The role of immune activation in contributing to vascular dysfunction and the pathophysiology of hypertension during preeclampsia.
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No Hypertensive Disorder of Pregnancy; No Preeclampsia-eclampsia; No Gestational Hypertension; No Hellp Syndrome. Vascular Disorder of Pregnancy Speaks for AllPreeclampsia: long-term consequences for vascular healthAngiotensin receptor agonistic autoantibodies and hypertension: preeclampsia and beyondExcess LIGHT contributes to placental impairment, increased secretion of vasoactive factors, hypertension, and proteinuria in preeclampsia.The role of inflammation in the pathology of preeclampsiaProgesterone supplementation attenuates hypertension and the autoantibody to the angiotensin II type I receptor in response to elevated interleukin-6 during pregnancy.Endothelin-1 is not a Mechanism of IL-17 Induced Hypertension during Pregnancy.Role of angiotensin II type I receptor agonistic autoantibodies (AT1-AA) in preeclampsia.CD4+ T-helper cells stimulated in response to placental ischemia mediate hypertension during pregnancy17-hydroxyprogesterone caproate significantly improves clinical characteristics of preeclampsia in the reduced uterine perfusion pressure rat modelPlacental lesions associated with maternal underperfusion are more frequent in early-onset than in late-onset preeclampsiaIL-6-induced pathophysiology during pre-eclampsia: potential therapeutic role for magnesium sulfate?Maternal plasma concentrations of angiogenic/anti-angiogenic factors are of prognostic value in patients presenting to the obstetrical triage area with the suspicion of preeclampsia.IL-17-mediated oxidative stress is an important stimulator of AT1-AA and hypertension during pregnancy.Hypertension in response to IL-6 during pregnancy: role of AT1-receptor activation.Epicardial fat tissue thickness in preeclamptic and normal pregnancies.Four Pathways Involving Innate Immunity in the Pathogenesis of Preeclampsia.Vitamin D supplementation improves pathophysiology in a rat model of preeclampsiaIL-10 supplementation increases Tregs and decreases hypertension in the RUPP rat model of preeclampsia.Identifying immune mechanisms mediating the hypertension during preeclampsiaAdministration of interleukin-17 soluble receptor C suppresses TH17 cells, oxidative stress, and hypertension in response to placental ischemia during pregnancy.The prediction of late-onset preeclampsia: Results from a longitudinal proteomics study.Proliferation of endogenous regulatory T cells improve the pathophysiology associated with placental ischaemia of pregnancy.Increased Angiotensin II Sensitivity Contributes to Microvascular Dysfunction in Women Who Have Had Preeclampsia.Pathophysiology and Current Clinical Management of Preeclampsia.Serelaxin improves the pathophysiology of placental ischemia in the reduced uterine perfusion pressure rat model of preeclampsia.Reduced uterine perfusion pressure T-helper 17 cells cause pathophysiology associated with preeclampsia during pregnancy.Continued Investigation Into 17-OHPC: Results From the Preclinical RUPP Rat Model of Preeclampsia.Disruption in the Regulation of Immune Responses in the Placental Subtype of Preeclampsia
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The role of immune activation in contributing to vascular dysfunction and the pathophysiology of hypertension during preeclampsia.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on April 2010
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
The role of immune activation ...... ertension during preeclampsia.
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The role of immune activation ...... ertension during preeclampsia.
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type
label
The role of immune activation ...... ertension during preeclampsia.
@en
The role of immune activation ...... ertension during preeclampsia.
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prefLabel
The role of immune activation ...... ertension during preeclampsia.
@en
The role of immune activation ...... ertension during preeclampsia.
@nl
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The role of immune activation ...... ertension during preeclampsia.
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2010-04-01T00:00:00Z