Hepatocytes produce TNF-α following hypoxia-reoxygenation and liver ischemia-reperfusion in a NADPH oxidase- and c-Src-dependent manner.
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Oxidative stress and hepatic Nox proteins in chronic hepatitis C and hepatocellular carcinoma.Pregnane X receptor modulates the inflammatory response in primary cultures of hepatocytesAggravation of post-ischemic liver injury by overexpression of insulin-like growth factor binding protein 3.Idh1 protects murine hepatocytes from endotoxin-induced oxidative stress by regulating the intracellular NADP(+)/NADPH ratio.Hyperglycemia Aggravates Hepatic Ischemia Reperfusion Injury by Inducing Chronic Oxidative Stress and Inflammation.Sirtuin 2 aggravates postischemic liver injury by deacetylating mitogen-activated protein kinase phosphatase-1.The basic biology of redoxosomes in cytokine-mediated signal transduction and implications for disease-specific therapies.Preconditioning methods in the management of hepatic ischemia reperfusion- induced injury: Update on molecular and future perspectives.Pre-conditions for eliminating mitochondrial dysfunction and maintaining liver function after hepatic ischaemia reperfusion.Hops (Humulus lupulus) Content in Beer Modulates Effects of Beer on the Liver After Acute Ingestion in Female Mice.Ischemic preconditioning modulates ROS to confer protection in liver ischemia and reperfusion.NADPH Oxidases Are Essential for Macrophage Differentiation.Slow induction of brain death leads to decreased renal function and increased hepatic apoptosis in rats.Melatonin application in targeting oxidative-induced liver injuries: A review.Caveolin-1-dependent activation of the metalloprotease TACE/ADAM17 by TGF-β in hepatocytes requires activation of Src and the NADPH oxidase NOX1.Superoxide deficiency attenuates promotion of hepatocarcinogenesis by cytotoxicity in NADPH oxidase knockout mice.Effect of acute beer ingestion on the liver: studies in female mice.Complement Activation in Liver Transplantation: Role of Donor Macrosteatosis and Implications in Delayed Graft Function.
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P2860
Hepatocytes produce TNF-α following hypoxia-reoxygenation and liver ischemia-reperfusion in a NADPH oxidase- and c-Src-dependent manner.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 02 May 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Hepatocytes produce TNF-α foll ...... e- and c-Src-dependent manner.
@en
Hepatocytes produce TNF-α foll ...... e- and c-Src-dependent manner.
@nl
type
label
Hepatocytes produce TNF-α foll ...... e- and c-Src-dependent manner.
@en
Hepatocytes produce TNF-α foll ...... e- and c-Src-dependent manner.
@nl
prefLabel
Hepatocytes produce TNF-α foll ...... e- and c-Src-dependent manner.
@en
Hepatocytes produce TNF-α foll ...... e- and c-Src-dependent manner.
@nl
P2093
P2860
P50
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P1476
Hepatocytes produce TNF-α foll ...... e- and c-Src-dependent manner.
@en
P2093
Duane Abbott
Meihui Luo
Netanya Y Spencer
Thomas J Lynch
Weihong Zhou
Yulong Zhang
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P304
P356
10.1152/AJPGI.00430.2012
P577
2013-05-02T00:00:00Z