Host responses to wild-type and attenuated herpes simplex virus infection in the absence of Stat1
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Herpes simplex virus 2 ICP0 mutant viruses are avirulent and immunogenic: implications for a genital herpes vaccineModeling host genetic regulation of influenza pathogenesis in the collaborative crossThe virion host shutoff endonuclease (UL41) of herpes simplex virus interacts with the cellular cap-binding complex eIF4F.Bioluminescent imaging reveals divergent viral pathogenesis in two strains of Stat1-deficient mice, and in αßγ interferon receptor-deficient mice.Genome-wide mouse mutagenesis reveals CD45-mediated T cell function as critical in protective immunity to HSV-1.Degeneration and regeneration of corneal nerves in response to HSV-1 infectionInnate and adaptive immune responses to herpes simplex virusFunctional genomics reveals an essential and specific role for Stat1 in protection of the central nervous system following herpes simplex virus corneal infectionExpression quantitative trait Loci for extreme host response to influenza a in pre-collaborative cross mice.Corneal replication is an interferon response-independent bottleneck for virulence of herpes simplex virus 1 in the absence of virion host shutoff.Role of the DNA Sensor STING in Protection from Lethal Infection following Corneal and Intracerebral Challenge with Herpes Simplex Virus 1.mRNA decay during herpes simplex virus (HSV) infections: mutations that affect translation of an mRNA influence the sites at which it is cleaved by the HSV virion host shutoff (Vhs) protein.Cellular responses to HSV-1 infection are linked to specific types of alterations in the host transcriptome.Synergistic control of herpes simplex virus pathogenesis by IRF-3, and IRF-7 revealed through non-invasive bioluminescence imaging.Functional prediction of differentially expressed lncRNAs in HSV-1 infected human foreskin fibroblasts.The differential interferon responses of two strains of Stat1-deficient mice do not alter susceptibility to HSV-1 and VSV in vivo.Cellular Protein WDR11 Interacts with Specific Herpes Simplex Virus Proteins at the trans-Golgi Network To Promote Virus Replication.The herpes simplex virus 1 vhs protein enhances translation of viral true late mRNAs and virus production in a cell type-dependent manner.Δγ₁134.5 herpes simplex viruses encoding human cytomegalovirus IRS1 or TRS1 induce interferon regulatory factor 3 phosphorylation and an interferon-stimulated gene response.IL-6 Contributes to Corneal Nerve Degeneration after Herpes Simplex Virus Type I Infection.The Herpes Simplex Virus Virion Host Shutoff Protein Enhances Translation of Viral True Late mRNAs Independently of Suppressing Protein Kinase R and Stress Granule Formation.Evidence for translational regulation by the herpes simplex virus virion host shutoff proteinHerpes simplex virus 1 counteracts tetherin restriction via its virion host shutoff activity.Insights into the pathogenesis of herpes simplex encephalitis from mouse models
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P2860
Host responses to wild-type and attenuated herpes simplex virus infection in the absence of Stat1
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 24 December 2008
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Host responses to wild-type an ...... ection in the absence of Stat1
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Host responses to wild-type an ...... ction in the absence of Stat1.
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type
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Host responses to wild-type an ...... ection in the absence of Stat1
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Host responses to wild-type an ...... ction in the absence of Stat1.
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Host responses to wild-type an ...... ection in the absence of Stat1
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Host responses to wild-type an ...... ction in the absence of Stat1.
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P2093
P2860
P356
P1433
P1476
Host responses to wild-type an ...... ection in the absence of Stat1
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P2093
Cristian Cilloniz
David A Leib
Sean C Proll
Thomas H Teal
Tracy Jo Pasieka
P2860
P304
P356
10.1128/JVI.02007-08
P407
P577
2008-12-24T00:00:00Z