NOX4 mediates hypoxia-induced proliferation of human pulmonary artery smooth muscle cells: the role of autocrine production of transforming growth factor-{beta}1 and insulin-like growth factor binding protein-3.

NOX4 mediates hypoxia-induced proliferation of human pulmonary artery smooth muscle cells: the role of autocrine production of transforming growth factor-{beta}1 and insulin-like growth factor binding protein-3.

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Q37138896

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Genetic risk factors for hepatopulmonary syndrome in patients with advanced liver disease Reciprocal regulation of TGF-β and reactive oxygen species: A perverse cycle for fibrosis Biochemistry, physiology, and pathophysiology of NADPH oxidases in the cardiovascular system NADPH oxidases in lung health and disease Hepatitis C Virus (HCV) Proteins Induce NADPH Oxidase 4 Expression in a Transforming Growth Factor -Dependent Manner: a New Contributor to HCV-Induced Oxidative Stress Oxidative stress in cancer and fibrosis: Opportunity for therapeutic intervention with antioxidant compounds, enzymes, and nanoparticles Oxidative stress and glutathione in TGF-beta-mediated fibrogenesis Reactive oxygen species in inflammation and tissue injury Enhanced depolarization-induced pulmonary vasoconstriction following chronic hypoxia requires EGFR-dependent activation of NAD(P)H oxidase 2 The effect of Lipoxin A4 on the interaction between macrophage and osteoblast: possible role in the treatment of aseptic loosening.Wild-type and mutant p53 differentially regulate NADPH oxidase 4 in TGF-β-mediated migration of human lung and breast epithelial cells NADPH oxidases in vascular pathology.NOX enzymes and pulmonary disease.NADPH oxidases: functions and pathologies in the vasculature.Oxidative modification of nuclear mitogen-activated protein kinase phosphatase 1 is involved in transforming growth factor beta1-induced expression of plasminogen activator inhibitor 1 in fibroblasts.The NADPH oxidase subunit NOX4 is a new target gene of the hypoxia-inducible factor-1.Resveratrol prevents hypoxia-induced arginase II expression and proliferation of human pulmonary artery smooth muscle cells via Akt-dependent signaling.NOX4 mediates BMP4-induced upregulation of TRPC1 and 6 protein expressions in distal pulmonary arterial smooth muscle cells The Nox4 inhibitor GKT137831 attenuates hypoxia-induced pulmonary vascular cell proliferation Interactions between calcium and reactive oxygen species in pulmonary arterial smooth muscle responses to hypoxia.NADPH oxidase 4 is expressed in pulmonary artery adventitia and contributes to hypertensive vascular remodeling NADPH oxidase activation in pancreatic cancer cells is mediated through Akt-dependent up-regulation of p22phox (Pro)renin receptor mediates both angiotensin II-dependent and -independent oxidative stress in neuronal cells.Is peroxisome proliferator-activated receptor gamma (PPARγ) a therapeutic target for the treatment of pulmonary hypertension?Effects of chrysin (5,7-dihydroxyflavone) on vascular remodeling in hypoxia-induced pulmonary hypertension in rats Chronic hypoxia promotes pulmonary artery endothelial cell proliferation through H2O2-induced 5-lipoxygenase.Increased oxidative stress and severe arterial remodeling induced by permanent high-flow challenge in experimental pulmonary hypertension.Activation of peroxisome proliferator-activated receptor γ ameliorates monocrotaline-induced pulmonary arterial hypertension in rats.miR-21 regulates chronic hypoxia-induced pulmonary vascular remodeling.Key Role of ROS in the Process of 15-Lipoxygenase/15-Hydroxyeicosatetraenoiccid-Induced Pulmonary Vascular Remodeling in Hypoxia Pulmonary Hypertension.Regulation of NAD(P)H oxidases by AMPK in cardiovascular systems Combating oxidative stress in vascular disease: NADPH oxidases as therapeutic targets Effect of exercise training on nitric oxide and superoxide/H₂O₂ signaling pathways in collateral-dependent porcine coronary arterioles NOX4-Dependent Hydrogen Peroxide Overproduction in Human Atrial Fibrillation and HL-1 Atrial Cells: Relationship to Hypertension.NADPH oxidase-derived ROS and the regulation of pulmonary vessel tone Molecular and cellular mechanisms of pulmonary fibrosis.Serum levels of nicotinamide-adenine dinucleotide phosphate oxidase 4 are associated with non-valvular atrial fibrillation.Transforming growth factor-beta 1 produced by vascular smooth muscle cells predicts fibrosis in the gastrocnemius of patients with peripheral artery disease.Peroxisome proliferator-activated receptor gamma (PPARγ) regulates thrombospondin-1 and Nox4 expression in hypoxia-induced human pulmonary artery smooth muscle cell proliferation Subcutaneous administration of bovine superoxide dismutase protects lungs from radiation-induced lung injury.

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P2860

NOX4 mediates hypoxia-induced proliferation of human pulmonary artery smooth muscle cells: the role of autocrine production of transforming growth factor-{beta}1 and insulin-like growth factor binding protein-3.

Item

http://www.wikidata.org/entity/Q37138896

description

2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年學術文章

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2008年學術文章

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2008年學術文章

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name

NOX4 mediates hypoxia-induced ...... owth factor binding protein-3.

@en

NOX4 mediates hypoxia-induced ...... owth factor binding protein-3.

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type

Item

label

NOX4 mediates hypoxia-induced ...... owth factor binding protein-3.

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NOX4 mediates hypoxia-induced ...... owth factor binding protein-3.

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NOX4 mediates hypoxia-induced ...... owth factor binding protein-3.

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NOX4 mediates hypoxia-induced ...... owth factor binding protein-3.

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P1476

NOX4 mediates hypoxia-induced ...... owth factor binding protein-3.

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P2093

Anne Sturrock

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Barbara Cahill

http://www.w3.org/2001/XMLSchema#string

John Hoidal

http://www.w3.org/2001/XMLSchema#string

Karl Sanders

http://www.w3.org/2001/XMLSchema#string

Kimberly Norman

http://www.w3.org/2001/XMLSchema#string

Ping Wu

http://www.w3.org/2001/XMLSchema#string

Saleh Ismail

http://www.w3.org/2001/XMLSchema#string

Thomas Huecksteadt

http://www.w3.org/2001/XMLSchema#string

Thomas Kennedy

http://www.w3.org/2001/XMLSchema#string

P2860

Cell transformation by the superoxide-generating oxidase Mox1

Identification of renox, an NAD(P)H oxidase in kidney

HIFalpha targeted for VHL-mediated destruction by proline hydroxylation: implications for O2 sensing

Altered growth responses of pulmonary artery smooth muscle cells from patients with primary pulmonary hypertension to transforming growth factor-beta(1) and bone morphogenetic proteins

C. elegans EGL-9 and mammalian homologs define a family of dioxygenases that regulate HIF by prolyl hydroxylation

AKT/PKB signaling: navigating downstream

HIF prolyl-hydroxylase 2 is the key oxygen sensor setting low steady-state levels of HIF-1alpha in normoxia

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Targeting of HIF-alpha to the von Hippel-Lindau ubiquitylation complex by O2-regulated prolyl hydroxylation

The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology

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L489-99

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scholarly article

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10.1152/AJPLUNG.90488.2008

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296

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2008-11-26T00:00:00Z

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23500682

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19036873

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hypoxia

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2660216

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NOX4 mediates hypoxia-induced proliferation of human pulmonary artery smooth muscle cells: the role of autocrine production of transforming growth factor-{beta}1 and insulin-like growth factor binding protein-3.

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P2860

P2860

NOX4 mediates hypoxia-induced proliferation of human pulmonary artery smooth muscle cells: the role of autocrine production of transforming growth factor-{beta}1 and insulin-like growth factor binding protein-3.

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