmonella typhimurium.An altered rpoS allele contributes to the avirulence of Salmonella typhimurium LT2.Natural proteoglycan receptor analogs determine the dynamics of Opa adhesin-mediated gonococcal infection of Chang epithelial cells.Exposure to sub-inhibitory concentrations of cefotaxime enhances the systemic colonization of Salmonella Typhimurium in BALB/c mice.Inherent Variability of Growth Media Impacts the Ability of Salmonella Typhimurium to Interact with Host Cells.Environmental signals controlling expression of virulence determinants in bacteriaMolecular and functional characterization of the Salmonella invasion gene invA: homology of InvA to members of a new protein familyGrowth regulation of a Salmonella plasmid gene essential for virulence.
P2860
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P2860
Anaerobiosis, type 1 fimbriae, and growth phase are factors that affect invasion of HEp-2 cells by Salmonella typhimurium
description
1990 nî lūn-bûn
@nan
1990年の論文
@ja
1990年論文
@yue
1990年論文
@zh-hant
1990年論文
@zh-hk
1990年論文
@zh-mo
1990年論文
@zh-tw
1990年论文
@wuu
1990年论文
@zh
1990年论文
@zh-cn
name
Anaerobiosis, type 1 fimbriae, ...... ells by Salmonella typhimurium
@en
type
label
Anaerobiosis, type 1 fimbriae, ...... ells by Salmonella typhimurium
@en
prefLabel
Anaerobiosis, type 1 fimbriae, ...... ells by Salmonella typhimurium
@en
P2093
P2860
P1476
Anaerobiosis, type 1 fimbriae, ...... ells by Salmonella typhimurium
@en
P2093
D M Dombroski
J M Merrick
P2860
P304
P407
P577
1990-06-01T00:00:00Z