Cross-species genomic and epigenomic landscape of retinoblastoma. - Wikidata - wikimedia - TriplyDB

Cross-species genomic and epigenomic landscape of retinoblastoma.

Cross-species genomic and epigenomic landscape of retinoblastoma.

http://www.wikidata.org/entity/Q37132228

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Transgenic Models in Retinoblastoma Research Orchestrating epigenetic roles targeting ocular tumors Single and Multiple Gene Manipulations in Mouse Models of Human Cancer Multimodality imaging methods for assessing retinoblastoma orthotopic xenograft growth and development CRISPR/Cas9 mediated knockout of rb1 and rbl1 leads to rapid and penetrant retinoblastoma development in Xenopus tropicalis Genetic and Epigenetic Discoveries in Human Retinoblastoma Usp7 protects genomic stability by regulating Bub3.Chromatin remodelers HELLS and UHRF1 mediate the epigenetic deregulation of genes that drive retinoblastoma tumor progression.Targeting the DNA repair pathway in Ewing sarcoma Preclinical Models Provide Scientific Justification and Translational Relevance for Moving Novel Therapeutics into Clinical Trials for Pediatric Cancer.Comparative epigenomics: defining and utilizing epigenomic variations across species, time-course, and individuals.Transgenic Mouse Models of SV40-Induced Cancer.The Childhood Solid Tumor Network: A new resource for the developmental biology and oncology research communities.Lessons from Retinoblastoma: Implications for Cancer, Development, Evolution, and Regenerative Medicine.Retinoblastoma cells activate the AKT pathway and are vulnerable to the PI3K/mTOR inhibitor NVP-BEZ235.Defining a new vision for the retinoblastoma gene: report from the 3rd International Rb Meeting Demethylation of microRNA-142 induced by demethylation agents plays a suppressive role in osteosarcoma cells Epigenetic regulators Rbbp4 and Hdac1 are overexpressed in a zebrafish model of RB1 embryonal brain tumor, and are required for neural progenitor survival and proliferation.Chromatin remodeling protein HELLS is upregulated by inactivation of the RB-E2F pathway and is nonessential for osteosarcoma tumorigenesis A three-dimensional organoid model recapitulates tumorigenic aspects and drug responses of advanced human retinoblastoma

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Cross-species genomic and epigenomic landscape of retinoblastoma.

http://www.wikidata.org/entity/Q37132228

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

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scientific article published on June 2013

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Cross-species genomic and epigenomic landscape of retinoblastoma.

@en

Cross-species genomic and epigenomic landscape of retinoblastoma.

@nl

type

label

Cross-species genomic and epigenomic landscape of retinoblastoma.

@en

Cross-species genomic and epigenomic landscape of retinoblastoma.

@nl

prefLabel

Cross-species genomic and epigenomic landscape of retinoblastoma.

@en

Cross-species genomic and epigenomic landscape of retinoblastoma.

@nl

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ONCOTARGET.1051

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Cross-species genomic and epigenomic landscape of retinoblastoma.

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Armita Bahrami

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Claudia A Benavente

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David Finkelstein

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Geoffrey Neale

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Guolian Kang

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Jamshid Temirov

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Jinghui Zhang

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Justina D McEvoy

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Lei Wei

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Michael A Dyer

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P2860

Loss of pRB causes centromere dysfunction and chromosomal instability

Extension of life-span by introduction of telomerase into normal human cells

BRCA2 is required for neurogenesis and suppression of medulloblastoma

Coexpression of normally incompatible developmental pathways in retinoblastoma genesis

A simple technique for quantitation of low levels of DNA damage in individual cells

A mechanism linking extra centrosomes to chromosomal instability

A human DNA segment with properties of the gene that predisposes to retinoblastoma and osteosarcoma

Topotecan combination chemotherapy in two new rodent models of retinoblastoma.

Human condensin function is essential for centromeric chromatin assembly and proper sister kinetochore orientation.

RNAi mediated acute depletion of retinoblastoma protein (pRb) promotes aneuploidy in human primary cells via micronuclei formation.

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scholarly article

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10.18632/ONCOTARGET.1051

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