Epithelial restitution and wound healing in inflammatory bowel disease.
about
Suprapubic catheterization complicated by an iatrogenic enterocutaneous fistula: a case reportIntegrating Immunologic Signaling Networks: The JAK/STAT Pathway in Colitis and Colitis-Associated CancerRole of regulatory T cell in the pathogenesis of inflammatory bowel diseaseNew insights into the role of fatty acids in the pathogenesis and resolution of inflammatory bowel diseaseHypoxia and Integrin-Mediated Epithelial Restitution during Mucosal InflammationThe tumor suppressor gene, RASSF1A, is essential for protection against inflammation -induced injurySaccharomyces boulardii improves intestinal epithelial cell restitution by inhibiting αvβ5 integrin activation stateK+ Channel Inhibition Differentially Regulates Migration of Intestinal Epithelial Cells in Inflamed vs. Non-Inflamed Conditions in a PI3K/Akt-Mediated MannerSaccharomyces boulardii improves intestinal cell restitution through activation of the α2β1 integrin collagen receptorSelective non-steroidal glucocorticoid receptor agonists attenuate inflammation but do not impair intestinal epithelial cell restitution in vitroOverexpression of translocator protein in inflammatory bowel disease: potential diagnostic and treatment valueEffects of Ganoderma lucidum polysaccharides on IEC-6 cell proliferation, migration and morphology of differentiation benefiting intestinal epithelium healing in vitro.A high-fiber diet may improve bowel function and health-related quality of life in patients with Crohn diseaseDietary fiber information for individuals with Crohn disease: reports of gastrointestinal effects.Enteric glia promote intestinal mucosal healing via activation of focal adhesion kinase and release of proEGFEnterocyte loss of polarity and gut wound healing rely upon the F-actin-severing function of villinDUSP10 regulates intestinal epithelial cell growth and colorectal tumorigenesis.Myogenic lineage differentiated mesenchymal stem cells enhance recovery from dextran sulfate sodium-induced colitis in the rat.NOD-like receptors in intestinal homeostasis and epithelial tissue repair.The impact of the level of the intestinal short chain Fatty acids in inflammatory bowel disease patients versus healthy subjects.Repair of damaged intestinal mucosa in a mouse model of sepsisLPS-binding protein enables intestinal epithelial restitution despite LPS exposure.PI3K-dependent GSK3ß(Ser9)-phosphorylation is implicated in the intestinal epithelial cell wound-healing response.Loss of protein tyrosine phosphatase N2 potentiates epidermal growth factor suppression of intestinal epithelial chloride secretion.Krüppel-like factor 5 protects against murine colitis and activates JAK-STAT signaling in vivo.Novel xylan-controlled delivery of therapeutic proteins to inflamed colon by the human anaerobic commensal bacteriumThe microbiota protects against ischemia/reperfusion-induced intestinal injury through nucleotide-binding oligomerization domain-containing protein 2 (NOD2) signaling.The Pseudomonas aeruginosa N-acylhomoserine lactone quorum sensing molecules target IQGAP1 and modulate epithelial cell migrationAbnormal cannabidiol attenuates experimental colitis in mice, promotes wound healing and inhibits neutrophil recruitment.Krüppel-like factor 5 protects against dextran sulfate sodium-induced colonic injury in mice by promoting epithelial repairRelevance of TNBS-colitis in rats: a methodological study with endoscopic, histologic and Transcriptomic [corrected] characterization and correlation to IBD.Activation of PKCβII by PMA facilitates enhanced epithelial wound repair through increased cell spreading and migration.Kinetic characterization of an intestinal trefoil factor receptor.Protein kinase C iota in the intestinal epithelium protects against dextran sodium sulfate-induced colitis.Mu opioid signaling protects against acute murine intestinal injury in a manner involving Stat3 signalingContribution of microbiota to the intestinal physicochemical barrier.Intestinal genetic inactivation of caspase-8 diminishes migration of enterocytes.Nutrient-induced inflammation in the intestineA Microbial Feed Additive Abates Intestinal Inflammation in Atlantic SalmonMilk fat globule-epidermal growth factor 8 is decreased in intestinal epithelium of ulcerative colitis patients and thereby causes increased apoptosis and impaired wound healing.
P2860
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P2860
Epithelial restitution and wound healing in inflammatory bowel disease.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on January 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Epithelial restitution and wound healing in inflammatory bowel disease.
@en
Epithelial restitution and wound healing in inflammatory bowel disease.
@nl
type
label
Epithelial restitution and wound healing in inflammatory bowel disease.
@en
Epithelial restitution and wound healing in inflammatory bowel disease.
@nl
prefLabel
Epithelial restitution and wound healing in inflammatory bowel disease.
@en
Epithelial restitution and wound healing in inflammatory bowel disease.
@nl
P2860
P356
P1476
Epithelial restitution and wound healing in inflammatory bowel disease.
@en
P2093
Andreas Sturm
Axel U Dignass
P2860
P304
P356
10.3748/WJG.14.348
P407
P577
2008-01-01T00:00:00Z