Critical tumor suppressor function mediated by epithelial Mig-6 in endometrial cancer. - Wikidata - wikimedia - TriplyDB

Critical tumor suppressor function mediated by epithelial Mig-6 in endometrial cancer.

Critical tumor suppressor function mediated by epithelial Mig-6 in endometrial cancer.

http://www.wikidata.org/entity/Q37100117

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ARID1A Is Essential for Endometrial Function during Early Pregnancy Steroid hormone intervenes in the endometrial tumorigenesis of pten ablation.Mig-6 suppresses endometrial cancer associated with Pten deficiency and ERK activation.Mig-6 gene knockout induces neointimal hyperplasia in the vascular smooth muscle cell.Mig-6 regulates endometrial genes involved in cell cycle and progesterone signaling.LKB1 loss promotes endometrial cancer progression via CCL2-dependent macrophage recruitment Disturbed cartilage and joint homeostasis resulting from a loss of mitogen-inducible gene 6 in a mouse model of joint dysfunction.Upregulation of mitogen-inducible gene 6 triggers antitumor effect and attenuates progesterone resistance in endometrial carcinoma cells.MIG-6 negatively regulates STAT3 phosphorylation in uterine epithelial cells.Panobinostat Enhances Growth Suppressive Effects of Progestin on Endometrial Carcinoma by Increasing Progesterone Receptor and Mitogen-Inducible Gene-6.Mig-6 Mouse Model of Endometrial Cancer.Role of Mig-6 in hepatic glucose metabolism.MIG-6 suppresses endometrial epithelial cell proliferation by inhibiting phospho-AKT.

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Critical tumor suppressor function mediated by epithelial Mig-6 in endometrial cancer.

http://www.wikidata.org/entity/Q37100117

description

article científic

@ca

article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 27 June 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Critical tumor suppressor function mediated by epithelial Mig-6 in endometrial cancer.

@en

Critical tumor suppressor function mediated by epithelial Mig-6 in endometrial cancer.

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type

label

Critical tumor suppressor function mediated by epithelial Mig-6 in endometrial cancer.

@en

Critical tumor suppressor function mediated by epithelial Mig-6 in endometrial cancer.

@nl

prefLabel

Critical tumor suppressor function mediated by epithelial Mig-6 in endometrial cancer.

@en

Critical tumor suppressor function mediated by epithelial Mig-6 in endometrial cancer.

@nl

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0008-5472.CAN-13-0241

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Cancer Research

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Critical tumor suppressor function mediated by epithelial Mig-6 in endometrial cancer.

@en

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Adrienne S McCampbell

http://www.w3.org/2001/XMLSchema#string

Bon Jeong Ku

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Dong-Kee Lee

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Grant D Orvis

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Jae-Wook Jeong

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John P Lydon

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Richard R Behringer

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Russell R Broaddus

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Sung-Nam Cho

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Tae Hoon Kim

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P2860

Cancer statistics, 2013

The behavior of endometrial hyperplasia. A long-term study of "untreated" hyperplasia in 170 patients

Loss of RALT/MIG-6 expression in ERBB2-amplified breast carcinomas enhances ErbB-2 oncogenic potency and favors resistance to Herceptin

Detection of progesterone receptor forms A and B by immunohistochemical analysis

Estrogen suppresses uterine epithelial apoptosis by inducing birc1 expression

Mig6 is a negative regulator of EGF receptor-mediated skin morphogenesis and tumor formation

IAP proteins: blocking the road to death's door

Differential uterine expression of estrogen and progesterone receptors correlates with uterine preparation for implantation and decidualization in the mouse.

The synergistic effect of Mig-6 and Pten ablation on endometrial cancer development and progression.

ERBB receptor feedback inhibitor 1 regulation of estrogen receptor activity is critical for uterine implantation in mice.

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5090-5099

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scholarly article

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10.1158/0008-5472.CAN-13-0241

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16

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73

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2013-06-27T00:00:00Z

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23811943

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3745525

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