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Absence of receptor interacting protein kinase 3 prevents ethanol-induced liver injury

Absence of receptor interacting protein kinase 3 prevents ethanol-induced liver injury

http://www.wikidata.org/entity/Q36774034

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An outline of necrosome triggers The Role of Innate Immunity in Alcoholic Liver Disease Interleukin-1 Family Cytokines in Liver Diseases Necroptosis: an emerging type of cell death in liver diseases Photoreceptor cell death and rescue in retinal detachment and degenerations New advances in molecular mechanisms and emerging therapeutic targets in alcoholic liver diseases Programmed necrosis in the cross talk of cell death and inflammation The necroptosis-inducing kinase RIPK3 dampens adipose tissue inflammation and glucose intolerance.Infiltrating monocytes in liver injury and repair Questions and controversies: the role of necroptosis in liver disease Receptor interacting protein kinase 1 mediates murine acetaminophen toxicity independent of the necrosome and not through necroptosis.Mitoneet mediates TNFα-induced necroptosis promoted by exposure to fructose and ethanol.Animals models of gastrointestinal and liver diseases. Animal models of alcohol-induced liver disease: pathophysiology, translational relevance, and challenges Molecular and cellular mechanisms responsible for cellular stress and low-grade inflammation induced by a super-low dose of endotoxin New drug targets for alcoholic liver disease.A Mechanistic Review of Cell Death in Alcohol-Induced Liver Injury.Novel Akt activator SC-79 is a potential treatment for alcohol-induced osteonecrosis of the femoral head Receptor interactive protein kinase 3 promotes Cisplatin-triggered necrosis in apoptosis-resistant esophageal squamous cell carcinoma cells Mechanisms of acetaminophen hepatotoxicity and their translation to the human pathophysiology.Animal Models of Alcoholic Liver Disease: Pathogenesis and Clinical Relevance Necroptosis mediates TNF-induced toxicity of hippocampal neurons.Autophagy in alcohol-induced multiorgan injury: mechanisms and potential therapeutic targets Fight or flight: regulation of emergency hematopoiesis by pyroptosis and necroptosis.A positive feedback loop between RIP3 and JNK controls non-alcoholic steatohepatitis A plug release mechanism for membrane permeation by MLKL.Protective role of HO-1 and carbon monoxide in ethanol-induced hepatocyte cell death and liver injury in mice RIP kinases: key decision makers in cell death and innate immunity.Receptor interacting protein 3-induced RGC-5 cell necroptosis following oxygen glucose deprivation.Upregulation of the receptor-interacting protein 3 expression and involvement in neural tissue damage after spinal cord injury in mice.Cell death and cell death responses in liver disease: mechanisms and clinical relevance Regulation of NKT cell-mediated immune responses to tumours and liver inflammation by mitochondrial PGAM5-Drp1 signalling.Circulating cell death biomarker: good candidates of prognostic indicator for patients with hepatitis B virus related acute-on-chronic liver failure.The B-Raf(V600E) inhibitor dabrafenib selectively inhibits RIP3 and alleviates acetaminophen-induced liver injury.Characterization of RIPK3-mediated phosphorylation of the activation loop of MLKL during necroptosis.Linking Pathogenic Mechanisms of Alcoholic Liver Disease With Clinical Phenotypes.Differential contribution of complement receptor C5aR in myeloid and non-myeloid cells in chronic ethanol-induced liver injury in mice RIP3: a molecular switch for necrosis and inflammation.Increased hepatic receptor interacting protein kinase 3 expression due to impaired proteasomal functions contributes to alcohol-induced steatosis and liver injury.Activation of necroptosis in human and experimental cholestasis.The pseudokinase MLKL mediates programmed hepatocellular necrosis independently of RIPK3 during hepatitis

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P2860

Absence of receptor interacting protein kinase 3 prevents ethanol-induced liver injury

http://www.wikidata.org/entity/Q36774034

description

2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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2013年论文

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2013年论文

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name

Absence of receptor interacting protein kinase 3 prevents ethanol-induced liver injury

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Absence of receptor interacting protein kinase 3 prevents ethanol-induced liver injury

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Absence of receptor interacting protein kinase 3 prevents ethanol-induced liver injury

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Absence of receptor interacting protein kinase 3 prevents ethanol-induced liver injury

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Absence of receptor interacting protein kinase 3 prevents ethanol-induced liver injury

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Absence of receptor interacting protein kinase 3 prevents ethanol-induced liver injury

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Absence of receptor interacting protein kinase 3 prevents ethanol-induced liver injury

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Laura E Nagy

http://www.w3.org/2001/XMLSchema#string

Megan R McMullen

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Sanjoy Roychowdhury

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Sorana G Pisano

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Xiuli Liu

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P2860

Essential role of tumor necrosis factor alpha in alcohol-induced liver injury in mice

Receptor interacting protein kinase-3 determines cellular necrotic response to TNF-alpha

Many stimuli pull the necrotic trigger, an overview

Viral infection and the evolution of caspase 8-regulated apoptotic and necrotic death pathways

A liver full of JNK: signaling in regulation of cell function and disease pathogenesis, and clinical approaches

RIP3, an energy metabolism regulator that switches TNF-induced cell death from apoptosis to necrosis

Molecular mechanisms of necroptosis: an ordered cellular explosion

Necroptosis as an alternative form of programmed cell death.

CYP2E1 potentiation of LPS and TNFα-induced hepatotoxicity by mechanisms involving enhanced oxidative and nitrosative stress, activation of MAP kinases, and mitochondrial dysfunction.

In situ detection of phosphorylated platelet-derived growth factor receptor beta using a generalized proximity ligation method.

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1773-1783

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10.1002/HEP.26200

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5

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57

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2013-03-14T00:00:00Z

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23319235

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3628968

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