Common patterns of bcl-2 family gene expression in two traumatic brain injury models.
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Genetic manipulation of cell death and neuroplasticity pathways in traumatic brain injurySinomenine Provides Neuroprotection in Model of Traumatic Brain Injury via the Nrf2-ARE PathwayThe Fas Ligand/Fas Death Receptor Pathways Contribute to Propofol-Induced Apoptosis and Neuroinflammation in the Brain of Neonatal Rats.Reduced neuronal cell death after experimental brain injury in mice lacking a functional alternative pathway of complement activationErythropoietin improves histological and functional outcomes after traumatic brain injury in mice in the absence of the neural erythropoietin receptor.Transcriptome profiling of hippocampal CA1 after early-life seizure-induced preconditioning may elucidate new genetic therapies for epilepsy.Serum levels of caspase-cleaved cytokeratin-18 in patients with severe traumatic brain injury are associated with mortality: a pilot study.Recent findings on neuroprotection against excitotoxicity in the hippocampus of female rats.
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Common patterns of bcl-2 family gene expression in two traumatic brain injury models.
description
2004 nî lūn-bûn
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name
Common patterns of bcl-2 family gene expression in two traumatic brain injury models.
@en
type
label
Common patterns of bcl-2 family gene expression in two traumatic brain injury models.
@en
prefLabel
Common patterns of bcl-2 family gene expression in two traumatic brain injury models.
@en
P2093
P2860
P356
P1476
Common patterns of bcl-2 family gene expression in two traumatic brain injury models.
@en
P2093
Kenneth I Strauss
Raj K Narayan
Ramesh Raghupathi
P2860
P2888
P304
P356
10.1007/BF03033444
P577
2004-01-01T00:00:00Z