Inhibition of Rac1 reduces store overload-induced calcium release and protects against ventricular arrhythmia

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Inhibition of Rac1 reduces store overload-induced calcium release and protects against ventricular arrhythmia

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scientific article published on 25 May 2016

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Inhibition of Rac1 reduces sto ...... against ventricular arrhythmia

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Inhibition of Rac1 reduces sto ...... gainst ventricular arrhythmia.

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Inhibition of Rac1 reduces sto ...... against ventricular arrhythmia

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Inhibition of Rac1 reduces sto ...... gainst ventricular arrhythmia.

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Inhibition of Rac1 reduces sto ...... against ventricular arrhythmia

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Inhibition of Rac1 reduces sto ...... gainst ventricular arrhythmia.

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P1476

Inhibition of Rac1 reduces sto ...... against ventricular arrhythmia

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P2093

Guoping Wang

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Le Gui

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Lili Zhang

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Stephen M Sims

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Xiangru Lu

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Yan Wu

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Physiological and pathological functions of NADPH oxidases during myocardial ischemia-reperfusion

RyR2 mutations linked to ventricular tachycardia and sudden death reduce the threshold for store-overload-induced Ca2+ release (SOICR)

Ryanodine receptor leak mediated by caspase-8 activation leads to left ventricular injury after myocardial ischemia-reperfusion

The small GTP-binding protein Rac1 induces cardiac myocyte hypertrophy through the activation of apoptosis signal-regulating kinase 1 and nuclear factor-kappa B

Rational design and characterization of a Rac GTPase-specific small molecule inhibitor

Activating autoantibodies to the beta-1 adrenergic and m2 muscarinic receptors facilitate atrial fibrillation in patients with Graves' hyperthyroidism

Requirement of Rac1 in the development of cardiac hypertrophy.

Fundamentals of reperfusion injury for the clinical cardiologist.

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1513-1522

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scholarly article

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10.1111/JCMM.12840

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P433

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P478

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Qingping Feng

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2016-05-25T00:00:00Z

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27222313

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4956946

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