STX, a Novel Membrane Estrogen Receptor Ligand, Protects Against Amyloid-β Toxicity. - Wikidata - wikimedia - TriplyDB

STX, a Novel Membrane Estrogen Receptor Ligand, Protects Against Amyloid-β Toxicity.

STX, a Novel Membrane Estrogen Receptor Ligand, Protects Against Amyloid-β Toxicity.

http://www.wikidata.org/entity/Q37127229

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The Anti-Amyloid-β and Neuroprotective Properties of a Novel Tricyclic Pyrone Molecule.Membrane-initiated estrogen signaling via Gq-coupled GPCR in the central nervous system.

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STX, a Novel Membrane Estrogen Receptor Ligand, Protects Against Amyloid-β Toxicity.

http://www.wikidata.org/entity/Q37127229

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 03 February 2016

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

STX, a Novel Membrane Estrogen Receptor Ligand, Protects Against Amyloid-β Toxicity.

@en

STX, a Novel Membrane Estrogen Receptor Ligand, Protects Against Amyloid-β Toxicity.

@nl

type

label

STX, a Novel Membrane Estrogen Receptor Ligand, Protects Against Amyloid-β Toxicity.

@en

STX, a Novel Membrane Estrogen Receptor Ligand, Protects Against Amyloid-β Toxicity.

@nl

prefLabel

STX, a Novel Membrane Estrogen Receptor Ligand, Protects Against Amyloid-β Toxicity.

@en

STX, a Novel Membrane Estrogen Receptor Ligand, Protects Against Amyloid-β Toxicity.

@nl

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P1433

Journal of Alzheimer's Disease

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STX, a Novel Membrane Estrogen Receptor Ligand, Protects Against Amyloid-β Toxicity

@en

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Colleen Kawamoto

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Jonathan A Zweig

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Nora E Gray

http://www.w3.org/2001/XMLSchema#string

Philip F Copenhaver

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P2860

Correlative Memory Deficits, Abeta Elevation, and Amyloid Plaques in Transgenic Mice

Neurotrophic and neurotoxic effects of amyloid beta protein: reversal by tachykinin neuropeptides

Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein

Forecasting the global burden of Alzheimer’s disease

Estrogen, mitochondria, and growth of cancer and non-cancer cells

Estrogen amelioration of Aβ-induced defects in mitochondria is mediated by mitochondrial signaling pathway involving ERβ, AKAP and Drp1.

Efficient selection for high-expression transfectants with a novel eukaryotic vector

Fiji: an open-source platform for biological-image analysis.

Estrogen receptor protein interaction with phosphatidylinositol 3-kinase leads to activation of phosphorylated Akt and extracellular signal-regulated kinase 1/2 in the same population of cortical neurons: a unified mechanism of estrogen action

Culturing hippocampal neurons

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391-403

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scholarly article

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10.3233/JAD-150756

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2

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51

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2016-01-01T00:00:00Z

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26890746

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4961356

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