An early complement-dependent and TLR-4-independent phase in the pathogenesis of ethanol-induced liver injury in mice.
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Macrophages and Alcohol-Related Liver InflammationKupffer cells in the liverAlcoholic liver disease: pathogenesis and new therapeutic targets.Laboratory models available to study alcohol-induced organ damage and immune variations: choosing the appropriate modelNew therapeutic targets in alcoholic hepatitis.Analysis of the liver mitochondrial proteome in response to ethanol and S-adenosylmethionine treatments: novel molecular targets of disease and hepatoprotection.Chronic alcohol exposure increases circulating bioactive oxidized phospholipids.Complement and the alternative pathway play an important role in LPS/D-GalN-induced fulminant hepatic failure.Toll-like receptors in the pathogenesis of alcoholic liver disease.Interleukin-22 treatment ameliorates alcoholic liver injury in a murine model of chronic-binge ethanol feeding: role of signal transducer and activator of transcription 3.Complement alternative pathway activation in human nonalcoholic steatohepatitis.Pathogenesis of alcoholic liver disease: role of oxidative metabolism.Alternative complement pathway component Factor D contributes to efficient clearance of tissue debris following acute CCl₄-induced injury.Molecular mechanisms of alcoholic liver disease: innate immunity and cytokines.Chronic exposure to a high-fat diet induces hepatic steatosis, impairs nitric oxide bioavailability, and modifies the mitochondrial proteome in mice.Redox signaling and the innate immune system in alcoholic liver disease.Upregulation of Kupffer cell α2A-Adrenoceptors and downregulation of MKP-1 mediate hepatic injury in chronic alcohol exposure.Identification of a cytochrome P4502E1/Bid/C1q-dependent axis mediating inflammation in adipose tissue after chronic ethanol feeding to mice.Complement and alcoholic liver disease: role of C1q in the pathogenesis of ethanol-induced liver injury in miceHepatoprotective and anti-inflammatory cytokines in alcoholic liver diseaseInflammatory PAF Receptor Signaling Initiates Hedgehog Signaling and Kidney Fibrogenesis During Ethanol Consumption.Complement activation in acetaminophen-induced liver injury in mice.Inhibition of apoptosis protects mice from ethanol-mediated acceleration of early markers of CCl4 -induced fibrosis but not steatosis or inflammation.NF-κB-DICER-miRs Axis Regulates TNF-α Expression in Responses to Endotoxin Stress.Skin Immunization Obviates Alcohol-Related Immune Dysfunction.Macrophage migration inhibitory factor contributes to ethanol-induced liver injury by mediating cell injury, steatohepatitis, and steatosis.Moderate (2%, v/v) Ethanol Feeding Alters Hepatic Wound Healing after Acute Carbon Tetrachloride Exposure in MiceAbsence of receptor interacting protein kinase 3 prevents ethanol-induced liver injurySoluble IgM links apoptosis to complement activation in early alcoholic liver disease in mice.Inflammation in alcoholic liver disease.Differences in innate immune signaling between alcoholic and non-alcoholic steatohepatitisDifferential contribution of complement receptor C5aR in myeloid and non-myeloid cells in chronic ethanol-induced liver injury in miceInnate immunity in alcoholic liver disease.STING-IRF3 pathway links endoplasmic reticulum stress with hepatocyte apoptosis in early alcoholic liver disease.Alcoholic lung injury: metabolic, biochemical and immunological aspects.Adiponectin and heme oxygenase-1 suppress TLR4/MyD88-independent signaling in rat Kupffer cells and in mice after chronic ethanol exposure.Focus on: Alcohol and the liver.Formation of gamma-ketoaldehyde-protein adducts during ethanol-induced liver injury in mice.Focus on: Alcohol and the immune system.The functional role of microRNAs in alcoholic liver injury.
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P2860
An early complement-dependent and TLR-4-independent phase in the pathogenesis of ethanol-induced liver injury in mice.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on April 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
An early complement-dependent ...... -induced liver injury in mice.
@en
An early complement-dependent ...... -induced liver injury in mice.
@nl
type
label
An early complement-dependent ...... -induced liver injury in mice.
@en
An early complement-dependent ...... -induced liver injury in mice.
@nl
prefLabel
An early complement-dependent ...... -induced liver injury in mice.
@en
An early complement-dependent ...... -induced liver injury in mice.
@nl
P2093
P2860
P356
P1433
P1476
An early complement-dependent ...... l-induced liver injury in mice
@en
P2093
Abram B Stavitsky
Laura E Nagy
M Edward Medof
Megan R McMullen
Nico van Rooijen
Sanjoy Roychowdhury
P2860
P304
P356
10.1002/HEP.22776
P407
P577
2009-04-01T00:00:00Z