Ablation of CD11c-positive cells normalizes insulin sensitivity in obese insulin resistant animals. - Wikidata - wikimedia - TriplyDB

Ablation of CD11c-positive cells normalizes insulin sensitivity in obese insulin resistant animals.

Ablation of CD11c-positive cells normalizes insulin sensitivity in obese insulin resistant animals.

http://www.wikidata.org/entity/Q37072370

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Menopause, obesity and inflammation: interactive risk factors for Alzheimer's disease B cells promote insulin resistance through modulation of T cells and production of pathogenic IgG antibodies Getting 'Smad' about obesity and diabetes Adipose Tissue Remodeling: Its Role in Energy Metabolism and Metabolic Disorders Emerging evidence for beneficial macrophage functions in atherosclerosis and obesity-induced insulin resistance N-3 Polyunsaturated Fatty Acids and Inflammation in Obesity: Local Effect and Systemic Benefit The "Big Bang" in obese fat: Events initiating obesity-induced adipose tissue inflammation Adipocytes: impact on tumor growth and potential sites for therapeutic intervention The immune system's involvement in obesity-driven type 2 diabetes Inflammation, obesity, and the promise of immunotherapy for metabolic disease The role of omega-3 fatty acid receptor GPR120 in insulin resistance Myeloid heme oxygenase-1 haploinsufficiency reduces high fat diet-induced insulin resistance by affecting adipose macrophage infiltration in mice Perinatal overnutrition exacerbates adipose tissue inflammation caused by high-fat feeding in C57BL/6J mice Neuropeptide Y is produced by adipose tissue macrophages and regulates obesity-induced inflammation Macrophage migration inhibitory factor deficiency ameliorates high-fat diet induced insulin resistance in mice with reduced adipose inflammation and hepatic steatosis Osteopontin deletion prevents the development of obesity and hepatic steatosis via impaired adipose tissue matrix remodeling and reduced inflammation and fibrosis in adipose tissue and liver in mice The Microbial Hypothesis: Contributions of Adenovirus Infection and Metabolic Endotoxaemia to the Pathogenesis of Obesity Minireview: Emerging Concepts in Islet Macrophage Biology in Type 2 Diabetes Revisiting the adipocyte: a model for integration of cytokine signaling in the regulation of energy metabolism Resistant starches for the management of metabolic diseases Renin-Angiotensin-aldosterone system in diabetes and hypertension Interactions between inflammation, sex steroids, and Alzheimer's disease risk factors Regulation of diet-induced adipose tissue and systemic inflammation by salicylates and pioglitazone Tff3, as a novel peptide, regulates hepatic glucose metabolism The NLRP3 inflammasome instigates obesity-induced inflammation and insulin resistance Cellular and molecular players in adipose tissue inflammation in the development of obesity-induced insulin resistance Dendritic cells and adipose tissue.The macrophage at the intersection of immunity and metabolism in obesity Characterization of immune cells in psoriatic adipose tissue.An MHC II-dependent activation loop between adipose tissue macrophages and CD4+ T cells controls obesity-induced inflammation.Reducing glycosphingolipid content in adipose tissue of obese mice restores insulin sensitivity, adipogenesis and reduces inflammation CX3CR1 deficiency does not influence trafficking of adipose tissue macrophages in mice with diet-induced obesity.CYP2J2 and Its Metabolites EETs Attenuate Insulin Resistance via Regulating Macrophage Polarization in Adipose Tissue.C/EBPα regulates macrophage activation and systemic metabolism.RAGE regulates the metabolic and inflammatory response to high-fat feeding in mice Adipocyte apoptosis, a link between obesity, insulin resistance, and hepatic steatosis.CD11c expression in adipose tissue and blood and its role in diet-induced obesity.Mechanisms of inflammatory responses in obese adipose tissue.Crosstalk between adipocytes and immune cells in adipose tissue inflammation and metabolic dysregulation in obesity.Role of pro- and anti-inflammatory phenomena in the physiopathology of type 2 diabetes and obesity

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Ablation of CD11c-positive cells normalizes insulin sensitivity in obese insulin resistant animals.

http://www.wikidata.org/entity/Q37072370

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

@tr

scientific article published on October 2008

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Ablation of CD11c-positive cel ...... ese insulin resistant animals.

@en

Ablation of CD11c-positive cel ...... ese insulin resistant animals.

@nl

type

label

Ablation of CD11c-positive cel ...... ese insulin resistant animals.

@en

Ablation of CD11c-positive cel ...... ese insulin resistant animals.

@nl

prefLabel

Ablation of CD11c-positive cel ...... ese insulin resistant animals.

@en

Ablation of CD11c-positive cel ...... ese insulin resistant animals.

@nl

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J.CMET.2008.08.015

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Cell Metabolism

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Ablation of CD11c-positive cel ...... ese insulin resistant animals.

@en

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David Patsouris

http://www.w3.org/2001/XMLSchema#string

Divya Thapar

http://www.w3.org/2001/XMLSchema#string

Jaap G Neels

http://www.w3.org/2001/XMLSchema#string

Justin Chapman

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Ping-Ping Li

http://www.w3.org/2001/XMLSchema#string

P2860

Obesity, Inflammation, and Insulin Resistance

Obesity is associated with macrophage accumulation in adipose tissue

JNK1 in hematopoietically derived cells contributes to diet-induced inflammation and insulin resistance without affecting obesity

Bone marrow-specific Cap gene deletion protects against high-fat diet-induced insulin resistance

Monocyte and macrophage heterogeneity

Obesity induces a phenotypic switch in adipose tissue macrophage polarization

The chemokine system in diverse forms of macrophage activation and polarization

MCP-1 contributes to macrophage infiltration into adipose tissue, insulin resistance, and hepatic steatosis in obesity

IKK-beta links inflammation to obesity-induced insulin resistance

In vivo depletion of CD11c+ dendritic cells abrogates priming of CD8+ T cells by exogenous cell-associated antigens

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301-309

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scholarly article

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10.1016/J.CMET.2008.08.015

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4

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8

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Jerrold M. Olefsky

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2008-10-01T00:00:00Z

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23304755

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18840360

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