Activation of caprine arthritis-encephalitis virus expression during maturation of monocytes to macrophages.
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Sequences in the visna virus long terminal repeat that control transcriptional activity and respond to viral trans-activation: involvement of AP-1 sites in basal activity and trans-activationVisna virus exhibits a complex transcriptional pattern: one aspect of gene expression shared with the acquired immunodeficiency syndrome retrovirusNeutralizing antibodies to visna lentivirus: mechanism of action and possible role in virus persistenceTropism of sheep lentiviruses for monocytes: susceptibility to infection and virus gene expression increase during maturation of monocytes to macrophagesIn vivo monocyte tropism of pathogenic feline immunodeficiency virusesHost hindrance to HIV-1 replication in monocytes and macrophages.Lentiviruses are etiological agents of chronic diseases in animals and acquired immunodeficiency syndrome in humans.Goat uterine epithelial cells are susceptible to infection with Caprine Arthritis Encephalitis Virus (CAEV) in vivoIn vitro and animal models of human immunodeficiency virus infection of the central nervous systemMonocytes mediate HIV neuropathogenesis: mechanisms that contribute to HIV associated neurocognitive disordersCellular localization of simian immunodeficiency virus in lymphoid tissues. I. Immunohistochemistry and electron microscopyIncreased susceptibility of differentiated mononuclear phagocytes to productive infection with human immunodeficiency virus-1 (HIV-1)Human T-cell lymphotropic virus type III shares sequence homology with a family of pathogenic lentiviruses.Pathogenesis of caprine arthritis encephalitis virus. Cellular localization of viral transcripts in tissues of infected goats.The pathogenetic basis of viral tropismThe vif gene is essential for efficient replication of caprine arthritis encephalitis virus in goat synovial membrane cells and affects the late steps of the virus replication cycleThe caprine arthritis encephalitis virus tat gene is dispensable for efficient viral replication in vitro and in vivo.Equine infectious anemia virus replication is upregulated during differentiation of blood monocytes from acutely infected horses.Replication properties of dUTPase-deficient mutants of caprine and ovine lentivirusesHuman immunodeficiency virus type 1 replication is blocked prior to reverse transcription and integration in freshly isolated peripheral blood monocytes.dUTPase-minus caprine arthritis-encephalitis virus is attenuated for pathogenesis and accumulates G-to-A substitutions.In vivo leukocyte tropism of bovine leukemia virus in sheep and cattle.Monocyte maturation controls expression of equine infectious anemia virus.Ovine lentivirus is macrophagetropic and does not replicate productively in T lymphocytes.Direct determination of the point mutation rate of a murine retrovirus.Wild-type equine infectious anemia virus replicates in vivo predominantly in tissue macrophages, not in peripheral blood monocytesInfection of human peripheral blood mononuclear cells with a temperature-sensitive mutant of measles virusActivation/proliferation and apoptosis of bystander goat lymphocytes induced by a macrophage-tropic chimeric caprine arthritis encephalitis virus expressing SIV Nef.Infection of the human monocytic cell line Mono Mac6 with human immunodeficiency virus types 1 and 2 results in long-term production of virus variants with increased cytopathogenicity for CD4+ T cells.The visna virus long terminal repeat directs expression of a reporter gene in activated macrophages, lymphocytes, and the central nervous systems of transgenic mice.Change in host cell tropism associated with in vitro replication of equine infectious anemia virus.Lentivirus-induced interferon inhibits maturation and proliferation of monocytes and restricts the replication of caprine arthritis-encephalitis virus.Infection of peritoneal macrophages in vitro and in vivo with feline immunodeficiency virus.Sequence homology between cloned caprine arthritis encephalitis virus and visna virus, two neurotropic lentivirusesLentiviruses are naturally resident in a latent form in long-term ovine fibroblast culturesLack of neutralizing antibodies to caprine arthritis-encephalitis lentivirus in persistently infected goats can be overcome by immunization with inactivated Mycobacterium tuberculosisThe visna virus genome: evidence for a hypervariable site in the env gene and sequence homology among lentivirus envelope proteins.Small-ruminant lentivirus enhances PrPSc accumulation in cultured sheep microglial cells.Small ruminant lentiviruses: genetic variability, tropism and diagnosis.Development of TaqMan-based qPCR method for detection of caprine arthritis-encephalitis virus (CAEV) infection
P2860
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P2860
Activation of caprine arthritis-encephalitis virus expression during maturation of monocytes to macrophages.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on July 1983
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Activation of caprine arthriti ...... n of monocytes to macrophages.
@en
Activation of caprine arthriti ...... n of monocytes to macrophages.
@nl
type
label
Activation of caprine arthriti ...... n of monocytes to macrophages.
@en
Activation of caprine arthriti ...... n of monocytes to macrophages.
@nl
prefLabel
Activation of caprine arthriti ...... n of monocytes to macrophages.
@en
Activation of caprine arthriti ...... n of monocytes to macrophages.
@nl
P2093
P2860
P1476
Activation of caprine arthriti ...... n of monocytes to macrophages.
@en
P2093
J E Clements
S Kennedy-Stoskopf
P2860
P407
P577
1983-07-01T00:00:00Z