Comparison of responses to double-strand breaks between Escherichia coli and Bacillus subtilis reveals different requirements for SOS induction
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Protein interactions in genome maintenance as novel antibacterial targetsDynamics and Cell-Type Specificity of the DNA Double-Strand Break Repair Protein RecN in the Developmental Cyanobacterium Anabaena sp. Strain PCC 7120Co-orientation of replication and transcription preserves genome integrity.Hydroxyurea induces hydroxyl radical-mediated cell death in Escherichia coli.RecO and RecR are necessary for RecA loading in response to DNA damage and replication fork stress.Nucleoid occlusion prevents cell division during replication fork arrest in Bacillus subtilisThe LexA regulated genes of the Clostridium difficile.Mismatch repair causes the dynamic release of an essential DNA polymerase from the replication forkRapid pairing and resegregation of distant homologous loci enables double-strand break repair in bacteria.The RecA-Dependent SOS Response Is Active and Required for Processing of DNA Damage during Bacillus subtilis Sporulation.DNA repair and genome maintenance in Bacillus subtilis.RecA protein recruits structural maintenance of chromosomes (SMC)-like RecN protein to DNA double-strand breaksDNA damage responses in prokaryotes: regulating gene expression, modulating growth patterns, and manipulating replication forks.Imaging mismatch repair and cellular responses to DNA damage in Bacillus subtilis.RecD2 helicase limits replication fork stress in Bacillus subtilis.Escherichia coli DNA ligase B may mitigate damage from oxidative stress.A Magic Spot in Genome Maintenance.Regulation of chromosomal replication initiation by oriC-proximal DnaA-box clusters in Bacillus subtilis.Live cell imaging of SOS and prophage dynamics in isogenic bacterial populations.Prophage-triggered membrane vesicle formation through peptidoglycan damage in Bacillus subtilis.Interlinked sister chromosomes arise in the absence of condensin during fast replication in B. subtilis.Bacillus subtilis RecA and its accessory factors, RecF, RecO, RecR and RecX, are required for spore resistance to DNA double-strand break.Mutations in the Bacillus subtilis beta clamp that separate its roles in DNA replication from mismatch repair.Red light activates the sigmaB-mediated general stress response of Bacillus subtilis via the energy branch of the upstream signaling cascade.DnaN clamp zones provide a platform for spatiotemporal coupling of mismatch detection to DNA replication.Investigating the Detrimental Effects of Low Pressure Plasma Sterilization on the Survival of Bacillus subtilis Spores Using Live Cell Microscopy.Sources of spontaneous mutagenesis in bacteria.Trapping and visualizing intermediate steps in the mismatch repair pathway in vivo.The Bacillus subtilis SftA (YtpS) and SpoIIIE DNA translocases play distinct roles in growing cells to ensure faithful chromosome partitioning.
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Comparison of responses to double-strand breaks between Escherichia coli and Bacillus subtilis reveals different requirements for SOS induction
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 05 December 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Comparison of responses to dou ...... requirements for SOS induction
@en
Comparison of responses to dou ...... equirements for SOS induction.
@nl
type
label
Comparison of responses to dou ...... requirements for SOS induction
@en
Comparison of responses to dou ...... equirements for SOS induction.
@nl
prefLabel
Comparison of responses to dou ...... requirements for SOS induction
@en
Comparison of responses to dou ...... equirements for SOS induction.
@nl
P2093
P2860
P356
P1476
Comparison of responses to dou ...... requirements for SOS induction
@en
P2093
Alan D Grossman
Alexi I Goranov
Bryan W Davies
Daniel S Yuan
Hajime Kobayashi
Lyle A Simmons
P2860
P304
P356
10.1128/JB.01292-08
P407
P50
P577
2008-12-05T00:00:00Z