Subcellular stress response and induction of molecular chaperones and folding proteins after transient global ischemia in rats.
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Adaptive preconditioning in neurological diseases – therapeutic insights from proteostatic perturbationsHeat shock proteins: cellular and molecular mechanisms in the central nervous system.Mesencephalic astrocyte-derived neurotrophic factor reduces cell apoptosis via upregulating HSP70 in SHSY-5Y cells.Fetal asphyctic preconditioning alters the transcriptional response to perinatal asphyxiaDanhong injection: A modulator for Golgi structural stability after cerebral ischemia-reperfusion injuryProtein misfolding induces hypoxic preconditioning via a subset of the unfolded protein response machinery.Post-ischemic activation of protein kinase C ε protects the hippocampus from cerebral ischemic injury via alterations in cerebral blood flow.Proteomic analysis of primary cultured rat cortical neurons in chemical ischemia.Unfolding the unfolded protein response: unique insights into brain ischemia.Development-dependent regulation of molecular chaperones after hypoxia-ischemia.Therapeutic window of globular adiponectin against cerebral ischemia in diabetic mice: the role of dynamic alteration of adiponectin/adiponectin receptor expressionDevelopment of stable HSPA1A promoter-driven luciferase reporter HepG2 cells for assessing the toxicity of organic pollutants present in air.Protein disulfide isomerase as a novel target for cyclopentenone prostaglandins: implications for hypoxic ischemic injuryCytokines, chaperones and neuroinflammatory responses in heroin-related death: what can we learn from different patterns of cellular expression?MicroRNA overexpression increases cortical neuronal vulnerability to injury.HuR function and translational state analysis following global brain ischemia and reperfusion.Ischemia activates the ATF6 branch of the endoplasmic reticulum stress responseEndoplasmic reticulum stress in age-related macular degeneration: trigger for neovascularization.Protein misfolding, aggregation, and autophagy after brain ischemia.Proteomic Identification of Nrf2-Mediated Phase II Enzymes Critical for Protection of Tao Hong Si Wu Decoction against Oxygen Glucose Deprivation Injury in PC12 Cells.Regulation of mRNA following brain ischemia and reperfusion.The unfolded protein response in the therapeutic effect of hydroxy-DHA against Alzheimer's disease.ER stress and impaired autophagy flux in neuronal degeneration and brain injury.Increased oxidative stress and activated heat shock proteins in human cell lines by silver nanoparticles.Genome-wide identification and characterization of HSP gene superfamily in whitefly (Bemisia tabaci) and expression profiling analysis under temperature stress.Intrathecal heat shock protein 60 mediates neurodegeneration and demyelination in the CNS through a TLR4- and MyD88-dependent pathway.(+)-Naltrexone is neuroprotective and promotes alternative activation in the mouse hippocampus after cardiac arrest/cardiopulmonary resuscitation.A Controversial Medicolegal Issue: Timing the Onset of Perinatal Hypoxic-Ischemic Brain Injury.The timing of perinatal hypoxia/ischemia events in term neonates: a retrospective autopsy study. HSPs, ORP-150 and COX2 are reliable markers to classify acute, perinatal eventsCalnexin silencing in mouse neonatal cardiomyocytes induces Ca2+ cycling defects, ER stress, and apoptosis.Cloning and characterization of rat Luman/CREB3, a transcription factor highly expressed in nervous system tissue.Endoplasmic reticulum stress contributes to heart protection induced by cyclophilin D inhibition.Tat-protein disulfide-isomerase A3: a possible candidate for preventing ischemic damage in the spinal cord.Disease of mRNA Regulation: Relevance for Ischemic Brain Injury.Agaricus blazei Murill Polysaccharides Protect Against Cadmium-Induced Oxidative Stress and Inflammatory Damage in Chicken Spleens.Autophagy regulates endoplasmic reticulum stress in ischemic preconditioning.Ischaemia-induced protein ubiquitinylation is differentially accompanied with heat-shock protein 70 expression after naïve and preconditioned ischaemia.Unfolded protein response to global ischemia following 48 h of reperfusion in the rat brain: the effect of age and meloxicam.Wolfram syndrome: MAMs' connection?
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Subcellular stress response and induction of molecular chaperones and folding proteins after transient global ischemia in rats.
description
article científic
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article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 28 October 2008
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Subcellular stress response an ...... sient global ischemia in rats.
@en
Subcellular stress response an ...... sient global ischemia in rats.
@nl
type
label
Subcellular stress response an ...... sient global ischemia in rats.
@en
Subcellular stress response an ...... sient global ischemia in rats.
@nl
prefLabel
Subcellular stress response an ...... sient global ischemia in rats.
@en
Subcellular stress response an ...... sient global ischemia in rats.
@nl
P2093
P2860
P1433
P1476
Subcellular stress response an ...... sient global ischemia in rats.
@en
P2093
Bingren Hu
Cindy L Liu
Jessie S Truettner
W Dalton Dietrich
P2860
P356
10.1016/J.BRAINRES.2008.10.032
P407
P577
2008-10-28T00:00:00Z