Calmodulin kinase II is required for fight or flight sinoatrial node physiology
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Ion Channels in the HeartPopeye domain-containing proteins and stress-mediated modulation of cardiac pacemakingCalmodulin-dependent protein kinase II: linking heart failure and arrhythmiasThe importance of Ca(2+)-dependent mechanisms for the initiation of the heartbeatDiabetes increases mortality after myocardial infarction by oxidizing CaMKIII(f) and SR Ca(2+) release both contribute to pacemaker activity in canine sinoatrial node cellsAge-associated abnormalities of intrinsic automaticity of sinoatrial nodal cells are linked to deficient cAMP-PKA-Ca(2+) signalingA coupled SYSTEM of intracellular Ca2+ clocks and surface membrane voltage clocks controls the timekeeping mechanism of the heart's pacemaker.Funny current provides a relatively modest contribution to spontaneous beating rate regulation of human and rabbit sinoatrial node cells.A paradigm shift for the heart's pacemaker.Aging and CaMKII alter intracellular Ca2+ transients and heart rhythm in Drosophila melanogaster.Electrophysiological remodeling in heart failureA novel quantitative explanation for the autonomic modulation of cardiac pacemaker cell automaticity via a dynamic system of sarcolemmal and intracellular proteins.Polybrominated diphenyl ether (PBDE) exposure in children: possible associations with cardiovascular and psychological functions.Three ways to die suddenly: do they all require calcium calmodulin-dependent protein kinase II?Tubulin polymerization disrupts cardiac β-adrenergic regulation of late INa.Sinoatrial node dysfunction induces cardiac arrhythmias in diabetic mice.Sarcoplasmic reticulum Ca2+ pumping kinetics regulates timing of local Ca2+ releases and spontaneous beating rate of rabbit sinoatrial node pacemaker cells.Ablation of the GNB3 gene in mice does not affect body weight, metabolism or blood pressure, but causes bradycardia.CaMKII inhibition rescues proarrhythmic phenotypes in the model of human ankyrin-B syndrome.Long-term simulated microgravity causes cardiac RyR2 phosphorylation and arrhythmias in mice.Role of CaMKII in cardiovascular health, disease, and arrhythmiaβ-Adrenergic stimulation increases Cav3.1 activity in cardiac myocytes through protein kinase AOxidized Ca(2+)/calmodulin-dependent protein kinase II triggers atrial fibrillation.Cardiac arrhythmia induced by genetic silencing of 'funny' (f) channels is rescued by GIRK4 inactivationBK channels regulate sinoatrial node firing rate and cardiac pacing in vivoThe ryanodine receptor in cardiac physiology and disease.Ca²+/calmodulin-dependent protein kinase II (CaMKII) activity and sinoatrial nodal pacemaker cell energetics.Catecholamine-independent heart rate increases require Ca2+/calmodulin-dependent protein kinase II.Myristoylated peptides potentiate the funny current (I(f)) in sinoatrial myocytes.Coordinating electrical activity of the heart: ankyrin polypeptides in human cardiac disease.A full range of mouse sinoatrial node AP firing rates requires protein kinase A-dependent calcium signaling.The mitochondrial uniporter controls fight or flight heart rate increases.Oxidized CaMKII causes cardiac sinus node dysfunction in mice.Functional roles of Ca(v)1.3, Ca(v)3.1 and HCN channels in automaticity of mouse atrioventricular cells: insights into the atrioventricular pacemaker mechanism.Function and dysfunction of human sinoatrial nodeInhibition of MCU forces extramitochondrial adaptations governing physiological and pathological stress responses in heart.CaMKII inhibition in heart failure, beneficial, harmful, or bothModern perspectives on numerical modeling of cardiac pacemaker cellAldosterone and cardiovascular disease: the heart of the matter.
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Calmodulin kinase II is required for fight or flight sinoatrial node physiology
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 10 March 2009
@en
vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Calmodulin kinase II is required for fight or flight sinoatrial node physiology
@en
Calmodulin kinase II is required for fight or flight sinoatrial node physiology.
@nl
type
label
Calmodulin kinase II is required for fight or flight sinoatrial node physiology
@en
Calmodulin kinase II is required for fight or flight sinoatrial node physiology.
@nl
prefLabel
Calmodulin kinase II is required for fight or flight sinoatrial node physiology
@en
Calmodulin kinase II is required for fight or flight sinoatrial node physiology.
@nl
P2093
P2860
P356
P1476
Calmodulin kinase II is required for fight or flight sinoatrial node physiology
@en
P2093
Isabella M Grumbach
Long-Sheng Song
Madhu V Singh
Mark E Anderson
Olha M Koval
Peter J Mohler
Satyam Sarma
Thomas J Hund
William Kutschke
P2860
P304
P356
10.1073/PNAS.0806422106
P407
P577
2009-03-10T00:00:00Z