DAP12 Inhibits Pulmonary Immune Responses to Cryptococcus neoformans

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Treatment with pCramoll Alone and in Combination with Fluconazole Provides Therapeutic Benefits in C. gattii Infected Mice.Innate Immune Responses to Cryptococcus.Host response to pulmonary fungal infections: A highlight on cell-driven immunity to Cryptococcus species and Aspergillus fumigatus.

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DAP12 Inhibits Pulmonary Immune Responses to Cryptococcus neoformans

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http://www.wikidata.org/entity/Q37002280

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 11 April 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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DAP12 Inhibits Pulmonary Immune Responses to Cryptococcus neoformans

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DAP12 Inhibits Pulmonary Immune Responses to Cryptococcus neoformans.

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DAP12 Inhibits Pulmonary Immune Responses to Cryptococcus neoformans

@en

DAP12 Inhibits Pulmonary Immune Responses to Cryptococcus neoformans.

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DAP12 Inhibits Pulmonary Immune Responses to Cryptococcus neoformans

@en

DAP12 Inhibits Pulmonary Immune Responses to Cryptococcus neoformans.

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DAP12 Inhibits Pulmonary Immune Responses to Cryptococcus neoformans

@en

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Tobias M Hohl

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Dectin-2 recognition of alpha-mannans and induction of Th17 cell differentiation is essential for host defense against Candida albicans

Involvement of CD14, toll-like receptors 2 and 4, and MyD88 in the host response to the fungal pathogen Cryptococcus neoformans in vivo

Characterization of a murine monoclonal antibody to Cryptococcus neoformans polysaccharide that is a candidate for human therapeutic studies.

Primary immunodeficiencies underlying fungal infections

TLR9 signaling is required for generation of the adaptive immune protection in Cryptococcus neoformans-infected lungs.

DAP12: an adapter protein with dual functionality.

The NK receptor NKp30 mediates direct fungal recognition and killing and is diminished in NK cells from HIV-infected patients.

Dectin-2 deficiency promotes Th2 response and mucin production in the lungs after pulmonary infection with Cryptococcus neoformans.

Infectious diseases in patients with IRAK-4, MyD88, NEMO, or IκBα deficiency.

Eosinophils contribute to IL-4 production and shape the T-helper cytokine profile and inflammatory response in pulmonary cryptococcosis.

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1879-1886

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scholarly article

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10.1128/IAI.00222-16

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English

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http://www.w3.org/2001/XMLSchema#string

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http://www.w3.org/2001/XMLSchema#string

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Lena J Heung

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2016-05-24T00:00:00Z

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27068093

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4907135

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DAP12 Inhibits Pulmonary Immune Responses to Cryptococcus neoformans

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P2860

P2860

DAP12 Inhibits Pulmonary Immune Responses to Cryptococcus neoformans

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P407

P433

P478

P50

P577

P698

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P407

P433

P478

P50

P577

P698

P932