ADAM17 controls endochondral ossification by regulating terminal differentiation of chondrocytes. - Wikidata - wikimedia - TriplyDB

ADAM17 controls endochondral ossification by regulating terminal differentiation of chondrocytes.

ADAM17 controls endochondral ossification by regulating terminal differentiation of chondrocytes.

http://www.wikidata.org/entity/Q37122908

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ADAMTS and ADAM metalloproteinases in osteoarthritis - looking beyond the 'usual suspects'.Activation of HIFa pathway in mature osteoblasts disrupts the integrity of the osteocyte/canalicular network iRhoms 1 and 2 are essential upstream regulators of ADAM17-dependent EGFR signaling Emerging targets in osteoarthritis therapy.Chondrocyte hypertrophy in skeletal development, growth, and disease.Lack of ADAM10 in endothelial cells affects osteoclasts at the chondro-osseus junction.Epithelial Cell-Derived a Disintegrin and Metalloproteinase-17 Confers Resistance to Colonic Inflammation Through EGFR Activation.Altered paracrine signaling from the injured knee joint impairs postnatal long bone growth Reduced EGFR signaling enhances cartilage destruction in a mouse osteoarthritis model.Cushioning the cartilage: a canonical Wnt restricting matter.Structural modeling defines transmembrane residues in ADAM17 that are crucial for Rhbdf2-ADAM17-dependent proteolysis.Elf3 Contributes to Cartilage Degradation in vivo in a Surgical Model of Post-Traumatic Osteoarthritis.EGFR controls bone development by negatively regulating mTOR-signaling during osteoblast differentiation.

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ADAM17 controls endochondral ossification by regulating terminal differentiation of chondrocytes.

http://www.wikidata.org/entity/Q37122908

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 03 June 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

ADAM17 controls endochondral o ...... fferentiation of chondrocytes.

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ADAM17 controls endochondral o ...... fferentiation of chondrocytes.

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ADAM17 controls endochondral o ...... fferentiation of chondrocytes.

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ADAM17 controls endochondral o ...... fferentiation of chondrocytes.

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prefLabel

ADAM17 controls endochondral o ...... fferentiation of chondrocytes.

@en

ADAM17 controls endochondral o ...... fferentiation of chondrocytes.

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Molecular and Cellular Biology

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ADAM17 controls endochondral o ...... fferentiation of chondrocytes.

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Adele Boskey

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Carl P Blobel

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Cecilia L Dragomir

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Daniel Hill

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Dara Froemel

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Darren A Plumb

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Howard C Crawford

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Katherine C Hall

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Licia Selleri

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Mary B Goldring

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MDC9, a widely expressed cellular disintegrin containing cytoplasmic SH3 ligand domains

Mice humanised for the EGF receptor display hypomorphic phenotypes in skin, bone and heart

Cloning of a disintegrin metalloproteinase that processes precursor tumour-necrosis factor-alpha

Untangling the ErbB signalling network

A metalloproteinase disintegrin that releases tumour-necrosis factor-alpha from cells

ADAMs: key components in EGFR signalling and development

Targeted disruption of mouse EGF receptor: effect of genetic background on mutant phenotype

Immune and inflammatory responses in TNF alpha-deficient mice: a critical requirement for TNF alpha in the formation of primary B cell follicles, follicular dendritic cell networks and germinal centers, and in the maturation of the humoral immune re

Collagen XXVII organises the pericellular matrix in the growth plate

Developmental regulation of the growth plate

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3077-3090

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scholarly article

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10.1128/MCB.00291-13

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16

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33

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2013-06-03T00:00:00Z

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237017099

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23732913

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