The delta isoform of CaM kinase II is required for pathological cardiac hypertrophy and remodeling after pressure overload. - Wikidata - wikimedia - TriplyDB

The delta isoform of CaM kinase II is required for pathological cardiac hypertrophy and remodeling after pressure overload.

The delta isoform of CaM kinase II is required for pathological cardiac hypertrophy and remodeling after pressure overload.

http://www.wikidata.org/entity/Q37114848

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Histone deacetylase signaling in cardioprotection Ongoing controversies surrounding cardiac remodeling: is it black and white-or rather fifty shades of gray?The role of late I Na in development of cardiac arrhythmias Signaling effectors underlying pathologic growth and remodeling of the heart Ca2+/calmodulin-dependent protein kinase II function in vascular remodelling CaMKII regulation of cardiac K channels Calmodulin-dependent protein kinase II: linking heart failure and arrhythmias The role of O-GlcNAc transferase in regulating the gene transcription of developing and failing hearts Mechanisms of altered Ca²⁺ handling in heart failure New therapeutic targets in cardiology: arrhythmias and Ca2+/calmodulin-dependent kinase II (CaMKII)Left Ventricular T-Cell Recruitment Contributes to the Pathogenesis of Heart Failure.Post-translational modifications regulate class IIa histone deacetylase (HDAC) function in health and disease miR-185 plays an anti-hypertrophic role in the heart via multiple targets in the calcium-signaling pathways The Murine Model of Mucopolysaccharidosis IIIB Develops Cardiopathies over Time Leading to Heart Failure The δA isoform of calmodulin kinase II mediates pathological cardiac hypertrophy by interfering with the HDAC4-MEF2 signaling pathway A β(IV)-spectrin/CaMKII signaling complex is essential for membrane excitability in mice Location matters: clarifying the concept of nuclear and cytosolic CaMKII subtypes Deletion of low molecular weight protein tyrosine phosphatase (Acp1) protects against stress-induced cardiomyopathy.Synergy between CaMKII substrates and β-adrenergic signaling in regulation of cardiac myocyte Ca(2+) handling.Diabetes increases mortality after myocardial infarction by oxidizing CaMKII Novel protective role of endogenous cardiac myocyte P2X4 receptors in heart failure Voltage-Gated Sodium Channel Phosphorylation at Ser571 Regulates Late Current, Arrhythmia, and Cardiac Function In Vivo.Oxidized calmodulin kinase II regulates conduction following myocardial infarction: a computational analysis.CaMKII inhibition reduces isoproterenol-induced ischemia and arrhythmias in hypertrophic mice.The gamma isoform of CaM kinase II controls mouse egg activation by regulating cell cycle resumption Cardioprotection by CaMKII-deltaB is mediated by phosphorylation of heat shock factor 1 and subsequent expression of inducible heat shock protein 70.Protein Kinases as Drug Development Targets for Heart Disease Therapy Calcium/calmodulin-dependent protein kinase II contributes to cardiac arrhythmogenesis in heart failure Aging and CaMKII alter intracellular Ca2+ transients and heart rhythm in Drosophila melanogaster.Loss of MD1 exacerbates pressure overload-induced left ventricular structural and electrical remodelling.Polybrominated diphenyl ether (PBDE) exposure in children: possible associations with cardiovascular and psychological functions.Effective post-insult neuroprotection by a novel Ca(2+)/ calmodulin-dependent protein kinase II (CaMKII) inhibitor.Inhibition of CaMKII does not attenuate cardiac hypertrophy in mice with dysfunctional ryanodine receptor HDAC4: mechanism of regulation and biological functions Tubulin polymerization disrupts cardiac β-adrenergic regulation of late INa.Genetics of hypertrophic cardiomyopathy.Differential cardiac remodeling in preload versus afterload.Essential role of sympathetic endothelin A receptors for adverse cardiac remodeling Heterogeneous myocyte enhancer factor-2 (Mef2) activation in myocytes predicts focal scarring in hypertrophic cardiomyopathy.Role of CaMKII in cardiovascular health, disease, and arrhythmia

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The delta isoform of CaM kinase II is required for pathological cardiac hypertrophy and remodeling after pressure overload.

http://www.wikidata.org/entity/Q37114848

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 28 January 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

The delta isoform of CaM kinas ...... eling after pressure overload.

@en

The delta isoform of CaM kinas ...... eling after pressure overload.

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type

label

The delta isoform of CaM kinas ...... eling after pressure overload.

@en

The delta isoform of CaM kinas ...... eling after pressure overload.

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prefLabel

The delta isoform of CaM kinas ...... eling after pressure overload.

@en

The delta isoform of CaM kinas ...... eling after pressure overload.

@nl

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PNAS.0813013106

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Proceedings of the National Academy of Sciences of the United States of America

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The delta isoform of CaM kinas ...... eling after pressure overload.

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Chad E Grueter

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Eric N Olson

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Hugo A Katus

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James A Richardson

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Johannes Backs

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Joseph A Hill

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Lars S Maier

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Lorenz H Lehmann

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Michael M Kreusser

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Rhonda Bassel-Duby

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P2860

Activation of the myocyte enhancer factor-2 transcription factor by calcium/calmodulin-dependent protein kinase-stimulated binding of 14-3-3 to histone deacetylase 5

CaM kinase II selectively signals to histone deacetylase 4 during cardiomyocyte hypertrophy

Transgenic CaMKIIdeltaC overexpression uniquely alters cardiac myocyte Ca2+ handling: reduced SR Ca2+ load and activated SR Ca2+ release

The deltaC isoform of CaMKII is activated in cardiac hypertrophy and induces dilated cardiomyopathy and heart failure

Role of Ca2+/calmodulin-dependent protein kinase (CaMK) in excitation-contraction coupling in the heart

Protein kinases C and D mediate agonist-dependent cardiac hypertrophy through nuclear export of histone deacetylase 5

Identification and expression of delta-isoforms of the multifunctional Ca2+/calmodulin-dependent protein kinase in failing and nonfailing human myocardium

Inactivation of the myocyte enhancer factor-2 repressor histone deacetylase-5 by endogenous Ca(2+) //calmodulin-dependent kinase II promotes depolarization-mediated cerebellar granule neuron survival

Impaired spatial learning in alpha-calcium-calmodulin kinase II mutant mice

Cardiac excitation-contraction coupling

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2342-2347

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scholarly article

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10.1073/PNAS.0813013106

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7

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106

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David M Patrick

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2009-01-28T00:00:00Z

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23959389

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19179290

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2650158

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