Contribution of guanine nucleotide exchange factor Vav2 to hyperhomocysteinemic glomerulosclerosis in rats. - Wikidata - wikimedia - TriplyDB

Contribution of guanine nucleotide exchange factor Vav2 to hyperhomocysteinemic glomerulosclerosis in rats.

Contribution of guanine nucleotide exchange factor Vav2 to hyperhomocysteinemic glomerulosclerosis in rats.

http://www.wikidata.org/entity/Q37151470

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Vav family exchange factors: an integrated regulatory and functional view Role of NADPH Oxidase in Metabolic Disease-Related Renal Injury: An Update Hypoxia-inducible factor prolyl-hydroxylase 2 senses high-salt intake to increase hypoxia inducible factor 1alpha levels in the renal medulla Coronary endothelial dysfunction induced by nucleotide oligomerization domain-like receptor protein with pyrin domain containing 3 inflammasome activation during hypercholesterolemia: beyond inflammation Silencing of hypoxia-inducible factor-1α gene attenuates chronic ischemic renal injury in two-kidney, one-clip rats.New insights into TRP channels: Interaction with pattern recognition receptors Redox signaling via lipid raft clustering in homocysteine-induced injury of podocytes.NMDA receptor-mediated activation of NADPH oxidase and glomerulosclerosis in hyperhomocysteinemic rats.Acid sphingomyelinase gene knockout ameliorates hyperhomocysteinemic glomerular injury in mice lacking cystathionine-β-synthase Combined ChIP-Seq and transcriptome analysis identifies AP-1/JunD as a primary regulator of oxidative stress and IL-1β synthesis in macrophages.Contribution of NADPH oxidase to membrane CD38 internalization and activation in coronary arterial myocytes Folic acid mitigates angiotensin-II-induced blood pressure and renal remodeling.Lipid raft redox signaling: molecular mechanisms in health and disease.NADPH oxidase-induced NALP3 inflammasome activation is driven by thioredoxin-interacting protein which contributes to podocyte injury in hyperglycemia Silencing of hypoxia-inducible factor-1α gene attenuated angiotensin II-induced renal injury in Sprague-Dawley rats.Enhanced epithelial-to-mesenchymal transition associated with lysosome dysfunction in podocytes: role of p62/Sequestosome 1 as a signaling hub.Acid sphingomyelinase gene deficiency ameliorates the hyperhomocysteinemia-induced glomerular injury in mice.Reversal by growth hormone of homocysteine-induced epithelial-to-mesenchymal transition through membrane raft-redox signaling in podocytes Implication of CD38 gene in podocyte epithelial-to-mesenchymal transition and glomerular sclerosis.Epithelial-to-mesenchymal transition in podocytes mediated by activation of NADPH oxidase in hyperhomocysteinemia Increased endogenous H2S generation by CBS, CSE, and 3MST gene therapy improves ex vivo renovascular relaxation in hyperhomocysteinemia.Overexpression of HIF prolyl-hydoxylase-2 transgene in the renal medulla induced a salt sensitive hypertension TIPE2, a novel regulator of immunity, protects against experimental stroke.NADPH oxidase-mediated triggering of inflammasome activation in mouse podocytes and glomeruli during hyperhomocysteinemia.Attenuation by statins of membrane raft-redox signaling in coronary arterial endothelium.Protective role of growth hormone against hyperhomocysteinemia-induced glomerular injury.Cross talk between ceramide and redox signaling: implications for endothelial dysfunction and renal disease.Enhancement of autophagy by simvastatin through inhibition of Rac1-mTOR signaling pathway in coronary arterial myocytes.Activation of Nod-like receptor protein 3 inflammasomes turns on podocyte injury and glomerular sclerosis in hyperhomocysteinemia.Role of NADPH oxidase-mediated reactive oxygen species in podocyte injury.Contribution of endogenously produced reactive oxygen species to the activation of podocyte NLRP3 inflammasomes in hyperhomocysteinemia.Inflammasome Activation in Chronic Glomerular Diseases.Endothelial NLRP3 inflammasome activation and enhanced neointima formation in mice by adipokine visfatin.Endothelin signaling via guanine exchange factor C3G in renal glomerular mesangial cells.Contribution of guanine nucleotide exchange factor Vav2 to NLRP3 inflammasome activation in mouse podocytes during hyperhomocysteinemia.Sphingolipids and Redox Signaling in Renal Regulation and Chronic Kidney Diseases.Translational Aspects of Sphingolipid Metabolism in Renal Disorders.High circulatory leptin mediated NOX-2-peroxynitrite-miR21 axis activate mesangial cells and promotes renal inflammatory pathology in nonalcoholic fatty liver disease.

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P2860

Contribution of guanine nucleotide exchange factor Vav2 to hyperhomocysteinemic glomerulosclerosis in rats.

http://www.wikidata.org/entity/Q37151470

description

2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年學術文章

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2008年學術文章

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2008年學術文章

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name

Contribution of guanine nucleo ...... ic glomerulosclerosis in rats.

@en

Contribution of guanine nucleo ...... ic glomerulosclerosis in rats.

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type

label

Contribution of guanine nucleo ...... ic glomerulosclerosis in rats.

@en

Contribution of guanine nucleo ...... ic glomerulosclerosis in rats.

@nl

prefLabel

Contribution of guanine nucleo ...... ic glomerulosclerosis in rats.

@en

Contribution of guanine nucleo ...... ic glomerulosclerosis in rats.

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Contribution of guanine nucleo ...... ic glomerulosclerosis in rats.

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Chun Zhang

http://www.w3.org/2001/XMLSchema#string

Fan Yi

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Lin Tang

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Min Xia

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Ningjun Li

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Pin-Lan Li

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P2860

Vav2 activates Rac1, Cdc42, and RhoA downstream from growth factor receptors but not beta1 integrins

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Ultrasound-microbubble-mediated gene transfer of inducible Smad7 blocks transforming growth factor-beta signaling and fibrosis in rat remnant kidney.

An efficient gene transfer method mediated by ultrasound and microbubbles into the kidney.

Myocardial fibrosis in DOCA-salt hypertensive rats: effect of endothelin ET(A) receptor antagonism.

Molecular basis for Rac2 regulation of phagocyte NADPH oxidase.

Mechanisms of homocysteine-induced glomerular injury and sclerosis.

Oxidative stress and chronic renal failure: markers and management.

Luciferase imaging of a neurotropic viral infection in intact animals.

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