Intrinsic-mediated caspase activation is essential for cardiomyocyte hypertrophy - Wikidata - wikimedia - TriplyDB

Intrinsic-mediated caspase activation is essential for cardiomyocyte hypertrophy

Intrinsic-mediated caspase activation is essential for cardiomyocyte hypertrophy

http://www.wikidata.org/entity/Q24605583

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The beneficial role of proteolysis in skeletal muscle growth and stress adaptation Spreading the word: non-autonomous effects of apoptosis during development, regeneration and disease Balancing functions of annexin A6 maintain equilibrium between hypertrophy and apoptosis in cardiomyocytes Dual small-molecule targeting of procaspase-3 dramatically enhances zymogen activation and anticancer activity The Role of Exercise in Cardiac Aging: From Physiology to Molecular Mechanisms Small-Molecule Procaspase-3 Activation Sensitizes Cancer to Treatment with Diverse Chemotherapeutics Differential and conditional activation of PKC-isoforms dictates cardiac adaptation during physiological to pathological hypertrophy.Expression of executioner procaspases and their activation by a procaspase-activating compound in chronic lymphocytic leukemia cells Dopamine modulates insulin release and is involved in the survival of rat pancreatic beta cells Removal of Metabolic Liabilities Enables Development of Derivatives of Procaspase-Activating Compound 1 (PAC-1) with Improved Pharmacokinetics Derivatives of Procaspase-Activating Compound 1 (PAC-1) and their Anticancer Activities.Caspase 3 cleavage of Pax7 inhibits self-renewal of satellite cells The ubiquitin E3 ligase TRAF6 exacerbates pathological cardiac hypertrophy via TAK1-dependent signalling.Par-4/NF-κB Mediates the Apoptosis of Islet β Cells Induced by Glucolipotoxicity.Pathophysiology of Myocardial Infarction.The Combination of Vemurafenib and Procaspase-3 Activation Is Synergistic in Mutant BRAF Melanomas.Doxorubicin-induced chronic dilated cardiomyopathy-the apoptosis hypothesis revisited.Cardiotrophin 1 stimulates beneficial myogenic and vascular remodeling of the heart.Synergistic and targeted therapy with a procaspase-3 activator and temozolomide extends survival in glioma rodent models and is feasible for the treatment of canine malignant glioma patients.MicroRNA-210-mediated proliferation, survival, and angiogenesis promote cardiac repair post myocardial infarction in rodents.Chronic activation of mTOR complex 1 by branched chain amino acids and organ hypertrophy.The role of K63-linked polyubiquitination in cardiac hypertrophy

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Intrinsic-mediated caspase activation is essential for cardiomyocyte hypertrophy

http://www.wikidata.org/entity/Q24605583

description

2013 nî lūn-bûn

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2013 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

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2013年論文

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2013年论文

@wuu

name

Intrinsic-mediated caspase activation is essential for cardiomyocyte hypertrophy

@ast

Intrinsic-mediated caspase activation is essential for cardiomyocyte hypertrophy

@en

Intrinsic-mediated caspase activation is essential for cardiomyocyte hypertrophy

@nl

type

label

Intrinsic-mediated caspase activation is essential for cardiomyocyte hypertrophy

@ast

Intrinsic-mediated caspase activation is essential for cardiomyocyte hypertrophy

@en

Intrinsic-mediated caspase activation is essential for cardiomyocyte hypertrophy

@nl

prefLabel

Intrinsic-mediated caspase activation is essential for cardiomyocyte hypertrophy

@ast

Intrinsic-mediated caspase activation is essential for cardiomyocyte hypertrophy

@en

Intrinsic-mediated caspase activation is essential for cardiomyocyte hypertrophy

@nl

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Intrinsic-mediated caspase activation is essential for cardiomyocyte hypertrophy

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P2093

Charis Putinski

http://www.w3.org/2001/XMLSchema#string

Mohammad Abdul-Ghani

http://www.w3.org/2001/XMLSchema#string

Pasan Fernando

http://www.w3.org/2001/XMLSchema#string

Rebecca Stiles

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Sarah A Dick

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Steve Brunette

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P2860

Essential myosin light chain as a target for caspase-3 in failing myocardium

Caspase 3/caspase-activated DNase promote cell differentiation by inducing DNA strand breaks

Signaling effectors underlying pathologic growth and remodeling of the heart

Metacaspase Yca1 is required for clearance of insoluble protein aggregates.

Functional consequences of caspase activation in cardiac myocytes

Class II histone deacetylases act as signal-responsive repressors of cardiac hypertrophy

PAC-1 activates procaspase-3 in vitro through relief of zinc-mediated inhibition

Small-molecule activation of procaspase-3 to caspase-3 as a personalized anticancer strategy

Rehabilitation of a contract killer: caspase-3 directs stem cell differentiation

MEF2 is upregulated during cardiac hypertrophy and is required for normal post-natal growth of the myocardium

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