Autoimmune cardiomyopathy and heart block develop spontaneously in HLA-DQ8 transgenic IAbeta knockout NOD mice - Wikidata - wikimedia - TriplyDB

Autoimmune cardiomyopathy and heart block develop spontaneously in HLA-DQ8 transgenic IAbeta knockout NOD mice

Autoimmune cardiomyopathy and heart block develop spontaneously in HLA-DQ8 transgenic IAbeta knockout NOD mice

http://www.wikidata.org/entity/Q37089400

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Small mammalian animal models of heart disease Impaired thymic tolerance to α-myosin directs autoimmunity to the heart in mice and humans Myocarditis: a defect in central immune tolerance?Myocardial infarction triggers chronic cardiac autoimmunity in type 1 diabetes.Cardiac autoimmunity as a novel biomarker, mediator, and therapeutic target of heart disease in type 1 diabetes.Gene-targeted mice lacking the Trex1 (DNase III) 3'-->5' DNA exonuclease develop inflammatory myocarditis.Humanized animal models for autoimmune diseases.Spontaneous myocarditis mimicking human disease occurs in the presence of an appropriate MHC and non-MHC background in transgenic mice.Localization of CD8 T cell epitope within cardiac myosin heavy chain-α334-352 that induces autoimmune myocarditis in A/J mice Role of impaired central tolerance to α-myosin in inflammatory heart disease.Programmed death ligand 1 regulates a critical checkpoint for autoimmune myocarditis and pneumonitis in MRL mice.Anti-heart autoantibodies in familial dilated cardiomyopathy.Mouse models for the study of autoimmune type 1 diabetes: a NOD to similarities and differences to human disease.Experiences with animal models of dermatitis herpetiformis: a review.Immune-mediated and autoimmune myocarditis: clinical presentation, diagnosis and management.Cardiac Autoimmunity: Myocarditis.FTY720 (Gilenya) treatment prevents spontaneous autoimmune myocarditis and dilated cardiomyopathy in transgenic HLA-DQ8-BALB/c mice.Species-specific immune responses generated by histidyl-tRNA synthetase immunization are associated with muscle and lung inflammation A humanized HLA-DR4 mouse model for autoimmune myocarditis.Understanding Autoimmune Diabetes through the Prism of the Tri-Molecular Complex.The Role of NOD Mice in Type 1 Diabetes Research: Lessons from the Past and Recommendations for the Future.

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Autoimmune cardiomyopathy and heart block develop spontaneously in HLA-DQ8 transgenic IAbeta knockout NOD mice

http://www.wikidata.org/entity/Q37089400

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 21 October 2003

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Autoimmune cardiomyopathy and ...... genic IAbeta knockout NOD mice

@en

Autoimmune cardiomyopathy and ...... enic IAbeta knockout NOD mice.

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type

label

Autoimmune cardiomyopathy and ...... genic IAbeta knockout NOD mice

@en

Autoimmune cardiomyopathy and ...... enic IAbeta knockout NOD mice.

@nl

prefLabel

Autoimmune cardiomyopathy and ...... genic IAbeta knockout NOD mice

@en

Autoimmune cardiomyopathy and ...... enic IAbeta knockout NOD mice.

@nl

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PNAS.2235552100

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Proceedings of the National Academy of Sciences of the United States of America

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Autoimmune cardiomyopathy and ...... genic IAbeta knockout NOD mice

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David Rayner

http://www.w3.org/2001/XMLSchema#string

Gang Liu

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John F Elliott

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Junliang Liu

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Katherine M Kavanagh

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Kunimasa Suzuki

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Murray A Robertson

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Norma Bautista-Lopez

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Patrick Nation

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Sarah L Wallbank

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P2860

Polymorphisms in the SOD2 and HLA-DRB1 genes are associated with nonfamilial idiopathic dilated cardiomyopathy in Japanese

Autoimmune dilated cardiomyopathy in PD-1 receptor-deficient mice

Prevention of type I diabetes in NOD mice by adjuvant immunotherapy.

Experimental autoimmune myocarditis in A/J mice is an interleukin-4-dependent disease with a Th2 phenotype.

The regulatory role of DR4 in a spontaneous diabetes DQ8 transgenic model

HLA-DQ8 transgenic mice are highly susceptible to collagen-induced arthritis: a novel model for human polyarthritis

Induction of insulitis by glutamic acid decarboxylase peptide-specific and HLA-DQ8-restricted CD4(+) T cells from human DQ transgenic mice.

Chlamydia infections and heart disease linked through antigenic mimicry.

Identification of cardiac myosin peptides capable of inducing autoimmune myocarditis in BALB/c mice.

Genetic control of autoimmune diabetes in the NOD mouse.

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13447-13452

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scholarly article

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10.1073/PNAS.2235552100

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23

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100

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2003-10-21T00:00:00Z

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231586220

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14570980

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263834

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