An early complement-dependent and TLR-4-independent phase in the pathogenesis of ethanol-induced liver injury in mice. - Wikidata - wikimedia - TriplyDB

An early complement-dependent and TLR-4-independent phase in the pathogenesis of ethanol-induced liver injury in mice.

An early complement-dependent and TLR-4-independent phase in the pathogenesis of ethanol-induced liver injury in mice.

http://www.wikidata.org/entity/Q37151689

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Macrophages and Alcohol-Related Liver Inflammation Kupffer cells in the liver Alcoholic liver disease: pathogenesis and new therapeutic targets.Laboratory models available to study alcohol-induced organ damage and immune variations: choosing the appropriate model New therapeutic targets in alcoholic hepatitis.Analysis of the liver mitochondrial proteome in response to ethanol and S-adenosylmethionine treatments: novel molecular targets of disease and hepatoprotection.Chronic alcohol exposure increases circulating bioactive oxidized phospholipids.Complement and the alternative pathway play an important role in LPS/D-GalN-induced fulminant hepatic failure.Toll-like receptors in the pathogenesis of alcoholic liver disease.Interleukin-22 treatment ameliorates alcoholic liver injury in a murine model of chronic-binge ethanol feeding: role of signal transducer and activator of transcription 3.Complement alternative pathway activation in human nonalcoholic steatohepatitis.Pathogenesis of alcoholic liver disease: role of oxidative metabolism.Alternative complement pathway component Factor D contributes to efficient clearance of tissue debris following acute CCl₄-induced injury.Molecular mechanisms of alcoholic liver disease: innate immunity and cytokines.Chronic exposure to a high-fat diet induces hepatic steatosis, impairs nitric oxide bioavailability, and modifies the mitochondrial proteome in mice.Redox signaling and the innate immune system in alcoholic liver disease.Upregulation of Kupffer cell α2A-Adrenoceptors and downregulation of MKP-1 mediate hepatic injury in chronic alcohol exposure.Identification of a cytochrome P4502E1/Bid/C1q-dependent axis mediating inflammation in adipose tissue after chronic ethanol feeding to mice.Complement and alcoholic liver disease: role of C1q in the pathogenesis of ethanol-induced liver injury in mice Hepatoprotective and anti-inflammatory cytokines in alcoholic liver disease Inflammatory PAF Receptor Signaling Initiates Hedgehog Signaling and Kidney Fibrogenesis During Ethanol Consumption.Complement activation in acetaminophen-induced liver injury in mice.Inhibition of apoptosis protects mice from ethanol-mediated acceleration of early markers of CCl4 -induced fibrosis but not steatosis or inflammation.NF-κB-DICER-miRs Axis Regulates TNF-α Expression in Responses to Endotoxin Stress.Skin Immunization Obviates Alcohol-Related Immune Dysfunction.Macrophage migration inhibitory factor contributes to ethanol-induced liver injury by mediating cell injury, steatohepatitis, and steatosis.Moderate (2%, v/v) Ethanol Feeding Alters Hepatic Wound Healing after Acute Carbon Tetrachloride Exposure in Mice Absence of receptor interacting protein kinase 3 prevents ethanol-induced liver injury Soluble IgM links apoptosis to complement activation in early alcoholic liver disease in mice.Inflammation in alcoholic liver disease.Differences in innate immune signaling between alcoholic and non-alcoholic steatohepatitis Differential contribution of complement receptor C5aR in myeloid and non-myeloid cells in chronic ethanol-induced liver injury in mice Innate immunity in alcoholic liver disease.STING-IRF3 pathway links endoplasmic reticulum stress with hepatocyte apoptosis in early alcoholic liver disease.Alcoholic lung injury: metabolic, biochemical and immunological aspects.Adiponectin and heme oxygenase-1 suppress TLR4/MyD88-independent signaling in rat Kupffer cells and in mice after chronic ethanol exposure.Focus on: Alcohol and the liver.Formation of gamma-ketoaldehyde-protein adducts during ethanol-induced liver injury in mice.Focus on: Alcohol and the immune system.The functional role of microRNAs in alcoholic liver injury.

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An early complement-dependent and TLR-4-independent phase in the pathogenesis of ethanol-induced liver injury in mice.

http://www.wikidata.org/entity/Q37151689

description

article científic

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article scientifique

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artigo científico

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bilimsel makale

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scientific article published on April 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

An early complement-dependent ...... -induced liver injury in mice.

@en

An early complement-dependent ...... -induced liver injury in mice.

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type

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An early complement-dependent ...... -induced liver injury in mice.

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An early complement-dependent ...... -induced liver injury in mice.

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prefLabel

An early complement-dependent ...... -induced liver injury in mice.

@en

An early complement-dependent ...... -induced liver injury in mice.

@nl

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An early complement-dependent ...... l-induced liver injury in mice

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Abram B Stavitsky

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Laura E Nagy

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M Edward Medof

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Megan R McMullen

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Nico van Rooijen

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Sanjoy Roychowdhury

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P2860

The C5a chemoattractant receptor mediates mucosal defence to infection

Cutting edge: Toll-like receptor 4 (TLR4)-deficient mice are hyporesponsive to lipopolysaccharide: evidence for TLR4 as the Lps gene product

Expression of a functional C5a receptor in regenerating hepatocytes and its involvement in a proliferative signaling pathway in rat

Cytokines in alcoholic and nonalcoholic steatohepatitis.

Functions of anaphylatoxin C5a in rat liver: direct and indirect actions on nonparenchymal and parenchymal cells.

Ethanol, oxidative stress, and cytokine-induced liver cell injury.

Recent insights into the role of the innate immune system in the development of alcoholic liver disease.

Differential contributions of C3, C5, and decay-accelerating factor to ethanol-induced fatty liver in mice.

Molecular aspects of alcohol metabolism: transcription factors involved in early ethanol-induced liver injury.

Regulation of Toll-like receptor-mediated inflammatory response by complement in vivo.

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1326-1334

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scholarly article

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10.1002/HEP.22776

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4

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49

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Michele T Pritchard

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2009-04-01T00:00:00Z

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23768396

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19133650

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Toll-like receptor

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2666108

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