SEC16A is a RAB10 effector required for insulin-stimulated GLUT4 trafficking in adipocytes.
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Secretion of Adipsin as an Assay to Measure Flux from the Endoplasmic Reticulum (ER).TANGO1 recruits Sec16 to coordinately organize ER exit sites for efficient secretion.Adenovirus Protein E4-ORF1 Activation of PI3 Kinase Reveals Differential Regulation of Downstream Effector Pathways in Adipocytes.Insulin sends SEC16A packing.Consequences of Rab GTPase dysfunction in genetic or acquired human diseases.Biochemical and cellular properties of insulin receptor signalling.Downregulation of a GPCR by β-Arrestin2-Mediated Switch from an Endosomal to a TGN Recycling Pathway.Rab3a and Rab10 are regulators of lysosome exocytosis and plasma membrane repair.The cell biology of systemic insulin function.
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SEC16A is a RAB10 effector required for insulin-stimulated GLUT4 trafficking in adipocytes.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 27 June 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
SEC16A is a RAB10 effector req ...... UT4 trafficking in adipocytes.
@en
SEC16A is a RAB10 effector req ...... UT4 trafficking in adipocytes.
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type
label
SEC16A is a RAB10 effector req ...... UT4 trafficking in adipocytes.
@en
SEC16A is a RAB10 effector req ...... UT4 trafficking in adipocytes.
@nl
prefLabel
SEC16A is a RAB10 effector req ...... UT4 trafficking in adipocytes.
@en
SEC16A is a RAB10 effector req ...... UT4 trafficking in adipocytes.
@nl
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SEC16A is a RAB10 effector req ...... UT4 trafficking in adipocytes.
@en
P2093
David Iaea
Joanne Bruno
Natasha Chaudhary
Timothy E McGraw
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P356
10.1083/JCB.201509052
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P577
2016-06-27T00:00:00Z