Par-4/NF-κB Mediates the Apoptosis of Islet β Cells Induced by Glucolipotoxicity. - Wikidata - wikimedia - TriplyDB

Par-4/NF-κB Mediates the Apoptosis of Islet β Cells Induced by Glucolipotoxicity.

Par-4/NF-κB Mediates the Apoptosis of Islet β Cells Induced by Glucolipotoxicity.

http://www.wikidata.org/entity/Q37000205

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Inhibitory effect of Par-4 combined with cisplatin on human Wilms' tumor cells.Anacardic acid induces cell apoptosis of prostatic cancer through autophagy by ER stress/DAPK3/Akt signaling pathway.TERT and Akt Are Involved in the Par-4-Dependent Apoptosis of Islet Cells in Type 2 Diabetes

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Par-4/NF-κB Mediates the Apoptosis of Islet β Cells Induced by Glucolipotoxicity.

http://www.wikidata.org/entity/Q37000205

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 31 May 2016

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

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name

Par-4/NF-κB Mediates the Apoptosis of Islet β Cells Induced by Glucolipotoxicity.

@en

Par-4/NF-κB Mediates the Apoptosis of Islet β Cells Induced by Glucolipotoxicity.

@nl

type

label

Par-4/NF-κB Mediates the Apoptosis of Islet β Cells Induced by Glucolipotoxicity.

@en

Par-4/NF-κB Mediates the Apoptosis of Islet β Cells Induced by Glucolipotoxicity.

@nl

prefLabel

Par-4/NF-κB Mediates the Apoptosis of Islet β Cells Induced by Glucolipotoxicity.

@en

Par-4/NF-κB Mediates the Apoptosis of Islet β Cells Induced by Glucolipotoxicity.

@nl

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Journal of Diabetes Research

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Par-4/NF-κB Mediates the Apoptosis of Islet β Cells Induced by Glucolipotoxicity.

@en

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Chen Bing

http://www.w3.org/2001/XMLSchema#string

Deng WuQuan

http://www.w3.org/2001/XMLSchema#string

Gan XiaGuang

http://www.w3.org/2001/XMLSchema#string

Lei XiaoTian

http://www.w3.org/2001/XMLSchema#string

Wu QiNan

http://www.w3.org/2001/XMLSchema#string

Zhang Ling

http://www.w3.org/2001/XMLSchema#string

P2860

The tumor suppressor Par-4 activates an extrinsic pathway for apoptosis

Intrinsic-mediated caspase activation is essential for cardiomyocyte hypertrophy

Targeted disruption of the Chop gene delays endoplasmic reticulum stress-mediated diabetes

Endoplasmic reticulum stress links obesity, insulin action, and type 2 diabetes

The unfolded protein response: from stress pathway to homeostatic regulation

Beta-cell deficit and increased beta-cell apoptosis in humans with type 2 diabetes

Tumour-suppression activity of the proapoptotic regulator Par4.

Impact of endoplasmic reticulum stress pathway on pancreatic beta-cells and diabetes mellitus.

Islets transplanted in immunoisolation devices: a review of the progress and the challenges that remain

Par-4 downregulation promotes breast cancer recurrence by preventing multinucleation following targeted therapy.

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4692478

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scholarly article

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10.1155/2016/4692478

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2016

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2016-05-31T00:00:00Z

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27340675

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